Hax1 lacks BH modules and is peripherally associated to heavy membranes: implications for Omi/HtrA2 and PARL activity in the regulation of mitochondrial stress and apoptosis.

Hax1 lacks BH modules and is peripherally associated to heavy membranes: implications for Omi/HtrA2 and PARL activity in the regulation of mitochondrial stress and apoptosis.

Item
http://www.wikidata.org/entity/Q34991732
Sequence-specific intramembrane proteolysis: identification of a recognition motif in rhomboid substratesHAX1 regulates E3 ubiquitin ligase activity of cIAPs by promoting their dimerizationHax1 regulates neutrophil adhesion and motility through RhoA.The role of PARL and HtrA2 in striatal neuronal injury after transient global cerebral ischemia.Competition through dimerization between antiapoptotic and proapoptotic HS-1-associated protein X-1 (Hax-1).The PARL family of mitochondrial rhomboid proteases.Mitochondrial quality control and neurological disease: an emerging connection.HAX-1: a family of apoptotic regulators in health and disease.Hax-1: a regulator of calcium signaling and apoptosis progression with multiple roles in human disease.Exploring the anti-apoptotic role of HAX-1 versus BCL-XL in cytokine-dependent bone marrow-derived cells from mice.Kostmann's Disease and HCLS1-Associated Protein X-1 (HAX1).The antiapoptotic OPA1/Parl couple participates in mitochondrial adaptation to heat shockGrb7 and Hax1 may colocalize partially to mitochondria in EGF-treated SKBR3 cells and their interaction can affect Caspase3 cleavage of Hax1Structural and mechanistic basis of Parl activity and regulation.Endoplasmic reticulum (ER) stress triggers Hax1-dependent mitochondrial apoptotic events in cardiac cells.Chemokine Signaling Facilitates Early-Stage Breast Cancer Survival and Invasion through Fibroblast-Dependent Mechanisms.Identification of the Functional Autophagy-Regulatory Domain in HCLS1-Associated Protein X-1 That Resists Against Oxidative Stress.
P2860

Hax1 lacks BH modules and is peripherally associated to heavy membranes: implications for Omi/HtrA2 and PARL activity in the regulation of mitochondrial stress and apoptosis.

Item
http://www.wikidata.org/entity/Q34991732
description
2009 nî lūn-bûn
@nan
2009 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Hax1 lacks BH modules and is p ...... hondrial stress and apoptosis.
@ast
Hax1 lacks BH modules and is p ...... hondrial stress and apoptosis.
@en
Hax1 lacks BH modules and is p ...... hondrial stress and apoptosis.
@nl
type
label
Hax1 lacks BH modules and is p ...... hondrial stress and apoptosis.
@ast
Hax1 lacks BH modules and is p ...... hondrial stress and apoptosis.
@en
Hax1 lacks BH modules and is p ...... hondrial stress and apoptosis.
@nl
prefLabel
Hax1 lacks BH modules and is p ...... hondrial stress and apoptosis.
@ast
Hax1 lacks BH modules and is p ...... hondrial stress and apoptosis.
@en
Hax1 lacks BH modules and is p ...... hondrial stress and apoptosis.
@nl
P1433
P1476
P2093
P2860
P2888
P304
P31
P356
P433
P478
P50
P577
P5875
P698
P932
P356
P1433
P1476
Hax1 lacks BH modules and is p ...... hondrial stress and apoptosis.
@en
P2093
P2860
P2888
P304
P31
P356
P433
P478
P50
P577
P5875
P698
P932