Transcription of the herpes simplex virus latency-associated transcript promotes the formation of facultative heterochromatin on lytic promoters.
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Role of ND10 nuclear bodies in the chromatin repression of HSV-1Regulatory RNAs discovered in unexpected placesHerpes simplex virus type 1 latency and reactivation: an updateA comparison of herpes simplex virus type 1 and varicella-zoster virus latency and reactivationThe molecular basis of herpes simplex virus latencyA cultured affair: HSV latency and reactivation in neuronsTransient reversal of episome silencing precedes VP16-dependent transcription during reactivation of latent HSV-1 in neuronsRestarting Lytic Gene Transcription at the Onset of Herpes Simplex Virus ReactivationNeuronal Stress Pathway Mediating a Histone Methyl/Phospho Switch Is Required for Herpes Simplex Virus Reactivation.The epigenetic landscape of latent Kaposi sarcoma-associated herpesvirus genomes.Host microRNA regulation of human cytomegalovirus immediate early protein translation promotes viral latency.Transcriptional coactivator HCF-1 couples the histone chaperone Asf1b to HSV-1 DNA replication componentsHerpes simplex virus VP16, but not ICP0, is required to reduce histone occupancy and enhance histone acetylation on viral genomes in U2OS osteosarcoma cells.Epigenetic analysis of KSHV latent and lytic genomes.Epigenetic regulation of latent HSV-1 gene expression.Transcription of the herpes simplex virus 1 genome during productive and quiescent infection of neuronal and nonneuronal cells.Changes to euchromatin on LAT and ICP4 following reactivation are more prevalent in an efficiently reactivating strain of HSV-1.Numerous conserved and divergent microRNAs expressed by herpes simplex viruses 1 and 2.Polycomb repressive complex 2 targets murine cytomegalovirus chromatin for modification and associates with viral replication centers.Herpes keratitis.Snapshots: chromatin control of viral infection.Tegument protein control of latent herpesvirus establishment and animation.A 2.5-kilobase deletion containing a cluster of nine microRNAs in the latency-associated-transcript locus of the pseudorabies virus affects the host response of porcine trigeminal ganglia during established latency.Long antisense non-coding RNAs function to direct epigenetic complexes that regulate transcription in human cells.Herpesviral replication compartments move and coalesce at nuclear speckles to enhance export of viral late mRNA.Biphasic euchromatin-to-heterochromatin transition on the KSHV genome following de novo infection.The checkpoints of viral gene expression in productive and latent infection: the role of the HDAC/CoREST/LSD1/REST repressor complexViral noncoding RNAs: more surprises.Characterization of herpes simplex virus 2 primary microRNA Transcript regulationInhibition of LSD1 reduces herpesvirus infection, shedding, and recurrence by promoting epigenetic suppression of viral genomesDynamic modulation of HSV chromatin drives initiation of infection and provides targets for epigenetic therapies.Nuclear sensing of viral DNA, epigenetic regulation of herpes simplex virus infection, and innate immunityBarrier-to-Autointegration Factor 1 (BAF/BANF1) Promotes Association of the SETD1A Histone Methyltransferase with Herpes Simplex Virus Immediate-Early Gene Promoters.Roles of the nuclear lamina in stable nuclear association and assembly of a herpesviral transactivator complex on viral immediate-early genes.The number of alphaherpesvirus particles infecting axons and the axonal protein repertoire determines the outcome of neuronal infectionHuman cytomegalovirus persistence.The HSV-1 Latency-Associated Transcript Functions to Repress Latent Phase Lytic Gene Expression and Suppress Virus Reactivation from Latently Infected NeuronsMammalian alphaherpesvirus miRNAs.Clues to mechanisms of herpesviral latent infection and potential curesInfluence of herpes simplex virus 1 latency-associated transcripts on the establishment and maintenance of latency in the ROSA26R reporter mouse model
P2860
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P2860
Transcription of the herpes simplex virus latency-associated transcript promotes the formation of facultative heterochromatin on lytic promoters.
description
2009 nî lūn-bûn
@nan
2009 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Transcription of the herpes si ...... ochromatin on lytic promoters.
@ast
Transcription of the herpes si ...... ochromatin on lytic promoters.
@en
Transcription of the herpes si ...... ochromatin on lytic promoters.
@nl
type
label
Transcription of the herpes si ...... ochromatin on lytic promoters.
@ast
Transcription of the herpes si ...... ochromatin on lytic promoters.
@en
Transcription of the herpes si ...... ochromatin on lytic promoters.
@nl
prefLabel
Transcription of the herpes si ...... ochromatin on lytic promoters.
@ast
Transcription of the herpes si ...... ochromatin on lytic promoters.
@en
Transcription of the herpes si ...... ochromatin on lytic promoters.
@nl
P2093
P2860
P356
P1433
P1476
Transcription of the herpes si ...... ochromatin on lytic promoters.
@en
P2093
Anna R Cliffe
David A Garber
David M Knipe
P2860
P304
P356
10.1128/JVI.00712-09
P407
P577
2009-06-10T00:00:00Z