Cigarette smoke extract suppresses the RIG-I-initiated innate immune response to influenza virus in the human lung.
about
Toxic elements in tobacco and in cigarette smoke: inflammation and sensitizationTargeting PI3K-p110α Suppresses Influenza Virus Infection in Chronic Obstructive Pulmonary Disease.Influenza A(H1N1)pdm09 virus suppresses RIG-I initiated innate antiviral responses in the human lung.Factors associated with post-seasonal serological titer and risk factors for infection with the pandemic A/H1N1 virus in the French general population.Cigarette smoke exposure exacerbates lung inflammation and compromises immunity to bacterial infection.Cigarette smoke attenuates the RIG-I-initiated innate antiviral response to influenza infection in two murine models.CD206+ cell number differentiates influenza A (H1N1)pdm09 from seasonal influenza A virus in fatal cases.COPD: balancing oxidants and antioxidants.Activation of the UPR protects against cigarette smoke-induced RPE apoptosis through up-regulation of Nrf2.RIG-I and TLR3 are both required for maximum interferon induction by influenza virus in human lung alveolar epithelial cells.Alteration of the nasal responses to influenza virus by tobacco smokeDNA methylation in nasal epithelial cells from smokers: identification of ULBP3-related effects.Human primary airway epithelial cells isolated from active smokers have epigenetically impaired antiviral responsesCigarette smoke enhances human rhinovirus-induced CXCL8 production via HuR-mediated mRNA stabilization in human airway epithelial cells.Innate immunity to influenza in chronic airways diseases.The inflammasome in lung diseasesNovel concepts of acute lung injury and alveolar-capillary barrier dysfunction.Rhinovirus-Induced Exacerbations of Asthma and COPD.Update on the pathological processes, molecular biology, and clinical utility of N-acetylcysteine in chronic obstructive pulmonary disease.Human lung ex vivo infection models.MCPIP1 attenuates the innate immune response to influenza A viruse by suppressing RIG-I expression in lung epithelial cells.RIG-I overexpression decreases mortality of cigarette smoke exposed mice during influenza A virus infection.Impaired Antiviral Stress Granule and IFN-β Enhanceosome Formation Enhances Susceptibility to Influenza Infection in Chronic Obstructive Pulmonary Disease Epithelium.Critical role of RIG-I-like receptors in inflammation in chronic obstructive pulmonary disease.Secondhand Smoke Induces Inflammation and Impairs Immunity to Respiratory Infections.
P2860
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P2860
Cigarette smoke extract suppresses the RIG-I-initiated innate immune response to influenza virus in the human lung.
description
2011 nî lūn-bûn
@nan
2011 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Cigarette smoke extract suppre ...... uenza virus in the human lung.
@ast
Cigarette smoke extract suppre ...... uenza virus in the human lung.
@en
type
label
Cigarette smoke extract suppre ...... uenza virus in the human lung.
@ast
Cigarette smoke extract suppre ...... uenza virus in the human lung.
@en
prefLabel
Cigarette smoke extract suppre ...... uenza virus in the human lung.
@ast
Cigarette smoke extract suppre ...... uenza virus in the human lung.
@en
P2093
P2860
P1476
Cigarette smoke extract suppre ...... uenza virus in the human lung.
@en
P2093
J Leland Booth
Jordan P Metcalf
Krupa B Patel
P2860
P304
P356
10.1152/AJPLUNG.00267.2010
P577
2011-02-18T00:00:00Z