about
Molecular mimicry, bystander activation, or viral persistence: infections and autoimmune disease.The crystal structure of coxsackievirus B3 RNA-dependent RNA polymerase in complex with its protein primer VPg confirms the existence of a second VPg binding site on Picornaviridae polymerasesThe microbiology of human hygiene and its impact on type 1 diabetesMutation variants generated from nonvirulent coxsackievirus B3 acquire virulence phenotypes by active virus replication.A single coxsackievirus B2 capsid residue controls cytolysis and apoptosis in rhabdomyosarcoma cells.Stress-activated protein kinases are involved in coxsackievirus B3 viral progeny release.Quantitative analysis of myocardial inflammation by flow cytometry in murine autoimmune myocarditis: correlation with cardiac function.Y genetic variation and phenotypic diversity in health and disease.Th2 regulation of viral myocarditis in mice: different roles for TLR3 versus TRIF in progression to chronic disease.Gene-targeted mice lacking the Trex1 (DNase III) 3'-->5' DNA exonuclease develop inflammatory myocarditis.Update on coxsackievirus B3 myocarditis.Myocarditis, microbes and autoimmunity.Host and virus determinants of picornavirus pathogenesis and tropism.Coxsackievirus B3-induced myocarditis: infection of females during the estrus phase of the ovarian cycle leads to activation of T regulatory cellsThe Y chromosome as a regulatory element shaping immune cell transcriptomes and susceptibility to autoimmune disease.Substance P is required for the pathogenesis of EMCV infection in miceCoxsackievirus B3 Directly Induced Th17 Cell Differentiation by Inhibiting Nup98 Expression in Patients with Acute Viral Myocarditis.Coxsackievirus B3 replication and pathogenesis.Antiviral activity of Folium isatidis derived extracts in vitro and in vivo.5' terminal deletions in the genome of a coxsackievirus B2 strain occurred naturally in human heart.Mapping of a quantitative trait locus controlling susceptibility to Coxsackievirus B3-induced viral hepatitis.TLR3 is required for survival following Coxsackievirus B3 infection by driving T lymphocyte activation and polarization: The role of dendritic cells.Coxsackievirus can persist in murine pancreas by deletion of 5' terminal genomic sequences.Curcumin protects mice from coxsackievirus B3-induced myocarditis by inhibiting the phosphatidylinositol 3 kinase/Akt/nuclear factor-κB pathway.Nanoscale tools for rapid and sensitive diagnosis of virusesThe cholinergic anti-inflammatory pathway ameliorates acute viral myocarditis in mice by regulating CD4 T cell differentiation
P2860
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P2860
description
2003 nî lūn-bûn
@nan
2003 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
name
Coxsackievirus experimental heart diseases.
@ast
Coxsackievirus experimental heart diseases.
@en
type
label
Coxsackievirus experimental heart diseases.
@ast
Coxsackievirus experimental heart diseases.
@en
prefLabel
Coxsackievirus experimental heart diseases.
@ast
Coxsackievirus experimental heart diseases.
@en
P356
P1476
Coxsackievirus experimental heart diseases.
@en
P2093
Charles Gauntt
Sally Huber
P304
P356
10.2741/928
P577
2003-01-01T00:00:00Z