The enzymatic inactivation of the fatty acid amide class of signaling lipids.
about
Endocannabinoid signaling in female reproductionIs lipid signaling through cannabinoid 2 receptors part of a protective system?Glial Modulation by N-acylethanolamides in Brain Injury and NeurodegenerationAlternative targets within the endocannabinoid system for future treatment of gastrointestinal diseasesMarijuana, Spice 'herbal high', and early neural development: implications for rescheduling and legalizationSex differences in cannabinoid pharmacology: a reflection of differences in the endocannabinoid system?Fatty acid amide hydrolase inhibitors: a patent review (2009-2014)Endothelium-dependent metabolism by endocannabinoid hydrolases and cyclooxygenases limits vasorelaxation to anandamide and 2-arachidonoylglycerolVolatile anesthetics and endogenous cannabinoid anandamide have additive and independent inhibitory effects on alpha(7)-nicotinic acetylcholine receptor-mediated responses in Xenopus oocytesEndocannabinoid contribution to Δ9-tetrahydrocannabinol discrimination in rodentsThe endogenous cannabinoid, anandamide, inhibits dopamine transporter function by a receptor-independent mechanism.Developmental trajectory of the endocannabinoid system in human dorsolateral prefrontal cortexIdentification of genetic suppressors of the Sin3A knockdown wing phenotypeAntinociceptive effects of tetrazole inhibitors of endocannabinoid inactivation: cannabinoid and non-cannabinoid receptor-mediated mechanisms.Fatty acid amide hydrolase deficiency limits early pregnancy events.Inhibition of monoacylglycerol lipase and fatty acid amide hydrolase by analogues of 2-arachidonoylglycerolEffects of fatty acid amide hydrolase inhibition on neuronal responses to nicotine, cocaine and morphine in the nucleus accumbens shell and ventral tegmental area: involvement of PPAR-alpha nuclear receptors.Pharmacological blockade of the fatty acid amide hydrolase (FAAH) alters neural proliferation, apoptosis and gliosis in the rat hippocampus, hypothalamus and striatum in a negative energy context.Lasting impacts of prenatal cannabis exposure and the role of endogenous cannabinoids in the developing brain.Lipopolysaccharide-induced murine embryonic resorption involves changes in endocannabinoid profiling and alters progesterone secretion and inflammatory response by a CB1-mediated fashion.Modulation of the endocannabinoid system in viable and non-viable first trimester pregnancies by pregnancy-related hormones.Primary fatty acid amide metabolism: conversion of fatty acids and an ethanolamine in N18TG2 and SCP cellsStructural commonalities among integral membrane enzymes.Endocannabinoid regulation in human endometrium across the menstrual cycleInhibiting fatty acid amide hydrolase normalizes endotoxin-induced enhanced gastrointestinal motility in mice.Silencing or amplification of endocannabinoid signaling in blastocysts via CB1 compromises trophoblast cell migrationAspects of endocannabinoid signaling in periimplantation biologyP2X7 receptors control 2-arachidonoylglycerol production by microglial cells.Modulating the endocannabinoid system in human health and disease--successes and failuresNo effect of an oleoylethanolamide-related phospholipid on satiety and energy intake: a randomised controlled trial of phosphatidylethanolamineFatty acid amide hydrolase (FAAH) inhibition enhances memory acquisition through activation of PPAR-alpha nuclear receptors.Synthetic cannabinoids and potential reproductive consequences.The endogenous cannabinoid system protects against colonic inflammation.Endocannabinoid signaling in the amygdala: anatomy, synaptic signaling, behavior, and adaptations to stress.Targeting the endocannabinoid system for gastrointestinal diseases: future therapeutic strategies.Cannabinoid signaling in health and disease.The mu-opioid receptor agonist morphine, but not agonists at delta- or kappa-opioid receptors, induces peripheral antinociception mediated by cannabinoid receptors.N-cyclohexanecarbonylpentadecylamine: a selective inhibitor of the acid amidase hydrolysing N-acylethanolamines, as a tool to distinguish acid amidase from fatty acid amide hydrolase.Central antinociception induced by mu-opioid receptor agonist morphine, but not delta- or kappa-, is mediated by cannabinoid CB1 receptor.Molecular identification of a functional homologue of the mammalian fatty acid amide hydrolase in Arabidopsis thaliana.
P2860
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P2860
The enzymatic inactivation of the fatty acid amide class of signaling lipids.
description
2002 nî lūn-bûn
@nan
2002 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2002 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2002年の論文
@ja
2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
@wuu
name
The enzymatic inactivation of the fatty acid amide class of signaling lipids.
@ast
The enzymatic inactivation of the fatty acid amide class of signaling lipids.
@en
type
label
The enzymatic inactivation of the fatty acid amide class of signaling lipids.
@ast
The enzymatic inactivation of the fatty acid amide class of signaling lipids.
@en
prefLabel
The enzymatic inactivation of the fatty acid amide class of signaling lipids.
@ast
The enzymatic inactivation of the fatty acid amide class of signaling lipids.
@en
P1476
The enzymatic inactivation of the fatty acid amide class of signaling lipids.
@en
P2093
Aron H Lichtman
P304
P356
10.1016/S0009-3084(02)00147-0
P577
2002-12-01T00:00:00Z