Leishmania disease development depends on the presence of apoptotic promastigotes in the virulent inoculum. - Wikidata - awgldk - TriplyDB

Leishmania disease development depends on the presence of apoptotic promastigotes in the virulent inoculum.

Leishmania disease development depends on the presence of apoptotic promastigotes in the virulent inoculum.

http://www.wikidata.org/entity/Q35025132

about

Proteomic analysis of the secretome of Leishmania donovani Peroxisome Proliferator-Activated Receptor-γ-Mediated Polarization of Macrophages in Leishmania Infection Ambivalent Outcomes of Cell Apoptosis: A Barrier or Blessing in Malaria Progression Study of Leishmania pathogenesis in mice: experimental considerations Phosphatidylserine is a global immunosuppressive signal in efferocytosis, infectious disease, and cancer A Review: The Current In Vivo Models for the Discovery and Utility of New Anti-leishmanial Drugs Targeting Cutaneous Leishmaniasis The Dialogue of the Host-Parasite Relationship: Leishmania spp. and Trypanosoma cruzi Infection Receptor-mediated phagocytosis of Leishmania: implications for intracellular survival Programmed cell death in Leishmania: biochemical evidence and role in parasite infectivity Cooperation between apoptotic and viable metacyclics enhances the pathogenesis of Leishmaniasis Human neutrophil peptide-1 (HNP-1): a new anti-leishmanial drug candidate The calpain inhibitor MDL28170 induces the expression of apoptotic markers in Leishmania amazonensis promastigotes Sand fly-Leishmania interactions: long relationships are not necessarily easy Chlamydia pneumoniae hides inside apoptotic neutrophils to silently infect and propagate in macrophages.Influence of Microbes on Neutrophil Life and Death.Ascorbate peroxidase from Leishmania major controls the virulence of infective stage of promastigotes by regulating oxidative stress.Phospholipid and sphingolipid metabolism in Leishmania.The protective Th1 response in mice is induced in the T-cell zone only three weeks after infection with Leishmania major and not during early T-cell activation.Impact of continuous axenic cultivation in Leishmania infantum virulence Efficient capture of infected neutrophils by dendritic cells in the skin inhibits the early anti-leishmania response.HIV-1 promotes intake of Leishmania parasites by enhancing phosphatidylserine-mediated, CD91/LRP-1-dependent phagocytosis in human macrophages.Exposure of phosphatidylserine on Leishmania amazonensis isolates is associated with diffuse cutaneous leishmaniasis and parasite infectivity Aziridine-2,3-dicarboxylate-based cysteine cathepsin inhibitors induce cell death in Leishmania major associated with accumulation of debris in autophagy-related lysosome-like vacuoles.Deception and manipulation: the arms of leishmania, a successful parasite Leishmania promastigotes lack phosphatidylserine but bind annexin V upon permeabilization or miltefosine treatment.Impact of protozoan cell death on parasite-host interactions and pathogenesis Transbilayer dynamics of phospholipids in the plasma membrane of the Leishmania genus LABCG2, a new ABC transporter implicated in phosphatidylserine exposure, is involved in the infectivity and pathogenicity of Leishmania On Programmed Cell Death in Plasmodium falciparum: Status Quo.Effect of pegylated phosphatidylserine-containing liposomes in experimental chronic arthritis Distinct Macrophage Fates after in vitro Infection with Different Species of Leishmania: Induction of Apoptosis by Leishmania (Leishmania) amazonensis, but Not by Leishmania (Viannia) guyanensis.Neutrophils Contribute to the Protection Conferred by ArtinM against Intracellular Pathogens: A Study on Leishmania major.Antagonic activities of Trypanosoma cruzi metacaspases affect the balance between cell proliferation, death and differentiation Apoptosis-like cell death in Leishmania donovani treated with KalsomeTM10, a new liposomal amphotericin B.Enhanced survival of Leishmania major in neutrophil granulocytes in the presence of apoptotic cells.Leishmania mexicana metacaspase is a negative regulator of amastigote proliferation in mammalian cells.Apoptosis-like programmed cell death induces antisense ribosomal RNA (rRNA) fragmentation and rRNA degradation in Leishmania.The Apoptotic Role of Metacaspase in Toxoplasma gondii.The skin as interface in the transmission of arthropod-borne pathogens.Suicide prevention: disruption of apoptotic pathways by protozoan parasites.

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P2860

Leishmania disease development depends on the presence of apoptotic promastigotes in the virulent inoculum.

http://www.wikidata.org/entity/Q35025132

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2006 nî lūn-bûn

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2006 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

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2006 թվականի օգոստոսին հրատարակված գիտական հոդված

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2006年の論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年论文

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Leishmania disease development ...... otes in the virulent inoculum.

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Leishmania disease development ...... otes in the virulent inoculum.

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Leishmania disease development ...... otes in the virulent inoculum.

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PNAS.0600843103

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Proceedings of the National Academy of Sciences of the United States of America

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Leishmania disease development ...... otes in the virulent inoculum.

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Alexander Wenzel

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Annalena Bollinger

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Antje Müller

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Christoph Hölscher

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David Sacks

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Ger van Zandbergen

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Matthias Klinger

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Reinhard Voll

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Shaden Kamhawi

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Tamás Laskay

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P2860

Copper chelation represses the vascular response to injury

Macrophages that have ingested apoptotic cells in vitro inhibit proinflammatory cytokine production through autocrine/paracrine mechanisms involving TGF-beta, PGE2, and PAF

Intracellular survival of Leishmania major in neutrophil granulocytes after uptake in the absence of heat-labile serum factors

Apoptotic cell removal.

Serum-derived protein S binds to phosphatidylserine and stimulates the phagocytosis of apoptotic cells.

On the origin, evolution, and nature of programmed cell death: a timeline of four billion years.

Lipophosphoglycan is a virulence factor distinct from related glycoconjugates in the protozoan parasite Leishmania major.

The role(s) of lipophosphoglycan (LPG) in the establishment of Leishmania major infections in mammalian hosts.

Clearance of apoptotic cells: getting rid of the corpses.

L-arginine-dependent suppression of apoptosis in Trypanosoma cruzi: contribution of the nitric oxide and polyamine pathways.

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13837-13842

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10.1073/PNAS.0600843103

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37

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103

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Martin Herrmann

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6842833

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16945916

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