Chronic NGF treatment of rat nociceptive DRG neurons in culture facilitates desensitization and deactivation of GABAA receptor-mediated currents.
about
Silencing the α2 subunit of γ-aminobutyric acid type A receptors in rat dorsal root ganglia reveals its major role in antinociception posttraumatic nerve injury.B-type natriuretic peptide-induced delayed modulation of TRPV1 and P2X3 receptors of mouse trigeminal sensory neurons.Inflammation-induced shift in spinal GABA(A) signaling is associated with a tyrosine kinase-dependent increase in GABA(A) current density in nociceptive afferents.
P2860
Chronic NGF treatment of rat nociceptive DRG neurons in culture facilitates desensitization and deactivation of GABAA receptor-mediated currents.
description
2004 nî lūn-bûn
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2004 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2004 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2004年の論文
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2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
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name
Chronic NGF treatment of rat n ...... AA receptor-mediated currents.
@ast
Chronic NGF treatment of rat n ...... AA receptor-mediated currents.
@en
type
label
Chronic NGF treatment of rat n ...... AA receptor-mediated currents.
@ast
Chronic NGF treatment of rat n ...... AA receptor-mediated currents.
@en
prefLabel
Chronic NGF treatment of rat n ...... AA receptor-mediated currents.
@ast
Chronic NGF treatment of rat n ...... AA receptor-mediated currents.
@en
P2860
P356
P1476
Chronic NGF treatment of rat n ...... AA receptor-mediated currents.
@en
P2093
Alessandra Fabbro
Andrea Nistri
P2860
P304
P356
10.1038/SJ.BJP.0705813
P407
P577
2004-05-17T00:00:00Z