Methamphetamine causes mitrochondrial oxidative damage in human T lymphocytes leading to functional impairment - Wikidata - awgldk - TriplyDB

Methamphetamine causes mitrochondrial oxidative damage in human T lymphocytes leading to functional impairment

Methamphetamine causes mitrochondrial oxidative damage in human T lymphocytes leading to functional impairment

http://www.wikidata.org/entity/Q35073066

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Methamphetamine administration targets multiple immune subsets and induces phenotypic alterations suggestive of immunosuppression Trace amine associated receptor 1 signaling in activated lymphocytes An Update of the Review of Neuropsychological Consequences of HIV and Substance Abuse: A Literature Review and Implications for Treatment and Future Research Impact of methamphetamine on infection and immunity Altered energy production, lowered antioxidant potential, and inflammatory processes mediate CNS damage associated with abuse of the psychostimulants MDMA and methamphetamine Methamphetamine and HIV-1-induced neurotoxicity: role of trace amine associated receptor 1 cAMP signaling in astrocytes Neuroimmune basis of methamphetamine toxicity Recent advances in methamphetamine neurotoxicity mechanisms and its molecular pathophysiology HIV-1, methamphetamine and astrocytes at neuroinflammatory Crossroads Plasma proteomic profiling in HIV-1 infected methamphetamine abusers Methamphetamine and inflammatory cytokines increase neuronal Na+/K+-ATPase isoform 3: relevance for HIV associated neurocognitive disorders P2X4 receptor regulates alcohol-induced responses in microglia Co-administration of betulinic acid and methamphetamine causes toxicity to dopaminergic and serotonergic nerve terminals in the striatum of late adolescent rats Total antioxidant capacity is significantly lower in cocaine-dependent and methamphetamine-dependent patients relative to normal controls: results from a preliminary study.Impairment of adult hippocampal neural progenitor proliferation by methamphetamine: role for nitrotyrosination.Involvement of metabotropic glutamate receptor 5, AKT/PI3K signaling and NF-κB pathway in methamphetamine-mediated increase in IL-6 and IL-8 expression in astrocytes.Methamphetamine Use in HIV-infected Individuals Affects T-cell Function and Viral Outcome during Suppressive Antiretroviral Therapy.Reactive oxygen species scavenger N-acetyl cysteine reduces methamphetamine-induced hyperthermia without affecting motor activity in mice.Impaired Subset Progression and Polyfunctionality of T Cells in Mice Exposed to Methamphetamine during Chronic LCMV Infection.Methamphetamine Alters the Antimicrobial Efficacy of Phagocytic Cells during Methicillin-Resistant Staphylococcus aureus Skin Infection.N-Acetyl Cysteine Protects against Methamphetamine-Induced Dopaminergic Neurodegeneration via Modulation of Redox Status and Autophagy in Dopaminergic Cells.Methamphetamine induces trace amine-associated receptor 1 (TAAR1) expression in human T lymphocytes: role in immunomodulation.Plasma Inflammatory Factors Are Associated with Anxiety, Depression, and Cognitive Problems in Adults with and without Methamphetamine Dependence: An Exploratory Protein Array Study.Mitochondrial injury and cognitive function in HIV infection and methamphetamine use.Voluntary exercise protects against methamphetamine-induced oxidative stress in brain microvasculature and disruption of the blood-brain barrier.Methamphetamine inhibits Toll-like receptor 9-mediated anti-HIV activity in macrophages Modulation of intracellular restriction factors contributes to methamphetamine-mediated enhancement of acquired immune deficiency syndrome virus infection of macrophages Acute bacterial meningitis with coincident methamphetamine use: a case report and review of the literature.Prosthodontics treatment considerations for methamphetamine-dependent patients.Methamphetamine alters occludin expression via NADPH oxidase-induced oxidative insult and intact caveolae.Methamphetamine decreases CD4 T cell frequency and alters pro-inflammatory cytokine production in a model of drug abuse.Methamphetamine mediates immune dysregulation in a murine model of chronic viral infection.Methamphetamine administration modifies leukocyte proliferation and cytokine production in murine tissues Brain imaging in methamphetamine-treated mice using a nitroxide contrast agent for EPR imaging of the redox status and a gadolinium contrast agent for MRI observation of blood-brain barrier function.Induction of mitochondrial permeability transition (MPT) pore opening and ROS formation as a mechanism for methamphetamine-induced mitochondrial toxicity.Methamphetamine alters T cell cycle entry and progression: role in immune dysfunction.

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P2860

Methamphetamine causes mitrochondrial oxidative damage in human T lymphocytes leading to functional impairment

http://www.wikidata.org/entity/Q35073066

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2010 nî lūn-bûn

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2010 թուականի Յուլիսին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի հուլիսին հրատարակված գիտական հոդված

@hy

2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Methamphetamine causes mitroch ...... ading to functional impairment

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Methamphetamine causes mitroch ...... ading to functional impairment

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Methamphetamine causes mitroch ...... ading to functional impairment

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Methamphetamine causes mitroch ...... ading to functional impairment

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Methamphetamine causes mitroch ...... ading to functional impairment

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Methamphetamine causes mitroch ...... ading to functional impairment

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Brenda Morsey

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Brian J Hawkins

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Holly Dykstra

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Jonathan M Cenna

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Servio H Ramirez

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Shongshan Fan

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Yuri Persidsky

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P2860

Calorie restriction induces mitochondrial biogenesis and bioenergetic efficiency

How mitochondria produce reactive oxygen species

Methamphetamine inhibits antigen processing, presentation, and phagocytosis

Mitochondrial calcium signalling and cell death: approaches for assessing the role of mitochondrial Ca2+ uptake in apoptosis

Mitochondrial membrane potential: a novel biomarker of oxidative environmental stress.

Mitochondrial dysfunction and oxidative stress in neurodegenerative diseases

Drosophila HtrA2 is dispensable for apoptosis but acts downstream of PINK1 independently from Parkin

Mitochondrial formation of reactive oxygen species

Normalizing mitochondrial superoxide production blocks three pathways of hyperglycaemic damage

Transaldolase is essential for maintenance of the mitochondrial transmembrane potential and fertility of spermatozoa.

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