Early deficits in glycolysis are specific to striatal neurons from a rat model of huntington disease. - Wikidata - awgldk - TriplyDB

Early deficits in glycolysis are specific to striatal neurons from a rat model of huntington disease.

Early deficits in glycolysis are specific to striatal neurons from a rat model of huntington disease.

http://www.wikidata.org/entity/Q35058593

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The chicken or the egg: mitochondrial dysfunction as a cause or consequence of toxicity in Huntington's disease Enhanced neuronal glucose transporter expression reveals metabolic choice in a HD Drosophila model Oxygen consumption deficit in Huntington disease mouse brain under metabolic stress Neuronal Mitophagy in Neurodegenerative Diseases Oxidative metabolism in YAC128 mouse model of Huntington's disease.Differential expression of glucose-metabolizing enzymes in multiple sclerosis lesions.The phasor-FLIM fingerprints reveal shifts from OXPHOS to enhanced glycolysis in Huntington Disease The Association of VDAC with Cell Viability of PC12 Model of Huntington's Disease.Oxidative metabolism and Ca2+ handling in isolated brain mitochondria and striatal neurons from R6/2 mice, a model of Huntington's disease.Lactate Shuttles in Neuroenergetics-Homeostasis, Allostasis and Beyond.Mutant Huntingtin and Elusive Defects in Oxidative Metabolism and Mitochondrial Calcium Handling.AMPK in Neurodegenerative Diseases.Mitochondrial membrane fluidity is consistently increased in different models of Huntington disease: restorative effects of olesoxime.Oxidative metabolism and Ca2+ handling in striatal mitochondria from YAC128 mice, a model of Huntington's disease.Manganese and the Insulin-IGF Signaling Network in Huntington's Disease and Other Neurodegenerative Disorders.Towards an Understanding of Energy Impairment in Huntington's Disease Brain.

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P2860

Early deficits in glycolysis are specific to striatal neurons from a rat model of huntington disease.

http://www.wikidata.org/entity/Q35058593

description

2013 nî lūn-bûn

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2013 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

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2013 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2013年の論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年论文

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name

Early deficits in glycolysis a ...... t model of huntington disease.

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Early deficits in glycolysis a ...... t model of huntington disease.

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JOURNAL.PONE.0081528

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Early deficits in glycolysis a ...... t model of huntington disease.

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Caroline Gouarné

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Gwenaëlle Tardif

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Jennifer Tracz

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Laura Emily Clemens

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Magali Michaud

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Rebecca M Pruss

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Thierry Bordet

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Virginie Latyszenok

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P2860

Vesicular glycolysis provides on-board energy for fast axonal transport

Functional brain abnormalities in young adults at genetic risk for late-onset Alzheimer's dementia

Glutamate uptake into astrocytes stimulates aerobic glycolysis: a mechanism coupling neuronal activity to glucose utilization

Cellular mechanisms of brain energy metabolism and their relevance to functional brain imaging.

Clinical correlation of striatal 1H MRS changes in Huntington's disease.

Heterogeneity in 1H-MRS profiles of presymptomatic and early manifest Huntington's disease.

Increased metabolism in the R6/2 mouse model of Huntington's disease.

Differential vulnerability of neurons in Huntington's disease: the role of cell type-specific features.

Glucose-sensing neurons: are they physiologically relevant?

Metabolic state determines sensitivity to cellular stress in Huntington disease: normalization by activation of PPARγ.

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e81528

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10.1371/JOURNAL.PONE.0081528

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11

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Huu Phuc Nguyen

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2013-11-26T00:00:00Z

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259156068

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corpus striatum

Huntington disease

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3841140

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