miR-223 is overexpressed in T-lymphocytes of patients affected by rheumatoid arthritis. - Wikidata - awgldk - TriplyDB

miR-223 is overexpressed in T-lymphocytes of patients affected by rheumatoid arthritis.

miR-223 is overexpressed in T-lymphocytes of patients affected by rheumatoid arthritis.

http://www.wikidata.org/entity/Q35014077

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Altered microRNA expression profile with miR-146a upregulation in CD4+ T cells from patients with rheumatoid arthritis MicroRNA regulation of lymphocyte tolerance and autoimmunity Progress and challenge of microRNA research in immunity T cell post-transcriptional miRNA-mRNA interaction networks identify targets associated with susceptibility/resistance to collagen-induced arthritis MicroRNA Expression Is Altered in an Ovalbumin-Induced Asthma Model and Targeting miR-155 with Antagomirs Reveals Cellular Specificity Sexual dimorphism in autoimmunity The role of epigenetic mechanisms and processes in autoimmune disorders MicroRNA-155 contributes to enhanced resistance to apoptosis in monocytes from patients with rheumatoid arthritis.Circulating microRNAs as biomarkers for inflammatory diseases.Early first trimester peripheral blood cell microRNA predicts risk of preterm delivery in pregnant women: Proof of concept.Genetics, genomics, and their relevance to pathology and therapy.Identification of microRNAs associated with ileal and colonic Crohn's disease Association of circulating miR-223 and miR-16 with disease activity in patients with early rheumatoid arthritis.Circulating MicroRNA in inflammatory bowel disease.MiR-223 regulates the differentiation of immature neurons The Role of MicroRNAs in Regulatory T Cells and in the Immune Response.MicroRNA, a new paradigm for understanding immunoregulation, inflammation, and autoimmune diseases.MicroRNAs in Human Diseases: From Cancer to Cardiovascular Disease.MicroRNAs in rheumatoid arthritis.MicroRNAs in systemic rheumatic diseases MicroRNAs in Human Diseases: From Autoimmune Diseases to Skin, Psychiatric and Neurodegenerative Diseases Decreased expression of microRNA-130a correlates with TNF-α in the development of osteoarthritis Age-Associated Differences in MiRNA Signatures Are Restricted to CD45RO Negative T Cells and Are Associated with Changes in the Cellular Composition, Activation and Cellular Ageing Utility of Select Plasma MicroRNA for Disease and Cardiovascular Risk Assessment in Patients with Rheumatoid Arthritis.The Impact of MicroRNA-223-3p on IL-17 Receptor D Expression in Synovial Cells.Notch-regulated miR-223 targets the aryl hydrocarbon receptor pathway and increases cytokine production in macrophages from rheumatoid arthritis patients Upregulation of CD4+T-Cell Derived MiR-223 in The Relapsing Phase of Multiple Sclerosis Patients X-Linked miRNAs Associated with Gender Differences in Rheumatoid Arthritis.Sexual dimorphism of miRNA expression: a new perspective in understanding the sex bias of autoimmune diseases.Comprehensive analysis of miRNA expression in T-cell subsets of rheumatoid arthritis patients reveals defined signatures of naive and memory Tregs.Loss of duplexmiR-223 (5p and 3p) aggravates myocardial depression and mortality in polymicrobial sepsis.MicroRNA epigenetics: a new avenue for wound healing research.A mini-review: microRNA in arthritis.Negative regulation of NF-κB and its involvement in rheumatoid arthritis.What do microRNAs mean for rheumatoid arthritis?MicroRNAs: Emerging Regulators of Immune-Mediated Diseases.Endogenous migration modulators as parent compounds for the development of novel cardiovascular and anti-inflammatory drugs.MicroRNAs in rheumatoid arthritis: potential role in diagnosis and therapy.Epigenetics in autoimmune diseases with focus on type 1 diabetes.MicroRNAs as active players in the pathogenesis of multiple sclerosis.

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P2860

miR-223 is overexpressed in T-lymphocytes of patients affected by rheumatoid arthritis.

http://www.wikidata.org/entity/Q35014077

description

2009 nî lūn-bûn

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2009 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

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2009 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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name

miR-223 is overexpressed in T-lymphocytes of patients affected by rheumatoid arthritis.

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miR-223 is overexpressed in T-lymphocytes of patients affected by rheumatoid arthritis.

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type

label

miR-223 is overexpressed in T-lymphocytes of patients affected by rheumatoid arthritis.

@ast

miR-223 is overexpressed in T-lymphocytes of patients affected by rheumatoid arthritis.

@en

prefLabel

miR-223 is overexpressed in T-lymphocytes of patients affected by rheumatoid arthritis.

@ast

miR-223 is overexpressed in T-lymphocytes of patients affected by rheumatoid arthritis.

@en

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J.HUMIMM.2009.11.008

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Human Immunology

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miR-223 is overexpressed in T-lymphocytes of patients affected by rheumatoid arthritis.

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Chiara Giannitti

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Debora Franceschini

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Franca Citarella

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Francesca Meloni

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Gian Domenico Sebastiani

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Gina Scappucci

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Giovanni Minisola

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Giuseppe Macino

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Teresa Colombo

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Valerio Fulci

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206-211

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scholarly article

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10.1016/J.HUMIMM.2009.11.008

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