Plasma fatty acids as diagnostic markers in autistic patients from Saudi Arabia. - Wikidata - awgldk - TriplyDB

Plasma fatty acids as diagnostic markers in autistic patients from Saudi Arabia.

Plasma fatty acids as diagnostic markers in autistic patients from Saudi Arabia.

http://www.wikidata.org/entity/Q35023635

about

Chemicals, Nutrition, and Autism Spectrum Disorder: A Mini-Review Perspective Biological Markers for Autism Spectrum Disorders: Advantages of the Use of Receiver Operating Characteristic Curves in Evaluating Marker Sensitivity and Specificity Neuroinflammation in Autism: Plausible Role of Maternal Inflammation, Dietary Omega 3, and Microbiota Metabolomics as a tool for discovery of biomarkers of autism spectrum disorder in the blood plasma of children MIRA: mutual information-based reporter algorithm for metabolic networks Increase of cytosolic phospholipase A2 as hydrolytic enzyme of phospholipids and autism cognitive, social and sensory dysfunction severity.Novel metabolic biomarkers related to sulfur-dependent detoxification pathways in autistic patients of Saudi Arabia.Comprehensive profiling of plasma fatty acid concentrations in young healthy Canadian adults.Systemic review of the epidemiology of autism in Arab Gulf countries Etiology of autistic features: the persisting neurotoxic effects of propionic acid Potential serum biomarkers from a metabolomics study of autism Relationship between Long Chain n-3 Polyunsaturated Fatty Acids and Autism Spectrum Disorder: Systematic Review and Meta-Analysis of Case-Control and Randomised Controlled Trials.The potential relevance of docosahexaenoic acid and eicosapentaenoic acid to the etiopathogenesis of childhood neuropsychiatric disorders.Lipid mediators in plasma of autism spectrum disorders.Comparison of the Deacylase and Deacetylase Activity of Zinc-Dependent HDACs.Erythrocyte fatty acid profiles in children are not predictive of autism spectrum disorder status: a case control study.Clinical and Molecular Characteristics of Mitochondrial Dysfunction in Autism Spectrum Disorder

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P2860

Plasma fatty acids as diagnostic markers in autistic patients from Saudi Arabia.

http://www.wikidata.org/entity/Q35023635

description

2011 nî lūn-bûn

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2011 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2011 թվականի ապրիլին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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name

Plasma fatty acids as diagnostic markers in autistic patients from Saudi Arabia.

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Plasma fatty acids as diagnostic markers in autistic patients from Saudi Arabia.

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type

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Plasma fatty acids as diagnostic markers in autistic patients from Saudi Arabia.

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Plasma fatty acids as diagnostic markers in autistic patients from Saudi Arabia.

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Plasma fatty acids as diagnostic markers in autistic patients from Saudi Arabia.

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Plasma fatty acids as diagnostic markers in autistic patients from Saudi Arabia.

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Lipids in Health and Disease

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Plasma fatty acids as diagnostic markers in autistic patients from Saudi Arabia.

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Abir G Ben Bacha

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Afaf K El-Ansary

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Layla Y Al-Ayahdi

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P2860

Mapping brain maturation

A simple method for the isolation and purification of total lipides from animal tissues

Disorders of mitochondrial fatty acyl-CoA beta-oxidation

Medium chain acyl-CoA dehydrogenase deficiency human genome epidemiology review

Neurobiological effects of intraventricular propionic acid in rats: Possible role of short chain fatty acids on the pathogenesis and characteristics of autism spectrum disorders

The autism diagnostic observation schedule-generic: a standard measure of social and communication deficits associated with the spectrum of autism

Increased activities of Na+/K+-ATPase and Ca2+/Mg2+-ATPase in the frontal cortex and cerebellum of autistic individuals

Secondary carnitine deficiency and impaired docosahexaenoic (22:6n-3) acid synthesis: a common denominator in the pathophysiology of diseases of oxidative phosphorylation and beta-oxidation.

Membrane docosahexaenoate is supplied to the developing brain and retina by the liver.

Impairment of social and moral behavior related to early damage in human prefrontal cortex.

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