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Carbonylation Modification Regulates Na/K-ATPase Signaling and Salt Sensitivity: A Review and a HypothesisOxidant Mechanisms in Renal Injury and DiseaseProton channels and renal hypertensive injury: a key piece of the Dahl salt-sensitive rat puzzle?Effects of Reactive Oxygen Species on Tubular Transport along the NephronReactive oxygen species, inflammation and calcium oxalate nephrolithiasis.Uric acid promotes apoptosis in human proximal tubule cells by oxidative stress and the activation of NADPH oxidase NOX 4.HV1 acts as a sodium sensor and promotes superoxide production in medullary thick ascending limb of Dahl salt-sensitive ratsReactive Oxygen Species Modulation of Na/K-ATPase Regulates Fibrosis and Renal Proximal Tubular Sodium HandlingInflammation and oxidative stress in obesity-related glomerulopathyThe multifaceted mineralocorticoid receptorNADPH oxidase 4 mediates flow-induced superoxide production in thick ascending limbs.ROS production as a common mechanism of ENaC regulation by EGF, insulin, and IGF-1.Involvement of reactive oxygen species in a feed-forward mechanism of Na/K-ATPase-mediated signaling transduction.Oxidative stress in hypertension: role of the kidney.Protocatechuic Aldehyde Attenuates Cisplatin-Induced Acute Kidney Injury by Suppressing Nox-Mediated Oxidative Stress and Renal Inflammation.NADPH oxidase 4 deficiency reduces aquaporin-2 mRNA expression in cultured renal collecting duct principal cells via increased PDE3 and PDE4 activity.Na/K-ATPase Signaling and Salt Sensitivity: The Role of Oxidative Stress.Nox4 and diabetic nephropathy: with a friend like this, who needs enemies?(Pro)renin receptor activation increases profibrotic markers and fibroblast-like phenotype through MAPK-dependent ROS formation in mouse renal collecting duct cells.Canonical Transient Receptor Potential 6 Channel: A New Target of Reactive Oxygen Species in Renal Physiology and Pathology.Lysine triggers apoptosis through a NADPH oxidase-dependent mechanism in human renal tubular cells.Protein Carbonylation of an Amino Acid Residue of the Na/K-ATPase α1 Subunit Determines Na/K-ATPase Signaling and Sodium Transport in Renal Proximal Tubular CellsNADPH oxidase 4 promotes cisplatin-induced acute kidney injury via ROS-mediated programmed cell death and inflammation.Renal molecular mechanisms underlying altered Na+ handling and genesis of hypertension during adulthood in prenatally undernourished rats.Sodium Citrate Increases Expression and Flux of Mg Transport Carriers Mediated by Activation of MEK/ERK/c-Fos Pathway in Renal Tubular Epithelial Cells
P2860
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P2860
description
2011 nî lūn-bûn
@nan
2011 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
NAD(P)H oxidase and renal epithelial ion transport
@ast
NAD(P)H oxidase and renal epithelial ion transport
@en
type
label
NAD(P)H oxidase and renal epithelial ion transport
@ast
NAD(P)H oxidase and renal epithelial ion transport
@en
prefLabel
NAD(P)H oxidase and renal epithelial ion transport
@ast
NAD(P)H oxidase and renal epithelial ion transport
@en
P2860
P1476
NAD(P)H oxidase and renal epithelial ion transport
@en
P2093
Carlos Schreck
Paul M O'Connor
P2860
P304
P356
10.1152/AJPREGU.00618.2010
P577
2011-01-26T00:00:00Z