Monitoring the T cell response to genital tract infection.
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The Role of the Immune Response in Chlamydia trachomatis Infection of the Male Genital Tract: A Double-Edged SwordDiscovery of the Elusive UDP-Diacylglucosamine Hydrolase in the Lipid A Biosynthetic Pathway in Chlamydia trachomatis.Identification of novel single nucleotide polymorphisms in inflammatory genes as risk factors associated with trachomatous trichiasis.The Role of Non-Cognate T Cell Stimulation during Intracellular Bacterial Infection.Compensatory T cell responses in IRG-deficient mice prevent sustained Chlamydia trachomatis infectionsImmunization with a combination of integral chlamydial antigens and a defined secreted protein induces robust immunity against genital chlamydial challenge.Temporal expression of bacterial proteins instructs host CD4 T cell expansion and Th17 development.CXCR3 and CCR5 are both required for T cell-mediated protection against C. trachomatis infection in the murine genital mucosa.B cells enhance antigen-specific CD4 T cell priming and prevent bacteria dissemination following Chlamydia muridarum genital tract infection.VACCINES. A mucosal vaccine against Chlamydia trachomatis generates two waves of protective memory T cells.Increased susceptibility to Salmonella infection in signal regulatory protein α-deficient mice.Resolution of Chlamydia trachomatis Infection Is Associated with a Distinct T Cell Response Profile.Effects of "Danzhi Decoction" on Chronic Pelvic Pain, Hemodynamics, and Proinflammatory Factors in the Murine Model of Sequelae of Pelvic Inflammatory DiseaseRole of T-cell epitope-based vaccine in prophylactic and therapeutic applications.CD4+ T cells are necessary and sufficient to confer protection against Chlamydia trachomatis infection in the murine upper genital tract.Vaccines for Chlamydia infections of the female genital tract.T cell responses in the absence of IFN-gamma exacerbate uterine infection with Chlamydia trachomatis.Integrin α4β1 is necessary for CD4+ T cell-mediated protection against genital Chlamydia trachomatis infectionSensing the enemy, containing the threat: cell-autonomous immunity to Chlamydia trachomatis.Chlamydia muridarum infection-induced destruction of male germ cells and sertoli cells is partially prevented by Chlamydia major outer membrane protein-specific immune CD4 cells.Immunity against a Chlamydia infection and disease may be determined by a balance of IL-17 signaling.A Chlamydia-Specific TCR-Transgenic Mouse Demonstrates Th1 Polyfunctionality with Enhanced Effector Function.
P2860
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P2860
Monitoring the T cell response to genital tract infection.
description
2006 nî lūn-bûn
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2006 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2006 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2006年の論文
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2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
name
Monitoring the T cell response to genital tract infection.
@ast
Monitoring the T cell response to genital tract infection.
@en
type
label
Monitoring the T cell response to genital tract infection.
@ast
Monitoring the T cell response to genital tract infection.
@en
prefLabel
Monitoring the T cell response to genital tract infection.
@ast
Monitoring the T cell response to genital tract infection.
@en
P2093
P2860
P356
P1476
Monitoring the T cell response to genital tract infection.
@en
P2093
Darren E Higgins
Michael N Starnbach
Nadia R Roan
Todd M Gierahn
P2860
P304
12069-12074
P356
10.1073/PNAS.0603866103
P407
P577
2006-07-31T00:00:00Z