CXCR3-dependent plasma blast migration to the central nervous system during viral encephalomyelitis. - Wikidata - awgldk - TriplyDB

CXCR3-dependent plasma blast migration to the central nervous system during viral encephalomyelitis.

CXCR3-dependent plasma blast migration to the central nervous system during viral encephalomyelitis.

http://www.wikidata.org/entity/Q35077540

about

Tissue-resident memory T cells Distinct populations of innate CD8+ T cells revealed in a CXCR3 reporter mouse.CXCL12-induced monocyte-endothelial interactions promote lymphocyte transmigration across an in vitro blood-brain barrier Persistent humoral immune responses in the CNS limit recovery of reactivated murine cytomegalovirus Compartmentalization of innate immune responses in the central nervous system during cryptococcal meningitis/HIV coinfection Progression from IgD+ IgM+ to isotype-switched B cells is site specific during coronavirus-induced encephalomyelitis.Enhancement of blood-brain barrier permeability is required for intravenously administered virus neutralizing antibodies to clear an established rabies virus infection from the brain and prevent the development of rabies in mice.Early differentiated CD138(high) MHCII+ IgG+ plasma cells express CXCR3 and localize into inflamed kidneys of lupus mice Plasmablasts as migratory IgG-producing cells in the pathogenesis of neuromyelitis optica T helper cell- and CD40-dependent germline IgM prevents chronic virus-induced demyelinating disease Distinct Immune Responses in Resistant and Susceptible Strains of Mice during Neurovirulent Alphavirus Encephalomyelitis.Myd88 Initiates Early Innate Immune Responses and Promotes CD4 T Cells during Coronavirus Encephalomyelitis.The spleen is the major source of antidonor antibody-secreting cells in murine heart allograft recipients The chemokine receptor CXCR2 and coronavirus-induced neurologic disease.Recruitment and retention of B cells in the central nervous system in response to alphavirus encephalomyelitis.Astrocyte-derived CXCL10 drives accumulation of antibody-secreting cells in the central nervous system during viral encephalomyelitis.CXCL13 promotes isotype-switched B cell accumulation to the central nervous system during viral encephalomyelitis.Genome-Wide Transcriptional Profiling Reveals Two Distinct Outcomes in Central Nervous System Infections of Rabies Virus.Glial cell activation, recruitment, and survival of B-lineage cells following MCMV brain infection Increased CXCR3 Expression of Infiltrating Plasma Cells in Hunner Type Interstitial Cystitis.IL-21 optimizes T cell and humoral responses in the central nervous system during viral encephalitis.Intrathecal humoral immunity to encephalitic RNA viruses.Tracking plasma cell differentiation and survival.Immune responses to non-tumor antigens in the central nervous system.CXCR3 signaling in glial cells ameliorates experimental autoimmune encephalomyelitis by restraining the generation of a pro-Th17 cytokine milieu and reducing CNS-infiltrating Th17 cells.An optimized method for enumerating CNS derived memory B cells during viral-induced inflammation.Interferon gamma modulation of disease manifestation and the local antibody response to alphavirus encephalomyelitis.Activated GL7+ B cells are maintained within the inflamed CNS in the absence of follicle formation during viral encephalomyelitis.Protective Humoral Immunity in the CNS Requires Peripheral CD19-Dependent Germinal Center Formation Following Coronavirus Encephalomyelitis.Death and gastrointestinal bleeding complicate encephalomyelitis in mice with delayed appearance of CNS IgM after intranasal alphavirus infection.Characterization of the Antibody Response after Cervical Spinal Cord Injury.Aging and lymphocyte changes by immunomodulatory therapies impact PML risk in multiple sclerosis patients.

P2860

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P2860

CXCR3-dependent plasma blast migration to the central nervous system during viral encephalomyelitis.

http://www.wikidata.org/entity/Q35077540

description

2011 nî lūn-bûn

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2011 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի ապրիլին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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name

CXCR3-dependent plasma blast m ...... uring viral encephalomyelitis.

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CXCR3-dependent plasma blast m ...... uring viral encephalomyelitis.

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CXCR3-dependent plasma blast m ...... uring viral encephalomyelitis.

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CXCR3-dependent plasma blast m ...... uring viral encephalomyelitis.

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CXCR3-dependent plasma blast m ...... uring viral encephalomyelitis.

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Journal of Virology

P1476

CXCR3-dependent plasma blast m ...... uring viral encephalomyelitis.

@en

P2093

Claudia Hindinger

http://www.w3.org/2001/XMLSchema#string

Cornelia C Bergmann

http://www.w3.org/2001/XMLSchema#string

Cristina P Marques

http://www.w3.org/2001/XMLSchema#string

David R Hinton

http://www.w3.org/2001/XMLSchema#string

Parul Kapil

http://www.w3.org/2001/XMLSchema#string

Richard M Ransohoff

http://www.w3.org/2001/XMLSchema#string

Stephen A Stohlman

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Stephen L Nutt

http://www.w3.org/2001/XMLSchema#string

Timothy W Phares

http://www.w3.org/2001/XMLSchema#string

P2860

CXCR7 influences leukocyte entry into the CNS parenchyma by controlling abluminal CXCL12 abundance during autoimmunity

Accumulation of plasma cells expressing CXCR3 in the synovial sublining regions of early rheumatoid arthritis in association with production of Mig/CXCL9 by synovial fibroblasts.

The production of antibody by invading B cells is required for the clearance of rabies virus from the central nervous system.

CXCR2 and CXCR4 antagonistically regulate neutrophil trafficking from murine bone marrow

Target-dependent B7-H1 regulation contributes to clearance of central nervous system infection and dampens morbidity.

Lymphoid chemokines in the CNS.

Factors supporting intrathecal humoral responses following viral encephalomyelitis.

B cells and multiple sclerosis.

Control of central nervous system viral persistence by neutralizing antibody.

The lymphoid chemokine, CXCL13, is dispensable for the initial recruitment of B cells to the acutely inflamed central nervous system

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6136-6147

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10.1128/JVI.00202-11

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P433

13

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P478

85

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2011-04-20T00:00:00Z

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51065465

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21507985

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3126522

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