High-content siRNA screen reveals global ENaC regulators and potential cystic fibrosis therapy targets.
about
Current and Emerging Therapies for the Treatment of Cystic Fibrosis or Mitigation of Its SymptomsRecent advances in quantitative high throughput and high content data analysis.RNA interference-based therapeutics for inherited long QT syndromeProtein traffic disorders: an effective high-throughput fluorescence microscopy pipeline for drug discovery.Hallmarks of therapeutic management of the cystic fibrosis functional landscape.Systematic high-content genome-wide RNAi screens of endothelial cell migration and morphology.Exploration of the Peptide Recognition of an Amiloride-sensitive FMRFamide Peptide-gated Sodium Channel.A Small Indel Mutant Mouse Model of Epidermolytic Palmoplantar Keratoderma and Its Application to Mutant-specific shRNA Therapy.ENaC inhibitors for the treatment of cystic fibrosis.Changing the Paradigm - Treating the Basic Defect in Cystic Fibrosis.Repairing the basic defect in cystic fibrosis - one approach is not enough.High-content drug screening for rare diseases.Airway surface liquid homeostasis in cystic fibrosis: pathophysiology and therapeutic targets.Finding new drugs to enhance anion secretion in cystic fibrosis: Toward suitable systems for better drug screening. Report on the pre-conference meeting to the 12th ECFS Basic Science Conference, Albufeira, 25-28 March 2015.Rab GTPases regulate the trafficking of channels and transporters - a focus on cystic fibrosis.Zinc Deficiency via a Splice Switch in Zinc Importer ZIP2/SLC39A2 Causes Cystic Fibrosis-Associated MUC5AC Hypersecretion in Airway Epithelial Cells.High-throughput screening identifies FAU protein as a regulator of mutant cystic fibrosis transmembrane conductance regulator channel.Investigating Alternative Transport of Integral Plasma Membrane Proteins from the ER to the Golgi: Lessons from the Cystic Fibrosis Transmembrane Conductance Regulator (CFTR).A Mathematical Model of the Phosphoinositide Pathway.Effective silencing of ENaC by siRNA delivered with epithelial-targeted nanocomplexes in human cystic fibrosis cells and in mouse lung.
P2860
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P2860
High-content siRNA screen reveals global ENaC regulators and potential cystic fibrosis therapy targets.
description
2013 nî lūn-bûn
@nan
2013 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2013 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
name
High-content siRNA screen reve ...... stic fibrosis therapy targets.
@ast
High-content siRNA screen reve ...... stic fibrosis therapy targets.
@en
High-content siRNA screen reve ...... stic fibrosis therapy targets.
@nl
type
label
High-content siRNA screen reve ...... stic fibrosis therapy targets.
@ast
High-content siRNA screen reve ...... stic fibrosis therapy targets.
@en
High-content siRNA screen reve ...... stic fibrosis therapy targets.
@nl
prefLabel
High-content siRNA screen reve ...... stic fibrosis therapy targets.
@ast
High-content siRNA screen reve ...... stic fibrosis therapy targets.
@en
High-content siRNA screen reve ...... stic fibrosis therapy targets.
@nl
P2093
P50
P1433
P1476
High-content siRNA screen reve ...... ystic fibrosis therapy targets
@en
P2093
Christian Conrad
Inna Uliyakina
Lalida Sirianant
Rainer Schreiber
P304
P356
10.1016/J.CELL.2013.08.045
P407
P50
P577
2013-09-01T00:00:00Z