DDAH1 deficiency attenuates endothelial cell cycle progression and angiogenesis.
about
A RASopathy gene commonly mutated in cancer: the neurofibromatosis type 1 tumour suppressorPlasmodium Infection Is Associated with Impaired Hepatic Dimethylarginine Dimethylaminohydrolase Activity and Disruption of Nitric Oxide Synthase Inhibitor/Substrate HomeostasisEffect of asymmetric dimethylarginine (ADMA) on heart failure development.Identification of key pathways and genes influencing prognosis in bladder urothelial carcinomaA new key player in VEGF-dependent angiogenesis in human hepatocellular carcinoma: dimethylarginine dimethylaminohydrolase 1.Cardiomyocyte dimethylarginine dimethylaminohydrolase-1 (DDAH1) plays an important role in attenuating ventricular hypertrophy and dysfunction.MiR-193b regulates breast cancer cell migration and vasculogenic mimicry by targeting dimethylarginine dimethylaminohydrolase 1.Chitosan oligosaccharide ameliorates acute lung injury induced by blast injury through the DDAH1/ADMA pathway.
P2860
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P2860
DDAH1 deficiency attenuates endothelial cell cycle progression and angiogenesis.
description
2013 nî lūn-bûn
@nan
2013 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2013 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
name
DDAH1 deficiency attenuates endothelial cell cycle progression and angiogenesis.
@en
type
label
DDAH1 deficiency attenuates endothelial cell cycle progression and angiogenesis.
@en
prefLabel
DDAH1 deficiency attenuates endothelial cell cycle progression and angiogenesis.
@en
P2093
P2860
P1433
P1476
DDAH1 deficiency attenuates endothelial cell cycle progression and angiogenesis.
@en
P2093
John Fassett
Ping Zhang
Robert J Bache
Yuqing Huo
P2860
P304
P356
10.1371/JOURNAL.PONE.0079444
P407
P50
P577
2013-11-18T00:00:00Z