Improvement of cardiac functions by chronic metformin treatment is associated with enhanced cardiac autophagy in diabetic OVE26 mice - Wikidata - awgldk - TriplyDB

Improvement of cardiac functions by chronic metformin treatment is associated with enhanced cardiac autophagy in diabetic OVE26 mice

Improvement of cardiac functions by chronic metformin treatment is associated with enhanced cardiac autophagy in diabetic OVE26 mice

http://www.wikidata.org/entity/Q35043474

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AMP-activated protein kinase, stress responses and cardiovascular diseases Novel nervous and multi-system regenerative therapeutic strategies for diabetes mellitus with mTOR Insulin resistance and hyperinsulinaemia in diabetic cardiomyopathy Erythropoietin and diabetes mellitus New mechanisms of metformin action: Focusing on mitochondria and the gut Animal models of diabetic macrovascular complications: key players in the development of new therapeutic approaches Use of metformin in diseases of aging Therapeutic targeting of autophagy: potential and concerns in treating cardiovascular disease Cardiovascular autophagy: crossroads of pathology, pharmacology and toxicology Past strategies and future directions for identifying AMP-activated protein kinase (AMPK) modulators Autophagy in cardiovascular biology Myocardial autophagic energy stress responses--macroautophagy, mitophagy, and glycophagy Interplay of oxidative, nitrosative/nitrative stress, inflammation, cell death and autophagy in diabetic cardiomyopathy Cardiac dysfunction and oxidative stress in the metabolic syndrome: an update on antioxidant therapies Congestive heart failure and diabetes mellitus: balancing glycemic control with heart failure improvement Heart failure and mitochondrial dysfunction: the role of mitochondrial fission/fusion abnormalities and new therapeutic strategies Mitochondria and cardiovascular aging Cellular and molecular mechanisms of metformin: an overview New promises for metformin: advances in the understanding of its mechanisms of action Erythropoietin and mTOR: A "One-Two Punch" for Aging-Related Disorders Accompanied by Enhanced Life Expectancy New Insights for Oxidative Stress and Diabetes Mellitus The Use of SGLT-2 Inhibitors in Type 2 Diabetes and Heart Failure Ischemic conditioning-induced endogenous brain protection: Applications pre-, per- or post-stroke Mitochondrial function in vascular endothelial cell in diabetes Mitochondrial hormesis and diabetic complications Prenatal metformin exposure in mice programs the metabolic phenotype of the offspring during a high fat diet at adulthood Heme oxygenase-1 prevents cardiac dysfunction in streptozotocin-diabetic mice by reducing inflammation, oxidative stress, apoptosis and enhancing autophagy Role of autophagy in metabolic syndrome-associated heart disease.Mir30c Is Involved in Diabetic Cardiomyopathy through Regulation of Cardiac Autophagy via BECN1 BRD7 mediates hyperglycaemia-induced myocardial apoptosis via endoplasmic reticulum stress signalling pathway.Diabetic cardiomyopathy: where we are and where we are going.Calcineurin suppresses AMPK-dependent cytoprotective autophagy in cardiomyocytes under oxidative stress.Metabolic and Biochemical Stressors in Diabetic Cardiomyopathy.Acute metformin preconditioning confers neuroprotection against focal cerebral ischaemia by pre-activation of AMPK-dependent autophagy.Quercetin alleviates high glucose-induced Schwann cell damage by autophagy.Resveratrol-enhanced autophagic flux ameliorates myocardial oxidative stress injury in diabetic mice.PRKAA1/AMPKα1 is required for autophagy-dependent mitochondrial clearance during erythrocyte maturation Autophagy and oxidative stress in cardiovascular diseases.Prenatal metformin exposure in a maternal high fat diet mouse model alters the transcriptome and modifies the metabolic responses of the offspring.Targeted deletion of PTEN in cardiomyocytes renders cardiac contractile dysfunction through interruption of Pink1-AMPK signaling and autophagy

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Improvement of cardiac functions by chronic metformin treatment is associated with enhanced cardiac autophagy in diabetic OVE26 mice

http://www.wikidata.org/entity/Q35043474

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Chaoyong He

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David Kem

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Hongliang Li

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Kai Lau

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Ming-Hui Zou

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Pedro Lozano

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Shradha Rathi

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Yunzhou Dong

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P2860

Metformin: An Update

Role of AMP-activated protein kinase in mechanism of metformin action

Association of glycaemia with macrovascular and microvascular complications of type 2 diabetes (UKPDS 35): prospective observational study

Upregulation of mitochondrial uncoupling protein-2 by the AMP-activated protein kinase in endothelial cells attenuates oxidative stress in diabetes

Selective degradation of mitochondria by mitophagy

TSC2 mediates cellular energy response to control cell growth and survival

Induction of autophagy and inhibition of tumorigenesis by beclin 1

The role of autophagy in cardiomyocytes in the basal state and in response to hemodynamic stress

Bax translocates to mitochondria of heart cells during simulated ischaemia: involvement of AMP-activated and p38 mitogen-activated protein kinases

Effect of intensive blood-glucose control with metformin on complications in overweight patients with type 2 diabetes (UKPDS 34). UK Prospective Diabetes Study (UKPDS) Group

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