Involvement of the Wnt/β-catenin signaling pathway in the cellular and molecular mechanisms of fibrosis in endometriosis
about
Curbing Inflammation in Multiple Sclerosis and Endometriosis: Should Mast Cells Be Targeted?Molecular aspects of development and regulation of endometriosisEndometriosis and ovarian cancer: links, risks, and challenges facedThe elusive and controversial roles of estrogen and progesterone receptors in human endometriosisNovel cancer chemotherapy hits by molecular topology: dual Akt and Beta-catenin inhibitorsDating Endometriotic Ovarian Cysts Based on the Content of Cyst Fluid and its Potential Clinical ImplicationsVascular endothelial growth factor receptor-2 inhibitor cediranib causes regression of endometriotic lesions in a rat modelTargeting the Wnt/β-catenin pathway in endometriosis: a potentially effective approach for treatment and prevention.Genome-wide enrichment analysis between endometriosis and obesity-related traits reveals novel susceptibility lociOvarian cancer in endometriosis: clinical and molecular aspects.Effects of U0126 and MK2206 on cell growth and re-growth of endometriotic stromal cells grown on substrates of varying stiffnessEffects of matrix stiffness on epithelial to mesenchymal transition-like processes of endometrial epithelial cells: Implications for the pathogenesis of endometriosis.Enhancer of Zeste homolog 2 (EZH2) induces epithelial-mesenchymal transition in endometriosis.Stem Cell Markers Describe a Transition From Somatic to Pluripotent Cell States in a Rat Model of Endometriosis.Corroborating evidence for platelet-induced epithelial-mesenchymal transition and fibroblast-to-myofibroblast transdifferentiation in the development of adenomyosis.Non-hormonal targets underlying endometriosis: A focus on molecular mechanisms.Reduced Expression of Eukaryotic Translation Initiation Factor 3 Subunit e and Its Possible Involvement in the Epithelial-Mesenchymal Transition in Endometriosis.Molecular Characteristics of the Endometrium in Uterine Adenomyosis and Its Biochemical Microenvironment.Antifibrotic properties of epigallocatechin-3-gallate in endometriosis.Epithelial-to-mesenchymal transition in the development of endometriosis.Cellular Changes Consistent With Epithelial-Mesenchymal Transition and Fibroblast-to-Myofibroblast Transdifferentiation in the Progression of Experimental Endometriosis in Baboons.Soft matrices inhibit cell proliferation and inactivate the fibrotic phenotype of deep endometriotic stromal cells in vitro.EZH2-mediated repression of Dkk1 promotes hepatic stellate cell activation and hepatic fibrosis.CD146 defines commitment of cultured annulus fibrosus cells to express a contractile phenotype.Advanced Role of Hippo Signaling in Endometrial Fibrosis: Implications for Intrauterine Adhesion.Ginsenoside Rg3 Decreases Fibrotic and Invasive Nature of Endometriosis by Modulating miRNA-27b: In Vitro and In Vivo Studies.In vitro and in vivo effects of MK2206 and chloroquine combination therapy on endometriosis: Autophagy may be required for regrowth of endometriosis.Histological and Immunohistochemical Characterization of the Similarity and Difference Between Ovarian Endometriomas and Deep Infiltrating Endometriosis.Co-operation between the AKT and ERK signaling pathways may support growth of deep endometriosis in a fibrotic microenvironment in vitro.17 β-Estradiol promotes vascular endothelial growth factor expression via the Wnt/β-catenin pathway during the pathogenesis of endometriosis.Hypoxia Promotes Invasion of Endometrial Stromal Cells via Hypoxia-Inducible Factor 1α Upregulation-Mediated β-Catenin Activation in Endometriosis.Epigenetic Modulation of Collagen 1A1: Therapeutic Implications in Fibrosis and Endometriosis.Endometriotic mesenchymal stem cells significantly promote fibrogenesis in ovarian endometrioma through the Wnt/β-catenin pathway by paracrine production of TGF-β1 and Wnt1.Intracellular Wnt/Beta-Catenin Signaling Underlying 17beta-Estradiol-Induced Matrix Metalloproteinase 9 Expression in Human Endometriosis.Effects of Pazopanib, Sunitinib, and Sorafenib, Anti-VEGF Agents, on the Growth of Experimental Endometriosis in Rats.Cancer driver mutations in endometriosis: Variations on the major theme of fibrogenesisEndometrial Stem Cells in Farm Animals: Potential Role in Uterine Physiology and Pathology
P2860
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P2860
Involvement of the Wnt/β-catenin signaling pathway in the cellular and molecular mechanisms of fibrosis in endometriosis
description
2013 nî lūn-bûn
@nan
2013 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2013 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
name
Involvement of the Wnt/β-caten ...... s of fibrosis in endometriosis
@ast
Involvement of the Wnt/β-caten ...... s of fibrosis in endometriosis
@en
type
label
Involvement of the Wnt/β-caten ...... s of fibrosis in endometriosis
@ast
Involvement of the Wnt/β-caten ...... s of fibrosis in endometriosis
@en
prefLabel
Involvement of the Wnt/β-caten ...... s of fibrosis in endometriosis
@ast
Involvement of the Wnt/β-caten ...... s of fibrosis in endometriosis
@en
P2860
P1433
P1476
Involvement of the Wnt/β-caten ...... s of fibrosis in endometriosis
@en
P2093
Claude Darcha
Sachiko Matsuzaki
P2860
P304
P356
10.1371/JOURNAL.PONE.0076808
P407
P577
2013-10-04T00:00:00Z