Excessive neutrophils and neutrophil extracellular traps contribute to acute lung injury of influenza pneumonitis. - Wikidata - awgldk - TriplyDB

Excessive neutrophils and neutrophil extracellular traps contribute to acute lung injury of influenza pneumonitis.

Excessive neutrophils and neutrophil extracellular traps contribute to acute lung injury of influenza pneumonitis.

http://www.wikidata.org/entity/Q35070188

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CD11c(+)/CD11b(+) cells are critical for organic dust-elicited murine lung inflammation Neutrophil Extracellular Traps in Pulmonary Diseases: Too Much of a Good Thing?Far beyond Phagocytosis: Phagocyte-Derived Extracellular Traps Act Efficiently against Protozoan Parasites In Vitro and In Vivo Platelets in Pulmonary Immune Responses and Inflammatory Lung Diseases Diagnostic value of plasma and bronchoalveolar lavage samples in acute lung allograft rejection: differential cytology How Neutrophil Extracellular Traps Become Visible Key mechanisms governing resolution of lung inflammation Interaction of Bacterial Exotoxins with Neutrophil Extracellular Traps: Impact for the Infected Host How Neutrophils Shape Adaptive Immune Responses Influenza and Bacterial Superinfection: Illuminating the Immunologic Mechanisms of Disease Adult lung stem cells and their contribution to lung tumourigenesis Neutrophil Extracellular Traps Induce Organ Damage during Experimental and Clinical Sepsis Protective immunity and safety of a genetically modified influenza virus vaccine Capillary plexuses are vulnerable to neutrophil extracellular traps Inflection points in sepsis biology: from local defense to systemic organ injury Modulation of neutrophil NETosis: interplay between infectious agents and underlying host physiology.NETosis in Cancer - Platelet-Neutrophil Crosstalk Promotes Tumor-Associated Pathology Neutrophil Extracellular Traps Go Viral The Role of Antimicrobial Peptides in Influenza Virus Infection and Their Potential as Antiviral and Immunomodulatory Therapy The immunology of influenza virus-associated bacterial pneumonia Pneumovirus-Induced Lung Disease in Mice Is Independent of Neutrophil-Driven Inflammation The acute respiratory distress syndrome: from mechanism to translation Virus-infected alveolar epithelial cells direct neutrophil chemotaxis and inhibit their apoptosis A Lipid Mediator Hepoxilin A3 Is a Natural Inducer of Neutrophil Extracellular Traps in Human Neutrophils A Role for Neutrophils in Viral Respiratory Disease.Viral pathogens and acute lung injury: investigations inspired by the SARS epidemic and the 2009 H1N1 influenza pandemic Switch from protective to adverse inflammation during influenza: viral determinants and hemostasis are caught as culprits.Bordetella parapertussis Circumvents Neutrophil Extracellular Bactericidal Mechanisms.Innate immune sensing and response to influenza.Swine Influenza Virus PA and Neuraminidase Gene Reassortment into Human H1N1 Influenza Virus Is Associated with an Altered Pathogenic Phenotype Linked to Increased MIP-2 Expression The Tie2-agonist Vasculotide rescues mice from influenza virus infection Lower Respiratory Tract Infection of the Ferret by 2009 H1N1 Pandemic Influenza A Virus Triggers Biphasic, Systemic, and Local Recruitment of Neutrophils.Arginine-rich histones have strong antiviral activity for influenza A viruses Constitutively Expressed IFITM3 Protein in Human Endothelial Cells Poses an Early Infection Block to Human Influenza Viruses.A De-O-acylated Lipooligosaccharide-Based Adjuvant System Promotes Antibody and Th1-Type Immune Responses to H1N1 Pandemic Influenza Vaccine in Mice Influenza and dengue virus co-infection impairs monocyte recruitment to the lung, increases dengue virus titers, and exacerbates pneumonia.Differential pathological and immune responses in newly weaned ferrets are associated with a mild clinical outcome of pandemic 2009 H1N1 infection.Clinical factors predictive of pneumonia caused by pandemic 2009 H1N1 influenza virus.Complement C5 activation during influenza A infection in mice contributes to neutrophil recruitment and lung injury.ETosis: A Microbicidal Mechanism beyond Cell Death.

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P2860

Excessive neutrophils and neutrophil extracellular traps contribute to acute lung injury of influenza pneumonitis.

http://www.wikidata.org/entity/Q35070188

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2011 nî lūn-bûn

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2011 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2011年の論文

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Excessive neutrophils and neut ...... jury of influenza pneumonitis.

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Excessive neutrophils and neut ...... jury of influenza pneumonitis.

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Excessive neutrophils and neut ...... jury of influenza pneumonitis.

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J.AJPATH.2011.03.013

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The American Journal of Pathology

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Excessive neutrophils and neut ...... jury of influenza pneumonitis.

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P2093

Audrey-Ann Liew

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Edwin Yang

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Huey Hian Ng

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Meng Chee Phoon

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Ramar Perumal Samy

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Teluguakula Narasaraju

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Vincent T Chow

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P2860

Neutrophil extracellular traps kill bacteria

Avian influenza A (H5N1) infection in humans

Characterization of the reconstructed 1918 Spanish influenza pandemic virus

Aberrant innate immune response in lethal infection of macaques with the 1918 influenza virus

Platelet TLR4 activates neutrophil extracellular traps to ensnare bacteria in septic blood

Netting neutrophils in autoimmune small-vessel vasculitis

Novel cell death program leads to neutrophil extracellular traps

Enhanced virulence of influenza A viruses with the haemagglutinin of the 1918 pandemic virus.

H5N1 and 1918 pandemic influenza virus infection results in early and excessive infiltration of macrophages and neutrophils in the lungs of mice.

Adaptation of human influenza H3N2 virus in a mouse pneumonitis model: insights into viral virulence, tissue tropism and host pathogenesis.

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