Myocardial injection of apelin-overexpressing bone marrow cells improves cardiac repair via upregulation of Sirt3 after myocardial infarction - Wikidata - awgldk - TriplyDB

Myocardial injection of apelin-overexpressing bone marrow cells improves cardiac repair via upregulation of Sirt3 after myocardial infarction

Myocardial injection of apelin-overexpressing bone marrow cells improves cardiac repair via upregulation of Sirt3 after myocardial infarction

http://www.wikidata.org/entity/Q34988169

about

Elabela-apelin receptor signaling pathway is functional in mammalian systems.The sirtuin 3 expression profile is associated with pathological and clinical outcomes in colon cancer patients Loss of Sirt3 limits bone marrow cell-mediated angiogenesis and cardiac repair in post-myocardial infarction.HIF-1α polymorphism in the susceptibility of cervical spondylotic myelopathy and its outcome after anterior cervical corpectomy and fusion treatment.Sirt3 is essential for apelin-induced angiogenesis in post-myocardial infarction of diabetes.Serum apelin is associated with left ventricular hypertrophy in untreated hypertension patients.Tumor PHD2 expression is correlated with clinical features and prognosis of patients with HCC receiving liver resection Serum apelin level predicts the major adverse cardiac events in patients with ST elevation myocardial infarction receiving percutaneous coronary intervention.LPS causes pericyte loss and microvascular dysfunction via disruption of Sirt3/angiopoietins/Tie-2 and HIF-2α/Notch3 pathways.The role of autophagy in cardiac hypertrophy.Apelin gene therapy increases myocardial vascular density and ameliorates diabetic cardiomyopathy via upregulation of sirtuin 3.A static pressure sensitive receptor APJ promote H9c2 cardiomyocyte hypertrophy via PI3K-autophagy pathway.Novel pathogenesis: regulation of apoptosis by Apelin/APJ system.Hypoxia promotes bone marrow-derived mesenchymal stem cell proliferation through apelin/APJ/autophagy pathway.Predictive value of apelin-12 in patients with ST-elevation myocardial infarction with different renal function: a prospective observational study.Effects of Apelin on Cardiovascular Aging.Therapeutic Benefit and Gene Network Regulation by Combined Gene Transfer of Apelin, FGF2, and SERCA2a into Ischemic Heart.SIRT3: A New Regulator of Cardiovascular Diseases.

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Myocardial injection of apelin-overexpressing bone marrow cells improves cardiac repair via upregulation of Sirt3 after myocardial infarction

http://www.wikidata.org/entity/Q34988169

description

2013 nî lūn-bûn

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2013 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

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2013 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2013年の論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年论文

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name

Myocardial injection of apelin ...... t3 after myocardial infarction

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Myocardial injection of apelin ...... t3 after myocardial infarction

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Myocardial injection of apelin ...... t3 after myocardial infarction

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type

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Myocardial injection of apelin ...... t3 after myocardial infarction

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Myocardial injection of apelin ...... t3 after myocardial infarction

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Myocardial injection of apelin ...... t3 after myocardial infarction

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Myocardial injection of apelin ...... t3 after myocardial infarction

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Myocardial injection of apelin ...... t3 after myocardial infarction

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Myocardial injection of apelin ...... t3 after myocardial infarction

@en

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Heng Zeng

http://www.w3.org/2001/XMLSchema#string

Jian-Xiong Chen

http://www.w3.org/2001/XMLSchema#string

Lanfang Li

http://www.w3.org/2001/XMLSchema#string

Xiaochen He

http://www.w3.org/2001/XMLSchema#string

Xuwei Hou

http://www.w3.org/2001/XMLSchema#string

P2860

SIRT3 is a stress-responsive deacetylase in cardiomyocytes that protects cells from stress-mediated cell death by deacetylation of Ku70

Apelin and its receptor control heart field formation during zebrafish gastrulation

Nucleocytoplasmic shuttling of the NAD+-dependent histone deacetylase SIRT1

Transplantation of progenitor cells and regeneration enhancement in acute myocardial infarction: final one-year results of the TOPCARE-AMI Trial.

Infarct remodeling after intracoronary progenitor cell treatment in patients with acute myocardial infarction (TOPCARE-AMI): mechanistic insights from serial contrast-enhanced magnetic resonance imaging.

Angiogenesis and vasculogenesis as therapeutic strategies for postnatal neovascularization

Diet and exercise signals regulate SIRT3 and activate AMPK and PGC-1alpha in skeletal muscle

Exogenous NAD blocks cardiac hypertrophic response via activation of the SIRT3-LKB1-AMP-activated kinase pathway

Acute myocardial infarction activates progenitor cells and increases Wnt signalling in the bone marrow.

Overexpression of angiopoietin-1 increases CD133+/c-kit+ cells and reduces myocardial apoptosis in db/db mouse infarcted hearts.

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