CYP2J2 overexpression protects against arrhythmia susceptibility in cardiac hypertrophy - Wikidata - awgldk - TriplyDB

CYP2J2 overexpression protects against arrhythmia susceptibility in cardiac hypertrophy

CYP2J2 overexpression protects against arrhythmia susceptibility in cardiac hypertrophy

http://www.wikidata.org/entity/Q34983743

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Dietary omega-3 fatty acids modulate the eicosanoid profile in man primarily via the CYP-epoxygenase pathway Epoxyeicosatrienoic acids and cardioprotection: the road to translation Soluble epoxide hydrolase: A potential target for metabolic diseases Mechanistic Pathways of Sex Differences in Cardiovascular Disease.Downregulation of connexin43 by microRNA-130a in cardiomyocytes results in cardiac arrhythmias.Ophiopogonin D maintains Ca2+ homeostasis in rat cardiomyocytes in vitro by upregulating CYP2J3/EETs and suppressing ER stress.CYP2J2 participates in atherogenesis by mediating cell proliferation, migration and foam cell formation Cytochrome P450 and myocardial ischemia: potential pharmacological implication for cardioprotection.Cytochrome P450 monooxygenase lipid metabolites are significant second messengers in the resolution of choroidal neovascularization.Soluble epoxide hydrolase inhibitors, t-AUCB, regulated microRNA-1 and its target genes in myocardial infarction mice.Orally Active Epoxyeicosatrienoic Acid Analogs.CYP2J2 expression in adult ventricular myocytes protects against external and drug induced ROS toxicity.Unsettled Issues and Future Directions for Research on Cardiovascular Diseases in Women

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P2860

CYP2J2 overexpression protects against arrhythmia susceptibility in cardiac hypertrophy

http://www.wikidata.org/entity/Q34983743

description

2013 nî lūn-bûn

@nan

2013 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2013 թվականի օգոստոսին հրատարակված գիտական հոդված

@hy

2013年の論文

@ja

2013年論文

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2013年論文

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2013年論文

@zh-hk

2013年論文

@zh-mo

2013年論文

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2013年论文

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name

CYP2J2 overexpression protects against arrhythmia susceptibility in cardiac hypertrophy

@ast

CYP2J2 overexpression protects against arrhythmia susceptibility in cardiac hypertrophy

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CYP2J2 overexpression protects against arrhythmia susceptibility in cardiac hypertrophy

@nl

type

label

CYP2J2 overexpression protects against arrhythmia susceptibility in cardiac hypertrophy

@ast

CYP2J2 overexpression protects against arrhythmia susceptibility in cardiac hypertrophy

@en

CYP2J2 overexpression protects against arrhythmia susceptibility in cardiac hypertrophy

@nl

prefLabel

CYP2J2 overexpression protects against arrhythmia susceptibility in cardiac hypertrophy

@ast

CYP2J2 overexpression protects against arrhythmia susceptibility in cardiac hypertrophy

@en

CYP2J2 overexpression protects against arrhythmia susceptibility in cardiac hypertrophy

@nl

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JOURNAL.PONE.0073490

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CYP2J2 overexpression protects against arrhythmia susceptibility in cardiac hypertrophy

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Anne Konkel

http://www.w3.org/2001/XMLSchema#string

Bastian Spallek

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Carola Schubert

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Christina Westphal

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Fatimunnisa Qadri

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J Alyce Bradbury

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Laura M DeGraff

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Robert Fischer

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Vera Regitz-Zagrosek

http://www.w3.org/2001/XMLSchema#string

Wolf-Hagen Schunck

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P2860

Molecular cloning and expression of CYP2J2, a human cytochrome P450 arachidonic acid epoxygenase highly expressed in heart

Enhanced postischemic functional recovery in CYP2J2 transgenic hearts involves mitochondrial ATP-sensitive K+ channels and p42/p44 MAPK pathway

Enhancement of cardiac L-type Ca2+ currents in transgenic mice with cardiac-specific overexpression of CYP2J2

Activation of ATP-sensitive K(+) channels by epoxyeicosatrienoic acids in rat cardiac ventricular myocytes

Soluble epoxide hydrolase inhibitors and heart failure

Endothelial expression of human cytochrome P450 epoxygenase CYP2C8 increases susceptibility to ischemia-reperfusion injury in isolated mouse heart

Role of epoxyeicosatrienoic acids in protecting the myocardium following ischemia/reperfusion injury

Conduction slowing and sudden arrhythmic death in mice with cardiac-restricted inactivation of connexin43

Phosphatase-resistant gap junctions inhibit pathological remodeling and prevent arrhythmias.

Female sex and estrogen receptor-beta attenuate cardiac remodeling and apoptosis in pressure overload.

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e73490

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scholarly article

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10.1371/JOURNAL.PONE.0073490

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8

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Dominik N. Muller

Darryl C Zeldin

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2013-08-30T00:00:00Z

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