Genetic variants synthesize to produce paneth cell phenotypes that define subtypes of Crohn's disease. - Wikidata - awgldk - TriplyDB

Genetic variants synthesize to produce paneth cell phenotypes that define subtypes of Crohn's disease.

Genetic variants synthesize to produce paneth cell phenotypes that define subtypes of Crohn's disease.

http://www.wikidata.org/entity/Q35001662

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Organoid Models of Human Gastrointestinal Development and Disease ATG16L1: A multifunctional susceptibility factor in Crohn disease Genetic variation in IBD: progress, clues to pathogenesis and possible clinical utility Atg16L1 T300A variant decreases selective autophagy resulting in altered cytokine signaling and decreased antibacterial defense Inflammatory bowel disease in pediatric and adolescent patients: a biomolecular and histopathological review Bacterial sensor Nod2 prevents inflammation of the small intestine by restricting the expansion of the commensal Bacteroides vulgatus.The gut microbiota and inflammatory bowel disease.Pathogenesis of Crohn's disease Epithelial Sel1L is required for the maintenance of intestinal homeostasis Role of Autophagy in the Maintenance of Intestinal Homeostasis.Intrinsic Defense Mechanisms of the Intestinal Epithelium Bugs, genes, fatty acids, and serotonin: Unraveling inflammatory bowel disease?Host and microbiota interactions are critical for development of murine Crohn's-like ileitis.Paneth cell defects in Crohn's disease patients promote dysbiosis.Identification of Candidate Genes Related to Inflammatory Bowel Disease Using Minimum Redundancy Maximum Relevance, Incremental Feature Selection, and the Shortest-Path Approach.LRRK2 but not ATG16L1 is associated with Paneth cell defect in Japanese Crohn's disease patients.The use of prognostic factors in inflammatory bowel diseases.Histologic features predicting postoperative Crohn's disease recurrence.Epidemiology and risk factors for IBD.Serological Epithelial Component Proteins Identify Intestinal Complications in Crohn's Disease.Human alpha defensin 5 is a candidate biomarker to delineate inflammatory bowel disease.Genetics and Pathogenesis of Inflammatory Bowel Disease.Epithelial ER Stress in Crohn's Disease and Ulcerative Colitis.The ErbB3 receptor tyrosine kinase negatively regulates Paneth cells by PI3K-dependent suppression of Atoh1.From sensing to shaping microbiota: insights into the role of NOD2 in intestinal homeostasis and progression of Crohn's disease.Pathomechanisms of Oxidative Stress in Inflammatory Bowel Disease and Potential Antioxidant Therapies.The protection role of Atg16l1 in CD11c+dendritic cells in murine colitis.Rip2 Is Required for Nod2-Mediated Lysozyme Sorting in Paneth Cells.Paneth cell α-defensin 6 (HD-6) is an antimicrobial peptide.Defective ATG16L1-mediated removal of IRE1α drives Crohn's disease-like ileitis.Spatial and temporal stability of paneth cell phenotypes in Crohn's disease: implications for prognostic cellular biomarker development.IBD candidate genes and intestinal barrier regulation.Paneth Cell Alterations in the Development and Phenotype of Crohn's Disease.Association of Ribonuclease T2 Gene Polymorphisms With Decreased Expression and Clinical Characteristics of Severity in Crohn's Disease.Myeloid ATG16L1 Facilitates Host-Bacteria Interactions in Maintaining Intestinal Homeostasis.Risk factors for postoperative recurrence of Crohn's disease with emphasis on surgical predictors.Environmental factors regulate Paneth cell phenotype and host susceptibility to intestinal inflammation in Irgm1-deficient mice.Shear stress induces noncanonical autophagy in intestinal epithelial monolayers.Mechanisms and function of autophagy in intestinal disease.Inflammatory Bowel Disease.

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P2860

Genetic variants synthesize to produce paneth cell phenotypes that define subtypes of Crohn's disease.

http://www.wikidata.org/entity/Q35001662

description

2013 nî lūn-bûn

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2013 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

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2013 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2013年の論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年论文

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Genetic variants synthesize to ...... e subtypes of Crohn's disease.

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Genetic variants synthesize to ...... e subtypes of Crohn's disease.

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Genetic variants synthesize to ...... e subtypes of Crohn's disease.

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Genetic variants synthesize to ...... e subtypes of Crohn's disease.

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Genetic variants synthesize to ...... e subtypes of Crohn's disease.

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Genetic variants synthesize to ...... e subtypes of Crohn's disease.

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J.GASTRO.2013.09.048

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Gastroenterology

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Genetic variants synthesize to ...... ne subtypes of Crohn's disease

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P2093

Dalin Li

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Deepti Dhall

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Dermot P B McGovern

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Fadi Towfic

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Kent D Taylor

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Nir Modiano

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Rachel Winter

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Ramnik J Xavier

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Stephan R Targan

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Talin Haritunians

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P2860

Inflammatory bowel disease: clinical aspects and established and evolving therapies

Inflammatory bowel disease epidemiology

Increasing Incidence and Prevalence of the Inflammatory Bowel Diseases With Time, Based on Systematic Review

Host-microbe interactions have shaped the genetic architecture of inflammatory bowel disease

A frameshift mutation in NOD2 associated with susceptibility to Crohn's disease

Deep resequencing of GWAS loci identifies independent rare variants associated with inflammatory bowel disease

Virus-plus-susceptibility gene interaction determines Crohn's disease gene Atg16L1 phenotypes in intestine

Genome-wide meta-analysis increases to 71 the number of confirmed Crohn's disease susceptibility loci

Association of NOD2 leucine-rich repeat variants with susceptibility to Crohn's disease

Host recognition of bacterial muramyl dipeptide mediated through NOD2. Implications for Crohn's disease

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200-209

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1

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Kelli L VanDussen

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257204430

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24076061

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Crohn's disease

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