Diminished bone formation during diabetic fracture healing is related to the premature resorption of cartilage associated with increased osteoclast activity - Wikidata - awgldk - TriplyDB

Diminished bone formation during diabetic fracture healing is related to the premature resorption of cartilage associated with increased osteoclast activity

Diminished bone formation during diabetic fracture healing is related to the premature resorption of cartilage associated with increased osteoclast activity

http://www.wikidata.org/entity/Q35029163

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The Relationship between Metabolic Syndrome and Osteoporosis: A Review Overview of biological mechanisms and applications of three murine models of bone repair: closed fracture with intramedullary fixation, distraction osteogenesis, and marrow ablation by reaming Diabetes mellitus related bone metabolism and periodontal disease Childhood obesity, bone development, and cardiometabolic risk factors A critical review of diabetes, glycemic control, and dental implant therapy The adverse effects of diabetes on osteoarthritis: update on clinical evidence and molecular mechanisms Dental implant survival in diabetic patients; review and recommendations.Lysyl oxidase-like-2 (LOXL2) is a major isoform in chondrocytes and is critically required for differentiation Skeletal Metabolism, Fracture Risk, and Fracture Outcomes in Type 1 and Type 2 Diabetes.Malfunction of bone marrow-derived osteoclasts and the delay of bone fracture healing in diabetic mice.Delayed fracture healing and increased callus adiposity in a C57BL/6J murine model of obesity-associated type 2 diabetes mellitus.Glycemic control and implant stabilization in type 2 diabetes mellitus.A review of osteocyte function and the emerging importance of sclerostin Osteoporosis in diabetes mellitus: Possible cellular and molecular mechanisms.Molecular mechanisms controlling bone formation during fracture healing and distraction osteogenesis Diabetic complications and dysregulated innate immunity.Plasminogen activator inhibitor-1 is involved in impaired bone repair associated with diabetes in female mice.Diabetes reduces mesenchymal stem cells in fracture healing through a TNFα-mediated mechanism Acute phosphate restriction leads to impaired fracture healing and resistance to BMP-2.The effects of elevated hemoglobin A(1c) in patients with type 2 diabetes mellitus on dental implants: Survival and stability at one year.Photobiomodulation and bone healing in diabetic rats: evaluation of bone response using a tibial defect experimental model.Osteogenic progenitors in bone marrow aspirates have clinical potential for tibial non-unions healing in diabetic patients.Altered fibroblast proliferation and apoptosis in diabetic gingival wounds.Diabetes and oral implant failure: a systematic review.Loss of Bone and Wnt10b Expression in Male Type 1 Diabetic Mice Is Blocked by the Probiotic Lactobacillus reuteri.Augmented LPS responsiveness in type 1 diabetes-derived osteoclasts.Ordinary and Activated Bone Grafts: Applied Classification and the Main Features.Diabetic foot osteomyelitis: bone markers and treatment outcomes.Moderate excess of pyruvate augments osteoclastogenesis.Interrelationship Between Periapical Lesion and Systemic Metabolic Disorders.Measurement of fracture callus material properties via nanoindentation Low dose aspirin therapy decreases blood glucose levels but does not prevent type i diabetes-induced bone loss Palmitic acid and DGAT1 deficiency enhance osteoclastogenesis, while oleic acid-induced triglyceride formation prevents it Diabetes Stimulates Osteoclastogenesis by Acidosis-Induced Activation of Transient Receptor Potential Cation Channels.Production of VEGF receptor 1 and 2 mRNA and protein during endochondral bone repair is differential and healing phase specific.Diabetes causes the accelerated loss of cartilage during fracture repair which is reversed by insulin treatment.High levels of tumor necrosis factor-alpha contribute to accelerated loss of cartilage in diabetic fracture healing.HMGB1 promotes the secretion of multiple cytokines and potentiates the osteogenic differentiation of mesenchymal stem cells through the Ras/MAPK signaling pathway.New insights into hyperglycemia-induced molecular changes in microvascular cells.Diabetes and bone health: latest evidence and clinical implications.

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Diminished bone formation during diabetic fracture healing is related to the premature resorption of cartilage associated with increased osteoclast activity

http://www.wikidata.org/entity/Q35029163

description

2007 nî lūn-bûn

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2007 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2007 թվականի ապրիլին հրատարակված գիտական հոդված

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2007年の論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年论文

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name

Diminished bone formation duri ...... increased osteoclast activity

@ast

Diminished bone formation duri ...... increased osteoclast activity

@en

type

label

Diminished bone formation duri ...... increased osteoclast activity

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Diminished bone formation duri ...... increased osteoclast activity

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prefLabel

Diminished bone formation duri ...... increased osteoclast activity

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Diminished bone formation duri ...... increased osteoclast activity

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Journal of Bone and Mineral Research

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Diminished bone formation duri ...... increased osteoclast activity

@en

P2093

Brian Allen

http://www.w3.org/2001/XMLSchema#string

Cataldo W Leone

http://www.w3.org/2001/XMLSchema#string

Dana T Graves

http://www.w3.org/2001/XMLSchema#string

Dimitris Tsatsas

http://www.w3.org/2001/XMLSchema#string

Elise F Morgan

http://www.w3.org/2001/XMLSchema#string

Maisa O Al-Sebaei

http://www.w3.org/2001/XMLSchema#string

Megan A Bauer

http://www.w3.org/2001/XMLSchema#string

Sanjeev Kakar

http://www.w3.org/2001/XMLSchema#string

P2860

Purification and cloning of aggrecanase-1: a member of the ADAMTS family of proteins

Expression profiling of metalloproteinases and their inhibitors in cartilage

A disintegrin-like and metalloprotease (reprolysin type) with thrombospondin type 1 motifs: the ADAMTS family

GLUT2 in pancreatic islets: crucial target molecule in diabetes induced with multiple low doses of streptozotocin in mice

Alterations of cartilage and collagen expression during fracture healing in experimental diabetes.

Type X collagen in fracture callus and the effects of experimental diabetes.

Growth factor regulation of fracture repair.

Increased bone adiposity and peroxisomal proliferator-activated receptor-gamma2 expression in type I diabetic mice.

Osteoprotegerin is a receptor for the cytotoxic ligand TRAIL.

Fracture healing as a post-natal developmental process: molecular, spatial, and temporal aspects of its regulation.

P304

560-568

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scholarly article

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10.1359/JBMR.070115

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4

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P478

22

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Thomas A Einhorn

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2007-04-01T00:00:00Z

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6559059

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17243865

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P932

3109431

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