Elucidating the elite: mechanisms of control in HIV-1 infection.
about
New Insights about Treg and Th17 Cells in HIV Infection and Disease ProgressionThe enduring tale of T cells in HIV immunopathogenesisIncreased Levels of Macrophage Inflammatory Proteins Result in Resistance to R5-Tropic HIV-1 in a Subset of Elite Controllers.Induction of Potent and Long-Lived Antibody and Cellular Immune Responses in the Genitorectal Mucosa Could be the Critical Determinant of HIV Vaccine Efficacy.Factors Associated With the Control of Viral Replication and Virologic Breakthrough in a Recently Infected HIV-1 ControllerMechanisms of Virologic Control and Clinical Characteristics of HIV+ Elite/Viremic ControllersMicroRNAs differentially present in the plasma of HIV elite controllers reduce HIV infection in vitroProlonged control of an HIV type 1 escape variant following treatment interruption in an HLA-B*27-positive patientInduction of CD8+ T-cell responses against novel glioma-associated antigen peptides and clinical activity by vaccinations with {alpha}-type 1 polarized dendritic cells and polyinosinic-polycytidylic acid stabilized by lysine and carboxymethylcelluloVital role for CD8+ cells in controlling retroviral infections.Increased HIV-specific CD8+ T-cell cytotoxic potential in HIV elite controllers is associated with T-bet expression.Transmitted virus fitness and host T cell responses collectively define divergent infection outcomes in two HIV-1 recipients.Vaccinomics and a new paradigm for the development of preventive vaccines against viral infections.CD4+ T cells from elite suppressors are more susceptible to HIV-1 but produce fewer virions than cells from chronic progressors.Post-treatment control of HIV infection.Hospitalization Rates and Reasons Among HIV Elite Controllers and Persons With Medically Controlled HIV Infection.Relationships of PBMC microRNA expression, plasma viral load, and CD4+ T-cell count in HIV-1-infected elite suppressors and viremic patientsMicroRNA Profile in CD8+ T-Lymphocytes from HIV-Infected Individuals: Relationship with Antiviral Immune Response and Disease Progression.A Murine Viral Outgrowth Assay to Detect Residual HIV Type 1 in Patients With Undetectable Viral LoadsEarly HLA-B*57-restricted CD8+ T lymphocyte responses predict HIV-1 disease progression.The effect of Ingenol-B on the suppressive capacity of elite suppressor HIV-specific CD8+ T cells.Stimulation of HIV-1-specific cytolytic T lymphocytes facilitates elimination of latent viral reservoir after virus reactivation.Potential To Streamline Heterologous DNA Prime and NYVAC/Protein Boost HIV Vaccine Regimens in Rhesus Macaques by Employing Improved AntigensHIV reservoirs and latency models.Quantifying T lymphocyte turnover.Natural killer cells in HIV controller patients express an activated effector phenotype and do not up-regulate NKp44 on IL-2 stimulationThe Effect of Latency Reversal Agents on Primary CD8+ T Cells: Implications for Shock and Kill Strategies for Human Immunodeficiency Virus Eradication.Differential requirements of cellular and humoral immune responses for Fv2-associated resistance to erythroleukemia and for regulation of retrovirus-induced myeloid leukemia development.CD8+ T cells from HLA-B*57 elite suppressors effectively suppress replication of HIV-1 escape mutants.The interplay between the X-DING-CD4, IFN-α and IL-8 gene activity in quiescent and mitogen- or HIV-1-exposed PBMCs from HIV-1 elite controllers, AIDS progressors and HIV-negative controls.An evaluation of HIV elite controller definitions within a large seroconverter cohort collaborationHIV persistence and the prospect of long-term drug-free remissions for HIV-infected individuals.Dynamic interplay between viral adaptation and immune recognition during HIV-1 infection.HIV sexual transmission and microbicides.HIV-1 and hematopoietic stem cell transplantation.Developing strategies for HIV-1 eradicationThe genetic basis of resistance to HIV infection and disease progression.Viral suppression of multiple escape mutants by de novo CD8(+) T cell responses in a human immunodeficiency virus-1 infected elite suppressor.Immune Control Despite Protracted Lymphopenia After Chemoradiation in an Elite Controller.Circulating monocytes are not a major reservoir of HIV-1 in elite suppressors
P2860
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P2860
Elucidating the elite: mechanisms of control in HIV-1 infection.
description
2009 nî lūn-bûn
@nan
2009 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Elucidating the elite: mechanisms of control in HIV-1 infection.
@ast
Elucidating the elite: mechanisms of control in HIV-1 infection.
@en
type
label
Elucidating the elite: mechanisms of control in HIV-1 infection.
@ast
Elucidating the elite: mechanisms of control in HIV-1 infection.
@en
prefLabel
Elucidating the elite: mechanisms of control in HIV-1 infection.
@ast
Elucidating the elite: mechanisms of control in HIV-1 infection.
@en
P2093
P1476
Elucidating the elite: mechanisms of control in HIV-1 infection.
@en
P2093
Joel N Blankson
Justin R Bailey
Karen A O'Connell
P304
P356
10.1016/J.TIPS.2009.09.005
P577
2009-12-01T00:00:00Z