Cardiac-specific overexpression of GTP cyclohydrolase 1 restores ischaemic preconditioning during hyperglycaemia - Wikidata - awgldk - TriplyDB

Cardiac-specific overexpression of GTP cyclohydrolase 1 restores ischaemic preconditioning during hyperglycaemia

Cardiac-specific overexpression of GTP cyclohydrolase 1 restores ischaemic preconditioning during hyperglycaemia

http://www.wikidata.org/entity/Q35073570

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The protein partners of GTP cyclohydrolase I in rat organs Tetrahydrobiopterin in cardiovascular health and disease.Transgenic overexpression of GTP cyclohydrolase 1 in cardiomyocytes ameliorates post-infarction cardiac remodeling.Increasing tetrahydrobiopterin in cardiomyocytes adversely affects cardiac redox state and mitochondrial function independently of changes in NO production.Impairment of endothelial-myocardial interaction increases the susceptibility of cardiomyocytes to ischemia/reperfusion injury.Up-regulation of microRNA-21 mediates isoflurane-induced protection of cardiomyocytes.Decreased tetrahydrobiopterin and disrupted association of Hsp90 with eNOS by hyperglycemia impair myocardial ischemic preconditioning.MicroRNA-21 Mediates Isoflurane-induced Cardioprotection against Ischemia-Reperfusion Injury via Akt/Nitric Oxide Synthase/Mitochondrial Permeability Transition Pore Pathway Isoflurane favorably modulates guanosine triphosphate cyclohydrolase-1 and endothelial nitric oxide synthase during myocardial ischemia and reperfusion injury in rats.Chronic Co-Administration of Sepiapterin and L-Citrulline Ameliorates Diabetic Cardiomyopathy and Myocardial Ischemia/Reperfusion Injury in Obese Type 2 Diabetic Mice Fenofibrate plus Metformin Produces Cardioprotection in a Type 2 Diabetes and Acute Myocardial Infarction Model.Cardiomyocyte GTP Cyclohydrolase 1 Protects the Heart Against Diabetic Cardiomyopathy Failure of Isoflurane Cardiac Preconditioning in Obese Type 2 Diabetic Mice Involves Aberrant Regulation of MicroRNA-21, Endothelial Nitric-oxide Synthase, and Mitochondrial Complex I.Connexins and Nitric Oxide Inside and Outside Mitochondria: Significance for Cardiac Protection and Adaptation.

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P2860

Cardiac-specific overexpression of GTP cyclohydrolase 1 restores ischaemic preconditioning during hyperglycaemia

http://www.wikidata.org/entity/Q35073570

description

2011 nî lūn-bûn

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2011 թուականի Մարտին հրատարակուած գիտական յօդուած

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2011 թվականի մարտին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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name

Cardiac-specific overexpressio ...... itioning during hyperglycaemia

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Cardiac-specific overexpressio ...... itioning during hyperglycaemia

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type

label

Cardiac-specific overexpressio ...... itioning during hyperglycaemia

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Cardiac-specific overexpressio ...... itioning during hyperglycaemia

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Cardiac-specific overexpressio ...... itioning during hyperglycaemia

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Cardiac-specific overexpressio ...... itioning during hyperglycaemia

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Cardiovascular Research

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Cardiac-specific overexpressio ...... itioning during hyperglycaemia

@en

P2093

Danijel Pravdic

http://www.w3.org/2001/XMLSchema#string

David C Warltier

http://www.w3.org/2001/XMLSchema#string

Galen M Pieper

http://www.w3.org/2001/XMLSchema#string

Irina A Ionova

http://www.w3.org/2001/XMLSchema#string

Jeannette Vásquez-Vivar

http://www.w3.org/2001/XMLSchema#string

Judy R Kersten

http://www.w3.org/2001/XMLSchema#string

Naoyuki Hirata

http://www.w3.org/2001/XMLSchema#string

Nikolina Vladic

http://www.w3.org/2001/XMLSchema#string

Phillip F Pratt

http://www.w3.org/2001/XMLSchema#string

Zhi-Dong Ge

http://www.w3.org/2001/XMLSchema#string

P2860

Augmented BH4 by gene transfer restores nitric oxide synthase function in hyperglycemic human endothelial cells

Tetrahydrobiopterin biosynthesis, regeneration and functions

Deficient BH4 production via de novo and salvage pathways regulates NO responses to cytokines in adult cardiac myocytes

Nitric oxide synthases: structure, function and inhibition

Preconditioning with ischemia: a delay of lethal cell injury in ischemic myocardium

Tetrahydrobiopterin improves endothelium-dependent vasodilation in chronic smokers : evidence for a dysfunctional nitric oxide synthase

Sepiapterin decreases acute rejection and apoptosis in cardiac transplants independently of changes in nitric oxide and inducible nitric-oxide synthase dimerization.

Opening of the mitochondrial permeability transition pore causes depletion of mitochondrial and cytosolic NAD+ and is a causative event in the death of myocytes in postischemic reperfusion of the heart.

The late phase of preconditioning.

Ischemic preconditioning: from adenosine receptor to KATP channel.

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340-349

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10.1093/CVR/CVR079

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2

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91

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2011-03-21T00:00:00Z

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50595692

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21422102

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3125073

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