Delayed repair of radiation induced clustered DNA damage: friend or foe?
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Novel Biological Approaches for Testing the Contributions of Single DSBs and DSB Clusters to the Biological Effects of High LET RadiationPathways for repairing and tolerating the spectrum of oxidative DNA lesionsDNA double-strand-break complexity levels and their possible contributions to the probability for error-prone processing and repair pathway choiceComments on potential health effects of MRI-induced DNA lesions: quality is more important to consider than quantityDNA Processing Proteins Involved in the UV-Induced Stress Response of SulfolobalesThe efficiency of homologous recombination and non-homologous end joining systems in repairing double-strand breaks during cell cycle progressionMechanisms and Consequences of Double-Strand DNA Break Formation in ChromatinA branching process model for the analysis of abortive colony size distributions in carbon ion-irradiated normal human fibroblasts.Interstrand cross-links arising from strand breaks at true abasic sites in duplex DNA.Nucleosomes suppress the formation of double-strand DNA breaks during attempted base excision repair of clustered oxidative damagesMechanisms of free radical-induced damage to DNA.⁹⁹mTc pyrene derivative complex causes double-strand breaks in dsDNA mainly through cluster-mediated indirect effect in aqueous solution.Requirements for 5'dRP/AP lyase activity in Ku.The potential and hurdles of targeted alpha therapy - clinical trials and beyond.Spatiotemporal dynamics of DNA repair proteins following laser microbeam induced DNA damage - when is a DSB not a DSB?The genome-wide effects of ionizing radiation on mutation induction in the mammalian germlineStress-induced DNA damage biomarkers: applications and limitations.Effects of indirect actions and oxygen on relative biological effectiveness: estimate of DSB induction and conversion induced by gamma rays and helium ions.Efficient cleavage of single and clustered AP site lesions within mono-nucleosome templates by CHO-K1 nuclear extract contrasts with retardation of incision by purified APE1.UVA-induced DNA double-strand breaks result from the repair of clustered oxidative DNA damages.Long-term Autophagy and Nrf2 Signaling in the Hippocampi of Developing Mice after Carbon Ion Exposure.Effects of expression level of DNA repair-related genes involved in the NHEJ pathway on radiation-induced cognitive impairment.The shape of the radiation dose response for DNA double-strand break induction and repair.Harnessing mutagenic homologous recombination for targeted mutagenesis in vivo by TaGTEAM.Cultivation of Staphylococcus epidermidis in the Human Spaceflight Environment Leads to Alterations in the Frequency and Spectrum of Spontaneous Rifampicin-Resistance Mutations in the rpoB Genep53 Ser15 phosphorylation and histone modifications contribute to IR-induced miR-34a transcription in mammary epithelial cells.Replication fork collapse is a major cause of the high mutation frequency at three-base lesion clustersCorrelation of bistranded clustered abasic DNA lesion processing with structural and dynamic DNA helix distortion.Scaffold attachment factor A (SAF-A) and Ku temporally regulate repair of radiation-induced clustered genome lesionsReduced repair capacity of a DNA clustered damage site comprised of 8-oxo-7,8-dihydro-2'-deoxyguanosine and 2-deoxyribonolactone results in an increased mutagenic potential of these lesions.Reversion of the ErbB malignant phenotype and the DNA damage response.Autophagy and genomic integrity.Current and emerging strategies to increase the efficacy of ionizing radiation in the treatment of cancer.Radiation-mediated formation of complex damage to DNA: a chemical aspect overview.Radiobiological modifiers in clinical radiation oncology: current reality and future potential.Complex DNA Damage: A Route to Radiation-Induced Genomic Instability and Carcinogenesis.Excess processing of oxidative damaged bases causes hypersensitivity to oxidative stress and low dose rate irradiation.The major DNA repair pathway after both proton and carbon-ion radiation is NHEJ, but the HR pathway is more relevant in carbon ions.Increased mutability and decreased repairability of a three-lesion clustered DNA-damaged site comprised of an AP site and bi-stranded 8-oxoG lesions.Effects of low-dose capecitabine on Samarium-153-EDTMP therapy for painful bone metastases
P2860
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P2860
Delayed repair of radiation induced clustered DNA damage: friend or foe?
description
2010 nî lūn-bûn
@nan
2010 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Delayed repair of radiation induced clustered DNA damage: friend or foe?
@ast
Delayed repair of radiation induced clustered DNA damage: friend or foe?
@en
type
label
Delayed repair of radiation induced clustered DNA damage: friend or foe?
@ast
Delayed repair of radiation induced clustered DNA damage: friend or foe?
@en
prefLabel
Delayed repair of radiation induced clustered DNA damage: friend or foe?
@ast
Delayed repair of radiation induced clustered DNA damage: friend or foe?
@en
P2093
P2860
P1433
P1476
Delayed repair of radiation induced clustered DNA damage: friend or foe?
@en
P2093
Laura J Eccles
Martine E Lomax
Peter O'Neill
P2860
P304
P356
10.1016/J.MRFMMM.2010.11.003
P407
P577
2010-12-02T00:00:00Z