The preferential beta3-adrenoceptor agonist BRL 37344 increases force via beta1-/beta2-adrenoceptors and induces endothelial nitric oxide synthase via beta3-adrenoceptors in human atrial myocardium.
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Beta 3-adrenoreceptor regulation of nitric oxide in the cardiovascular system.Are there functional β₃-adrenoceptors in the human heart?Human atrial β(1L)-adrenoceptor but not β₃-adrenoceptor activation increases force and Ca(2+) current at physiological temperature.Mechanisms of beta 3-adrenoceptor-induced eNOS activation in right atrial and left ventricular human myocardiumStress and cardiac beta adrenoceptors.beta3-adrenergic receptor activation increases human atrial tissue contractility and stimulates the L-type Ca2+ currentβ3 adrenergic receptor in the kidney may be a new player in sympathetic regulation of renal functionCardio-selective beta-blocker: pharmacological evidence and their influence on exercise capacity.Infantile hemangiomas, retinopathy of prematurity and cancer: a common pathogenetic role of the β-adrenergic system.β3-adrenergic receptor activity modulates melanoma cell proliferation and survival through nitric oxide signaling.Tissue functions mediated by beta(3)-adrenoceptors-findings and challenges.Mitochondriocentric pathway to cardiomyocyte necrosis in aldosteronism: cardioprotective responses to carvedilol and nebivolol.eNOS phosphorylation and translocation are altered in male but not female mice by increased activation of the Gαq protein.Beta3-adrenergic receptors modulate vascular endothelial growth factor release in response to hypoxia through the nitric oxide pathway in mouse retinal explants.Direct evidence of nitric oxide release from neuronal nitric oxide synthase activation in the left ventricle as a result of cervical vagus nerve stimulation.Response by Simpson et al to Letter Regarding Article, "Adrenergic Receptors in Individual Ventricular Myocytes: the Beta-1 and Alpha-1B Are in All Cells, the Alpha-1A Is in a Subpopulation, and the Beta-2 and Beta-3 Are Mostly Absent".Rabbit, a relevant model for the study of cardiac beta 3-adrenoceptors.eNOS translocation but not eNOS phosphorylation is dependent on intracellular Ca2+ in human atrial myocardium.The effect of combined treatment with a β3 AR agonist and a ROCK inhibitor on detrusor overactivity.Chronic bilateral renal denervation reduces cardiac hypertrophic remodelling but not β-adrenergic responsiveness in hypertensive type 1 diabetic rats.Phosphorylation of myocardial eNOS is altered in patients suffering from type 2 diabetes.
P2860
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P2860
The preferential beta3-adrenoceptor agonist BRL 37344 increases force via beta1-/beta2-adrenoceptors and induces endothelial nitric oxide synthase via beta3-adrenoceptors in human atrial myocardium.
description
2003 nî lūn-bûn
@nan
2003 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
name
The preferential beta3-adrenoc ...... rs in human atrial myocardium.
@en
type
label
The preferential beta3-adrenoc ...... rs in human atrial myocardium.
@en
prefLabel
The preferential beta3-adrenoc ...... rs in human atrial myocardium.
@en
P2093
P2860
P356
P1476
The preferential beta3-adrenoc ...... rs in human atrial myocardium.
@en
P2093
A Bundkirchen
D Steinritz
R H G Schwinger
U Mehlhorn
P2860
P304
P356
10.1038/SJ.BJP.0705065
P407
P577
2003-02-01T00:00:00Z