Deficiency in repair of the mitochondrial genome sensitizes proliferating myoblasts to oxidative damage
about
Opposing roles of mitochondrial and nuclear PARP1 in the regulation of mitochondrial and nuclear DNA integrity: implications for the regulation of mitochondrial functionRegulation of satellite cell function in sarcopeniaMitochondrial DNA damage induced autophagy, cell death, and diseaseVitamin E in sarcopenia: current evidences on its role in prevention and treatmentInhibition of hydrogen sulfide biosynthesis sensitizes lung adenocarcinoma to chemotherapeutic drugs by inhibiting mitochondrial DNA repair and suppressing cellular bioenergetics.AP39, a Mitochondria-Targeted Hydrogen Sulfide Donor, Supports Cellular Bioenergetics and Protects against Alzheimer's Disease by Preserving Mitochondrial Function in APP/PS1 Mice and NeuronsA domain in human EXOG converts apoptotic endonuclease to DNA-repair exonuclease.Regulation of mitochondrial poly(ADP-Ribose) polymerase activation by the β-adrenoceptor/cAMP/protein kinase A axis during oxidative stress.AP39, a novel mitochondria-targeted hydrogen sulfide donor, stimulates cellular bioenergetics, exerts cytoprotective effects and protects against the loss of mitochondrial DNA integrity in oxidatively stressed endothelial cells in vitro.Differentiation-Associated Downregulation of Poly(ADP-Ribose) Polymerase-1 Expression in Myoblasts Serves to Increase Their Resistance to Oxidative Stress.Ethanol Extract of Ganoderma lucidum Augments Cellular Anti-oxidant Defense through Activation of Nrf2/HO-1.Classical non-homologous end-joining pathway utilizes nascent RNA for error-free double-strand break repair of transcribed genesTocotrienol-Rich Fraction Ameliorates Antioxidant Defense Mechanisms and Improves Replicative Senescence-Associated Oxidative Stress in Human Myoblasts.Risky repair: DNA-protein crosslinks formed by mitochondrial base excision DNA repair enzymes acting on free radical lesions.In EXOG-depleted cardiomyocytes cell death is marked by a decreased mitochondrial reserve capacity of the electron transport chain.Loss of mitochondrial exo/endonuclease EXOG affects mitochondrial respiration and induces ROS-mediated cardiomyocyte hypertrophy.Melatonin prevents mitochondrial dysfunctions and death in differentiated skeletal muscle cells.The MacroH2A1.1 - PARP1 Axis at the Intersection Between Stress Response and Metabolism
P2860
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P2860
Deficiency in repair of the mitochondrial genome sensitizes proliferating myoblasts to oxidative damage
description
2013 nî lūn-bûn
@nan
2013 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2013 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2013年の論文
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2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
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name
Deficiency in repair of the mi ...... myoblasts to oxidative damage
@ast
Deficiency in repair of the mi ...... myoblasts to oxidative damage
@en
Deficiency in repair of the mi ...... myoblasts to oxidative damage
@nl
type
label
Deficiency in repair of the mi ...... myoblasts to oxidative damage
@ast
Deficiency in repair of the mi ...... myoblasts to oxidative damage
@en
Deficiency in repair of the mi ...... myoblasts to oxidative damage
@nl
prefLabel
Deficiency in repair of the mi ...... myoblasts to oxidative damage
@ast
Deficiency in repair of the mi ...... myoblasts to oxidative damage
@en
Deficiency in repair of the mi ...... myoblasts to oxidative damage
@nl
P2093
P2860
P1433
P1476
Deficiency in repair of the mi ...... myoblasts to oxidative damage
@en
P2093
Bartosz Szczesny
Csaba Szabo
Dillon K Walker
Gabor Olah
Sankar Mitra
P2860
P304
P356
10.1371/JOURNAL.PONE.0075201
P407
P577
2013-09-16T00:00:00Z