Inflammation induced by infection potentiates tau pathological features in transgenic mice. - Wikidata - awgldk - TriplyDB

Inflammation induced by infection potentiates tau pathological features in transgenic mice.

Inflammation induced by infection potentiates tau pathological features in transgenic mice.

http://www.wikidata.org/entity/Q35071072

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Therapeutic potential of mood stabilizers lithium and valproic acid: beyond bipolar disorder Microglial Priming and Alzheimer's Disease: A Possible Role for (Early) Immune Challenges and Epigenetics?Herpes Simplex Virus Type 1 and Other Pathogens are Key Causative Factors in Sporadic Alzheimer's Disease Ameliorative Effects of Antioxidants on the Hippocampal Accumulation of Pathologic Tau in a Rat Model of Blast-Induced Traumatic Brain Injury Autoimmune manifestations in the 3xTg-AD model of Alzheimer's disease.Gene knockout of 5-lipoxygenase rescues synaptic dysfunction and improves memory in the triple-transgenic model of Alzheimer's disease.Herpes simplex virus type 1 and Alzheimer's disease: increasing evidence for a major role of the virus.Microglia in Alzheimer's disease The role of tau protein in HIV-associated neurocognitive disorders Inflammasomes as therapeutic targets for Alzheimer's disease.Models of β-amyloid induced Tau-pathology: the long and "folded" road to understand the mechanism.Atherosclerosis and Alzheimer--diseases with a common cause? Inflammation, oxysterols, vasculature Molecular mechanisms responsible for the selective and low-grade induction of proinflammatory mediators in murine macrophages by lipopolysaccharide Exendin-4 protected against cognitive dysfunction in hyperglycemic mice receiving an intrahippocampal lipopolysaccharide injection.Neuronal Cell Death and Degeneration through Increased Nitroxidative Stress and Tau Phosphorylation in HIV-1 Transgenic Rats.Chronic lithium feeding reduces upregulated brain arachidonic acid metabolism in HIV-1 transgenic rat.Can infections cause Alzheimer's disease?Molecular mechanisms responsible for the reduced expression of cholesterol transporters from macrophages by low-dose endotoxin Lithium and neuroprotection: translational evidence and implications for the treatment of neuropsychiatric disorders.LPS-induced Murine Neuroinflammation Model: Main Features and Suitability for Pre-clinical Assessment of Nutraceuticals Hydrogen sulfide protects against amyloid beta-peptide induced neuronal injury via attenuating inflammatory responses in a rat model.Systemic and localized extra-central nervous system bacterial infections and the risk of dementia among US veterans: A retrospective cohort study.Modulation of lipopolysaccharide-induced memory insult, γ-secretase, and neuroinflammation in triple transgenic mice by 5-lipoxygenase.Synthesis and biological evaluation studies of novel quinazolinone derivatives as antibacterial and anti-inflammatory agents Enhanced susceptibility of triple transgenic Alzheimer's disease (3xTg-AD) mice to acute infection.Modelling early responses to neurodegenerative mutations in mice.Glycogen synthase kinase-3 inhibitors: Rescuers of cognitive impairments.Modulation of inflammation in transgenic models of Alzheimer's disease Innate immunity in Alzheimer's disease.The Role of PGRN in Alzheimer's Disease.Involvement of inflammation in Alzheimer's disease pathogenesis and therapeutic potential of anti-inflammatory agents.LISPRO mitigates β-amyloid and associated pathologies in Alzheimer's mice.Stressed and Inflamed, Can GSK3 Be Blamed?A state of delirium: Deciphering the effect of inflammation on tau pathology in Alzheimer's disease.The olfactory bulb as the entry site for prion-like propagation in neurodegenerative diseases.The Role of Microglia in the Etiology and Evolution of Chronic Traumatic Encephalopathy.No support for premature central nervous system aging in HIV-1 when measured by cerebrospinal fluid phosphorylated tau (p-tau).IFN-γ promotes τ phosphorylation without affecting mature tangles.Microglia function in Alzheimer's disease.Progranulin reduction is associated with increased tau phosphorylation in P301L tau transgenic mice.

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Inflammation induced by infection potentiates tau pathological features in transgenic mice.

http://www.wikidata.org/entity/Q35071072

description

2011 nî lūn-bûn

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2011 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի ապրիլին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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Inflammation induced by infect ...... l features in transgenic mice.

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Inflammation induced by infect ...... l features in transgenic mice.

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Inflammation induced by infect ...... l features in transgenic mice.

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Inflammation induced by infect ...... l features in transgenic mice.

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J.AJPATH.2011.02.012

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The American Journal of Pathology

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Inflammation induced by infect ...... l features in transgenic mice.

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David Cheng

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Frank M Laferla

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Lucia Whitman

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Masashi Kitazawa

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Michael Sy

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Thomas E Lane

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P2860

Role of Infection in the Pathogenesis of Alzheimerʼs Disease

Vascular risk factors and dementia: how to move forward?

Inhibition of glycogen synthase kinase-3 by insulin mediated by protein kinase B

Synapse loss and microglial activation precede tangles in a P301S tauopathy mouse model

Inflammation and Alzheimer's disease

Sequential phosphorylation of Tau by glycogen synthase kinase-3beta and protein kinase A at Thr212 and Ser214 generates the Alzheimer-specific epitope of antibody AT100 and requires a paired-helical-filament-like conformation

Identification and localization of Chlamydia pneumoniae in the Alzheimer's brain

GSK-3beta is required for memory reconsolidation in adult brain

Interleukin-1 mediates pathological effects of microglia on tau phosphorylation and on synaptophysin synthesis in cortical neurons through a p38-MAPK pathway

Cdk5 is involved in neuregulin-induced AChR expression at the neuromuscular junction

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10.1016/J.AJPATH.2011.02.012

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6

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178

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Rodrigo Medeiros

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2011-04-30T00:00:00Z

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51087642

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