TGF-β-induced IRAK-M expression in tumor-associated macrophages regulates lung tumor growth.
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Validation of the effects of TGF-β1 on tumor recurrence and prognosis through tumor retrieval and cell mechanical propertiesMesenchymal stem cells in cardiac regeneration: a detailed progress report of the last 6 years (2010-2015)IL-1 Receptor-Associated Kinase Signaling and Its Role in Inflammation, Cancer Progression, and Therapy ResistanceDysregulation of TGFβ1 Activity in Cancer and Its Influence on the Quality of Anti-Tumor ImmunityuPAR induces expression of transforming growth factor β and interleukin-4 in cancer cells to promote tumor-permissive conditioning of macrophages.Palmitoylated SCP1 is targeted to the plasma membrane and negatively regulates angiogenesis.DMXAA causes tumor site-specific vascular disruption in murine non-small cell lung cancer, and like the endogenous non-canonical cyclic dinucleotide STING agonist, 2'3'-cGAMP, induces M2 macrophage repolarizationInterleukin-1 receptor-associated kinase-3 is a key inhibitor of inflammation in obesity and metabolic syndrome.TGFβ in T cell biology and tumor immunity: Angel or devil?Tumor-produced versican V1 enhances hCAP18/LL-37 expression in macrophages through activation of TLR2 and vitamin D3 signaling to promote ovarian cancer progression in vitro.PPAR agonist-induced reduction of Mcp1 in atherosclerotic plaques of obese, insulin-resistant mice depends on adiponectin-induced Irak3 expression.Glucocorticoids suppress inflammation via the upregulation of negative regulator IRAK-M.IRAK-M promotes alternative macrophage activation and fibroproliferation in bleomycin-induced lung injury.Cathelicidin, an antimicrobial peptide produced by macrophages, promotes colon cancer by activating the Wnt/β-catenin pathwayMushroom β-Glucan May Immunomodulate the Tumor-Associated Macrophages in the Lewis Lung CarcinomaCellular and molecular immunology of lung cancer: therapeutic implications.Pulmonary surfactant protein A and surfactant lipids upregulate IRAK-M, a negative regulator of TLR-mediated inflammation in human macrophages.Morphological study and comprehensive cellular constituents of milky spots in the human omentum.Effect of tumor-associated macrophages on gastric cancer stem cell in omental milky spots and lymph node micrometastasisGene expression changes in human lung cells exposed to arsenic, chromium, nickel or vanadium indicate the first steps in cancerAltered gut microbiota promotes colitis-associated cancer in IL-1 receptor-associated kinase M-deficient mice.Grapefruit-derived nanovectors deliver miR-18a for treatment of liver metastasis of colon cancer by induction of M1 macrophagesThe persistence of low-grade inflammatory monocytes contributes to aggravated atherosclerosis.Enhanced Mucosal Defense and Reduced Tumor Burden in Mice with the Compromised Negative Regulator IRAK-M.Inflammation and Cancer: Extra- and Intracellular Determinants of Tumor-Associated Macrophages as Tumor Promoters.Immune alterations and emerging immunotherapeutic approaches in lung cancer.Anti-tumour strategies aiming to target tumour-associated macrophages.Sepsis-induced expansion of granulocytic myeloid-derived suppressor cells promotes tumour growth through Toll-like receptor 4.Immunological Consequences of Epithelial-Mesenchymal Transition in Tumor Progression.IRAK-M regulates the inhibition of TLR-mediated macrophage immune response during late in vitro Leishmania donovani infection.HMGB1 was a pivotal synergistic effecor for CpG oligonucleotide to enhance the progression of human lung cancer cells.Deficiency in IRAK4 activity attenuates manifestations of murine Lupus.Acute myeloid leukemia creates an arginase-dependent immunosuppressive microenvironment.Breaking the tolerance for tumor: Targeting negative regulators of TLR signaling.High tumor-associated macrophages infiltration is associated with poor prognosis and may contribute to the phenomenon of epithelial-mesenchymal transition in gastric cancerImmunomodulatory Molecule IRAK-M Balances Macrophage Polarization and Determines Macrophage Responses during Renal Fibrosis.TUDCA: An Agonist of the Bile Acid Receptor GPBAR1/TGR5 With Anti-Inflammatory Effects in Microglial Cells.Defining the role of the tumor vasculature in antitumor immunity and immunotherapy.TGF-β and the Tissue Microenvironment: Relevance in Fibrosis and Cancer.MKP-1 coordinates ordered macrophage-phenotype transitions essential for stem cell-dependent tissue repair.
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P2860
TGF-β-induced IRAK-M expression in tumor-associated macrophages regulates lung tumor growth.
description
2011 nî lūn-bûn
@nan
2011 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
TGF-β-induced IRAK-M expressio ...... s regulates lung tumor growth.
@ast
TGF-β-induced IRAK-M expressio ...... s regulates lung tumor growth.
@en
type
label
TGF-β-induced IRAK-M expressio ...... s regulates lung tumor growth.
@ast
TGF-β-induced IRAK-M expressio ...... s regulates lung tumor growth.
@en
prefLabel
TGF-β-induced IRAK-M expressio ...... s regulates lung tumor growth.
@ast
TGF-β-induced IRAK-M expressio ...... s regulates lung tumor growth.
@en
P2093
P2860
P356
P1433
P1476
TGF-β-induced IRAK-M expressio ...... s regulates lung tumor growth.
@en
P2093
P2860
P2888
P304
P356
10.1038/ONC.2010.619
P407
P577
2011-01-31T00:00:00Z
P5875
P6179
1013076232