SMAD-PI3K-Akt-mTOR pathway mediates BMP-7 polarization of monocytes into M2 macrophages.
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Immune Cell Regulatory Pathways Unexplored as Host-Directed Therapeutic Targets for Mycobacterium tuberculosis: An Opportunity to Apply Precision Medicine Innovations to Infectious DiseasesOverview of Traumatic Brain Injury: An Immunological ContextMicroglial and macrophage polarization—new prospects for brain repairMuch More than M1 and M2 Macrophages, There are also CD169(+) and TCR(+) Macrophages.BMP-7 Treatment Increases M2 Macrophage Differentiation and Reduces Inflammation and Plaque Formation in Apo E-/- Mice.Microglia activation is essential for BMP7-mediated retinal reactive gliosis.Evolutionary roots of arginase expression and regulation.Regulation of innate immune cell function by mTOR.A neuropeptide, Substance-P, directly induces tissue-repairing M2 like macrophages by activating the PI3K/Akt/mTOR pathway even in the presence of IFNγ.Primary human monocytes differentiate into M2 macrophages and involve Notch-1 pathway.Regulation of Notch 1 signaling in THP-1 cells enhances M2 macrophage differentiation.Akt Signaling Pathway in Macrophage Activation and M1/M2 Polarization.Metabolism in Immune Cell Differentiation and Function.Ubiquitylation of MFHAS1 by the ubiquitin ligase praja2 promotes M1 macrophage polarization by activating JNK and p38 pathways.Hcmv-miR-UL22A-5p: A Biomarker in Transplantation With Broad Impact on Host Gene Expression and Potential Immunological Implications.Quercetin prevents hepatic fibrosis by inhibiting hepatic stellate cell activation and reducing autophagy via the TGF-β1/Smads and PI3K/Akt pathways.The mTOR kinase inhibitors polarize glioma-activated microglia to express a M1 phenotype.Therapeutic effects of OP-1 on metal wear particle induced osteoblasts injury in vitro.Agmatine Modulates the Phenotype of Macrophage Acute Phase after Spinal Cord Injury in Rats.Genetic variations in the PI3K/PTEN/AKT/mTOR pathway predict tumor response and disease-free survival in locally advanced rectal cancer patients receiving preoperative chemoradiotherapy and radical surgery.Trypanosomatid Infections: How Do Parasites and Their Excreted-Secreted Factors Modulate the Inducible Metabolism of l-Arginine in Macrophages?Glioblastoma stem cell-derived exosomes induce M2 macrophages and PD-L1 expression on human monocytes.
P2860
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P2860
SMAD-PI3K-Akt-mTOR pathway mediates BMP-7 polarization of monocytes into M2 macrophages.
description
2013 nî lūn-bûn
@nan
2013 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2013 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
name
SMAD-PI3K-Akt-mTOR pathway mediates BMP-7 polarization of monocytes into M2 macrophages.
@ast
SMAD-PI3K-Akt-mTOR pathway mediates BMP-7 polarization of monocytes into M2 macrophages.
@en
type
label
SMAD-PI3K-Akt-mTOR pathway mediates BMP-7 polarization of monocytes into M2 macrophages.
@ast
SMAD-PI3K-Akt-mTOR pathway mediates BMP-7 polarization of monocytes into M2 macrophages.
@en
prefLabel
SMAD-PI3K-Akt-mTOR pathway mediates BMP-7 polarization of monocytes into M2 macrophages.
@ast
SMAD-PI3K-Akt-mTOR pathway mediates BMP-7 polarization of monocytes into M2 macrophages.
@en
P2860
P1433
P1476
SMAD-PI3K-Akt-mTOR pathway mediates BMP-7 polarization of monocytes into M2 macrophages.
@en
P2093
Crystal Rocher
Dinender K Singla
P2860
P304
P356
10.1371/JOURNAL.PONE.0084009
P407
P577
2013-12-20T00:00:00Z