Caveolae control the anti-inflammatory phenotype of senescent endothelial cells. - Wikidata - awgldk - TriplyDB

Caveolae control the anti-inflammatory phenotype of senescent endothelial cells.

Caveolae control the anti-inflammatory phenotype of senescent endothelial cells.

http://www.wikidata.org/entity/Q35080556

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Mitochondria in the spotlight of aging and idiopathic pulmonary fibrosis The RhoGAP protein ARHGAP18/SENEX localizes to microtubules and regulates their stability in endothelial cells Caveolin-1 controls mitochondrial function through regulation of m-AAA mitochondrial protease Versatile Functions of Caveolin-1 in Aging-related Diseases.The prognostic values of caveolin-1 immunoreactivity in peritubular capillaries in patients with kidney transplantation.Caveolin-1-Knockout Mouse as a Model of Inflammatory Diseases

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Caveolae control the anti-inflammatory phenotype of senescent endothelial cells.

http://www.wikidata.org/entity/Q35080556

description

2014 nî lūn-bûn

@nan

2014 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

@zh-hk

2014年論文

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2014年論文

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2014年论文

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name

Caveolae control the anti-inflammatory phenotype of senescent endothelial cells.

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Caveolae control the anti-inflammatory phenotype of senescent endothelial cells.

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type

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Caveolae control the anti-inflammatory phenotype of senescent endothelial cells.

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Caveolae control the anti-inflammatory phenotype of senescent endothelial cells.

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Caveolae control the anti-inflammatory phenotype of senescent endothelial cells.

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Caveolae control the anti-inflammatory phenotype of senescent endothelial cells.

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Caveolae control the anti-inflammatory phenotype of senescent endothelial cells.

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P2093

Angelina J Lay

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Elizabeth E Powter

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Jennifer R Gamble

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Mai H Tran

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Mathew A Vadas

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Paul R Coleman

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P2860

Constitutive and growth factor-regulated phosphorylation of caveolin-1 occurs at the same site (Tyr-14) in vivo: identification of a c-Src/Cav-1/Grb7 signaling cassette

Transcriptional evidence for the "Reverse Warburg Effect" in human breast cancer tumor stroma and metastasis: similarities with oxidative stress, inflammation, Alzheimer's disease, and "Neuron-Glia Metabolic Coupling"

Senescence of activated stellate cells limits liver fibrosis

NF-kappaB controls the global pro-inflammatory response in endothelial cells: evidence for the regulation of a pro-atherogenic program

Oncogene-induced senescence relayed by an interleukin-dependent inflammatory network.

The senescence-associated secretory phenotype: the dark side of tumor suppression

SDPR induces membrane curvature and functions in the formation of caveolae.

Phospho-caveolin-1 mediates integrin-regulated membrane domain internalization.

Role of caveolae in shear stress-mediated endothelium-dependent dilation in coronary arteries.

Caveolin-1 influences vascular protease activity and is a potential stabilizing factor in human atherosclerotic disease.

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102-111

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scholarly article

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10.1111/ACEL.12270

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1

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14

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Robert G. Parton

Patrick Bertolino

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2014-11-19T00:00:00Z

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268795959

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