Early elevation in random plasma IL-6 after severe injury is associated with development of organ failure - Wikidata - awgldk - TriplyDB

Early elevation in random plasma IL-6 after severe injury is associated with development of organ failure

Early elevation in random plasma IL-6 after severe injury is associated with development of organ failure

http://www.wikidata.org/entity/Q35065087

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The diagnostic and prognostic value of systems biology research in major traumatic and thermal injury: a review Deferoxamine attenuates lipid peroxidation, blocks interleukin-6 production, ameliorates sepsis inflammatory response syndrome, and confers renoprotection after acute hepatic ischemia in pigs.Delayed neutralization of interleukin 6 reduces organ injury, selectively suppresses inflammatory mediator, and partially normalizes immune dysfunction following trauma and hemorrhagic shock.X chromosome-linked IRAK-1 polymorphism is a strong predictor of multiple organ failure and mortality postinjury.Innate immune function predicts the development of nosocomial infection in critically injured children.Role of the mTOR pathway in LPS-activated monocytes: influence of hypertonic saline Lymph is not a plasma ultrafiltrate: a proteomic analysis of injured patients The changing pattern and implications of multiple organ failure after blunt injury with hemorrhagic shock.Persistence of elevated plasma CXCL8 concentrations following red blood cell transfusion in a trauma cohort.Predictive value of cytokines for developing complications after polytrauma.Gene Expression of Proresolving Lipid Mediator Pathways Is Associated With Clinical Outcomes in Trauma Patients.Acute kidney injury is associated with early cytokine changes after trauma Impact of the body mass on complications and outcome in multiple trauma patients: what does the weight weigh?Elevated Admission Base Deficit Is Associated with a Complex Dynamic Network of Systemic Inflammation Which Drives Clinical Trajectories in Blunt Trauma Patients.Temporal Patterns of Circulating Inflammation Biomarker Networks Differentiate Susceptibility to Nosocomial Infection Following Blunt Trauma in Humans.Interleukin-6 as inflammatory marker referring to multiple organ dysfunction syndrome in severely injured children.Prehospital immune responses and development of multiple organ dysfunction syndrome following traumatic injury: A prospective cohort study.Individual-specific principal component analysis of circulating inflammatory mediators predicts early organ dysfunction in trauma patients.Comparison of the acute-phase response after laparoscopic versus open aortobifemoral bypass surgery: a substudy of a randomized controlled trial.Association between increased blood interleukin-6 levels on emergency department arrival and prolonged length of intensive care unit stay for blunt trauma The ubiquitous interleukin-6: a time for reappraisal.Innate immune response to pulmonary contusion: identification of cell type-specific inflammatory responses An overview of cytokines and heat shock response in polytraumatized patients.Using IL-6 concentrations in the first 24 h following trauma to predict immunological complications and mortality in trauma patients: a meta-analysis.The grapes and wrath: using resveratrol to treat the pathophysiology of hemorrhagic shock.Acute inflammation in young children inhibits C-type natriuretic peptide.Pharmacological inhibition of IL-6 trans-signaling improves compromised fracture healing after severe trauma.

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Early elevation in random plasma IL-6 after severe injury is associated with development of organ failure

http://www.wikidata.org/entity/Q35065087

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2010 nî lūn-bûn

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2010 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Early elevation in random plas ...... h development of organ failure

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Early elevation in random plas ...... h development of organ failure

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Early elevation in random plas ...... h development of organ failure

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Eileen Bulger

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Ernest E Moore

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Inflammation and the Host Response to Injury Collaborative Research Program

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Blood substitutes: the future is now

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Daniel G Remick

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