Targeting calcium cycling proteins in heart failure through gene transfer. - Wikidata - awgldk - TriplyDB

Targeting calcium cycling proteins in heart failure through gene transfer.

Targeting calcium cycling proteins in heart failure through gene transfer.

http://www.wikidata.org/entity/Q35037546

about

Regulation of phosphodiesterase 3 and inducible cAMP early repressor in the heart Cytosolic H2O2 mediates hypertrophy, apoptosis, and decreased SERCA activity in mice with chronic hemodynamic overload.Investigations into tropomyosin function using mouse models High-throughput FRET assay yields allosteric SERCA activators Normal and pathological excitation-contraction coupling in the heart -- an overview.Concomitant intravenous nitroglycerin with intracoronary delivery of AAV1.SERCA2a enhances gene transfer in porcine hearts.Chronic heart failure: an overview of conventional treatment versus novel approaches.Role of xanthine oxidoreductase in cardiac nitroso-redox imbalance The Ca2+ ATPase of cardiac sarcoplasmic reticulum: Physiological role and relevance to diseases Striated muscle tropomyosin isoforms differentially regulate cardiac performance and myofilament calcium sensitivity.SERCA2a gene therapy in heart failure: an anti-arrhythmic positive inotrope.Targeted SERCA2a gene expression identifies molecular mechanism and therapeutic target for arrhythmogenic cardiac alternans.The role of CaMKII regulation of phospholamban activity in heart disease.Molecular cardiology in translation: gene, cell and chemical-based experimental therapeutics for the failing heart.Therapeutic potential of genes in cardiac repair.Treatment of heart failure by calcium cycling gene therapy.Effects of levosimendan on cardiac remodeling and cardiomyocyte apoptosis in hypertensive Dahl/Rapp rats.Short-term akt activation in cardiac muscle cells improves contractile function in failing hearts.Levosimendan improves cardiac function and survival in rats with angiotensin II-induced hypertensive heart failure.Genetic manipulation of calcium-handling proteins in cardiac myocytes. II. Mathematical modeling studies.Genetic manipulation of calcium-handling proteins in cardiac myocytes. I. Experimental studies.Heart failure: a silver bullet to treat heart failure.Phenylephrine hypertrophy, Ca2+-ATPase (SERCA2), and Ca2+ signaling in neonatal rat cardiac myocytes.Sarcoplasmic reticulum adenosine triphosphatase overexpression in the L-type Ca2+ channel mouse results in cardiomyopathy and Ca2+ -induced arrhythmogenesis.Sex differences in the response of rat heart ventricle to calcium.Calcium handler mishandles heart.

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Targeting calcium cycling proteins in heart failure through gene transfer.

http://www.wikidata.org/entity/Q35037546

description

2003 nî lūn-bûn

@nan

2003 թուականի Յունուարին հրատարակուած գիտական յօդուած

@hyw

2003 թվականի հունվարին հրատարակված գիտական հոդված

@hy

2003年の論文

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2003年論文

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2003年論文

@zh-hant

2003年論文

@zh-hk

2003年論文

@zh-mo

2003年論文

@zh-tw

2003年论文

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name

Targeting calcium cycling proteins in heart failure through gene transfer.

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Targeting calcium cycling proteins in heart failure through gene transfer.

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Targeting calcium cycling proteins in heart failure through gene transfer.

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Targeting calcium cycling proteins in heart failure through gene transfer.

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Targeting calcium cycling proteins in heart failure through gene transfer.

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Targeting calcium cycling proteins in heart failure through gene transfer.

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JPHYSIOL.2002.026732

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The Journal of Physiology

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Targeting calcium cycling proteins in heart failure through gene transfer.

@en

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Federica del Monte

http://www.w3.org/2001/XMLSchema#string

Roger J Hajjar

http://www.w3.org/2001/XMLSchema#string

P2860

Restoration of contractile function in isolated cardiomyocytes from failing human hearts by gene transfer of SERCA2a.

PKA phosphorylation dissociates FKBP12.6 from the calcium release channel (ryanodine receptor): defective regulation in failing hearts

Expression of a beta-adrenergic receptor kinase 1 inhibitor prevents the development of myocardial failure in gene-targeted mice

Calcium and cardiac rhythms: physiological and pathophysiological

Cardiac excitation-contraction coupling

The failing heart

Improvement in survival and cardiac metabolism after gene transfer of sarcoplasmic reticulum Ca(2+)-ATPase in a rat model of heart failure

Chronic phospholamban-sarcoplasmic reticulum calcium ATPase interaction is the critical calcium cycling defect in dilated cardiomyopathy

Targeted ablation of the phospholamban gene is associated with markedly enhanced myocardial contractility and loss of beta-agonist stimulation

Sarco/endoplasmic reticulum calcium ATPase-2 expression is regulated by ATF6 during the endoplasmic reticulum stress response: intracellular signaling of calcium stress in a cardiac myocyte model system

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49-61

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scholarly article

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10.1113/JPHYSIOL.2002.026732

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Pt 1

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546

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2003-01-01T00:00:00Z

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6073144

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12509478

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2342481

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