Hyper-responsive phenotype in localized aggressive periodontitis. - Wikidata - awgldk - TriplyDB

Hyper-responsive phenotype in localized aggressive periodontitis.

Hyper-responsive phenotype in localized aggressive periodontitis.

http://www.wikidata.org/entity/Q34988686

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Bacterial invasion and persistence: critical events in the pathogenesis of periodontitis?Macrophage inflammatory protein-1α shows predictive value as a risk marker for subjects and sites vulnerable to bone loss in a longitudinal model of aggressive periodontitis Localized aggressive periodontitis treatment response in primary and permanent dentitions A consortium of Aggregatibacter actinomycetemcomitans, Streptococcus parasanguinis, and Filifactor alocis is present in sites prior to bone loss in a longitudinal study of localized aggressive periodontitis.Correlation of Aggregatibacter actinomycetemcomitans detection with clinical/immunoinflammatory profile of localized aggressive periodontitis using a 16S rRNA microarray method: a cross-sectional study.Role of exopolysaccharide in Aggregatibacter actinomycetemcomitans-induced bone resorption in a rat model for periodontal disease.Local inflammatory markers and systemic endotoxin in aggressive periodontitis.Periodontal treatment reduces matrix metalloproteinase levels in localized aggressive periodontitis Microbiological characterization in children with aggressive periodontitis.Influence of Periodontal Therapy on Systemic Lipopolysaccharides in Children with Localized Aggressive Periodontitis.Benefits of early systemic antibiotics in localized aggressive periodontitis: a retrospective study Innate immune response to oral bacteria and the immune evasive characteristics of periodontal pathogens.LPS-induced inflammatory response after therapy of aggressive periodontitis Inflammatory Response Influences Treatment of Localized Aggressive Periodontitis Do subjects with aggressive and chronic periodontitis exhibit a different cytokine/chemokine profile in the gingival crevicular fluid? A systematic review.The implication of periodontitis in vascular endothelial dysfunction.Transcriptome of Healthy Gingival Tissue from Edentulous Sites in Patients with a History of Aggressive Periodontitis.Do Genetic Markers of Inflammation Modify the Relationship between Periodontitis and Nonalcoholic Fatty Liver Disease? Findings from the SHIP Study.Estrogen-Induced Monocytic Response Correlates with TMD Pain: A Case Control Study.Systemic inflammatory responses in patients with type 2 diabetes with chronic periodontitis.The association between periodontal disease and periosteal lesions in the St. Mary Graces cemetery, London, England A.D. 1350-1538.Periodontitis and Non-alcoholic Fatty Liver Disease, a population based cohort investigation in the Study of Health in Pomerania.Do leprosy and tuberculosis generate a systemic inflammatory shift? Setting the ground for a new dialogue between experimental immunology and bioarchaeology.Cytokine response patterns to complex biofilms by mononuclear cells discriminate patient disease status and biofilm dysbiosis Epigenetic regulation of inflammation in localized aggressive periodontitis.Localized aggressive periodontitis immune response to healthy and diseased subgingival plaque.Local and Plasma Biomarker Profiles in Localized Aggressive Periodontitis.The Role of NrF2 in the Regulation of Periodontal Health and Disease.Non-Surgical Therapy Reduces Presence of JP2 Clone in Localized Aggressive Periodontitis.Aggregatibacter actinomycetemcomitans in African Americans with Localized Aggressive Periodontitis.Cytokine profile in gingival crevicular fluid and plasma of patients with aggressive periodontitis.The role of extracellular matrix metalloproteinase inducer glycosylation in regulating matrix metalloproteinases in periodontitis.Long-term clinical response to treatment and maintenance of localized aggressive periodontitis: a cohort study.Structural and functional analysis of de-N-acetylase PgaB from periodontopathogen Aggregatibacter actinomycetemcomitans.Clinical Characteristics of Localized Aggressive Periodontitis in Primary Dentition.Linkage of Infection to Adverse Systemic Complications: Periodontal Disease, Toll-Like Receptors, and Other Pattern Recognition Systems.Salivary and Serum Markers Related to Innate Immunity in Generalized Aggressive Periodontitis.Interleukin-17 and interleukin-23 levels in gingival crevicular fluid of patients with chronic and aggressive periodontitis.Multidisciplinary Clinical Management of a Localized Aggressive Periodontitis diagnosed in a Child with Glanzmann's Thrombasthenia

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P2860

Hyper-responsive phenotype in localized aggressive periodontitis.

http://www.wikidata.org/entity/Q34988686

description

2009 nî lūn-bûn

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2009 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի դեկտեմբերին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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name

Hyper-responsive phenotype in localized aggressive periodontitis.

@ast

Hyper-responsive phenotype in localized aggressive periodontitis.

@en

Hyper-responsive phenotype in localized aggressive periodontitis.

@nl

type

label

Hyper-responsive phenotype in localized aggressive periodontitis.

@ast

Hyper-responsive phenotype in localized aggressive periodontitis.

@en

Hyper-responsive phenotype in localized aggressive periodontitis.

@nl

prefLabel

Hyper-responsive phenotype in localized aggressive periodontitis.

@ast

Hyper-responsive phenotype in localized aggressive periodontitis.

@en

Hyper-responsive phenotype in localized aggressive periodontitis.

@nl

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Journal of Dental Research

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Hyper-responsive phenotype in localized aggressive periodontitis.

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P2860

Pathogen recognition and innate immunity

TLR4 links innate immunity and fatty acid-induced insulin resistance

Macrophage inflammatory protein-1alpha is an osteoclastogenic factor in myeloma that is independent of receptor activator of nuclear factor kappaB ligand.

Development of a classification system for periodontal diseases and conditions.

The role of immune responses in bone loss during periodontal disease.

Clinical, pathological and immunological aspects of periodontal disease.

Purification of a human monocyte-derived neutrophil chemotactic factor that has peptide sequence similarity to other host defense cytokines.

Commonality in chronic inflammatory diseases: periodontitis, diabetes, and coronary artery disease.

Increased toll-like receptor (TLR) 2 and TLR4 expression in monocytes from patients with type 1 diabetes: further evidence of a proinflammatory state.

Determinants of host susceptibility in aggressive periodontitis.

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