Molecular pathogenesis of chronic wounds: the role of beta-catenin and c-myc in the inhibition of epithelialization and wound healing - Wikidata - awgldk - TriplyDB

Molecular pathogenesis of chronic wounds: the role of beta-catenin and c-myc in the inhibition of epithelialization and wound healing

Molecular pathogenesis of chronic wounds: the role of beta-catenin and c-myc in the inhibition of epithelialization and wound healing

http://www.wikidata.org/entity/Q35084866

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Molecular markers in patients with chronic wounds to guide surgical debridement Epigenetic Regulation of Epidermal Stem Cell Biomarkers and Their Role in Wound Healing Chemokines as Therapeutic Targets to Improve Healing Efficiency of Chronic Wounds Palatogenesis and cutaneous repair: A two-headed coin Cutaneous wound healing: recruiting developmental pathways for regeneration Thyrotropin-releasing hormone (TRH) promotes wound re-epithelialisation in frog and human skin Application of on-line nanoLC-IT-TOF in the identification of serum β-catenin complex in mice scald model Induction of specific microRNAs inhibits cutaneous wound healing UV fluorescence excitation imaging of healing of wounds in skin: Evaluation of wound closure in organ culture model.Genetic and epigenetic events in diabetic wound healing.Glucose toxic effects on granulation tissue productive cells: the diabetics' impaired healing.12-Hydroxyheptadecatrienoic acid promotes epidermal wound healing by accelerating keratinocyte migration via the BLT2 receptor Equine CTNNB1 and PECAM1 nucleotide structure and expression analyses in an experimental model of normal and pathological wound repair.Attenuation of the transforming growth factor beta-signaling pathway in chronic venous ulcers Farnesyl pyrophosphate inhibits epithelialization and wound healing through the glucocorticoid receptor Human CD133+ progenitor cells promote the healing of diabetic ischemic ulcers by paracrine stimulation of angiogenesis and activation of Wnt signaling.Early effects of Staphylococcus aureus biofilm secreted products on inflammatory responses of human epithelial keratinocytes Topical androgen antagonism promotes cutaneous wound healing without systemic androgen deprivation by blocking β-catenin nuclear translocation and cross-talk with TGF-β signaling in keratinocytes.Network based transcription factor analysis of regenerating axolotl limbs Altered ECM deposition by diabetic foot ulcer-derived fibroblasts implicates fibronectin in chronic wound repair.Evaluation of the collaborative network of highly correlating skin proteins and its change following treatment with glucocorticoids The redox enzyme p66Shc contributes to diabetes and ischemia-induced delay in cutaneous wound healing.Human lactoferrin stimulates skin keratinocyte function and wound re-epithelialization.Keratinocyte-targeted overexpression of the glucocorticoid receptor delays cutaneous wound healing.Using gene transcription patterns (bar coding scans) to guide wound debridement and healing.High cost of stage IV pressure ulcers.Regional anaesthesia with sedation protocol to safely debride sacral pressure ulcers.Hyperbaric oxygen, vasculogenic stem cells, and wound healing.'See-saw' expression of microRNA-198 and FSTL1 from a single transcript in wound healing.Wound edge biopsy sites in chronic wounds heal rapidly and do not result in delayed overall healing of the wounds.Towards elucidating the connection between epithelial-mesenchymal transitions and stemness Cellular and molecular basis of wound healing in diabetes.Cortisol synthesis in epidermis is induced by IL-1 and tissue injury.Glucocorticoid receptor localizes to adherens junctions at the plasma membrane of keratinocytes.Gene therapy of c-myc suppressor FUSE-binding protein-interacting repressor by Sendai virus delivery prevents tracheal stenosis.Influence of 4% icodextrin solution on peritoneal tissue response and adhesion formation.Deregulation of epidermal stem cell niche contributes to pathogenesis of nonhealing venous ulcers Increased number of Langerhans cells in the epidermis of diabetic foot ulcers correlates with healing outcome.Quality assessment of tissue specimens for studies of diabetic foot ulcers.Three-dimensional human tissue models that incorporate diabetic foot ulcer-derived fibroblasts mimic in vivo features of chronic wounds.

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Molecular pathogenesis of chronic wounds: the role of beta-catenin and c-myc in the inhibition of epithelialization and wound healing

http://www.wikidata.org/entity/Q35084866

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2005 nî lūn-bûn

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The American Journal of Pathology

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Molecular pathogenesis of chro ...... elialization and wound healing

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P2093

Ankit Merchant

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Brian Lee

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Constantinos Vouthounis

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Harold Brem

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John Fallon

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Marjana Tomic-Canic

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Michael Stallcup

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Olivera Stojadinovic

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Robert D Galiano

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P2860

The stem cell compartment in human interfollicular epidermis

Identification of c-MYC as a target of the APC pathway

Mutations in the plakophilin 1 gene result in ectodermal dysplasia/skin fragility syndrome

Onset of keratin 17 expression coincides with the definition of major epithelial lineages during skin development

Becaplermin: recombinant platelet derived growth factor, a new treatment for healing diabetic foot ulcers

Are desmosomes more than tethers for intermediate filaments?

From cadherins to catenins: cytoplasmic protein interactions and regulation of cell adhesion

Delayed wound healing in keratin 6a knockout mice

Inhibitory effects of antisense oligodeoxynucleotides targeting c-myc mRNA on smooth muscle cell proliferation and migration

Beta-catenin binds to the activation function 2 region of the androgen receptor and modulates the effects of the N-terminal domain and TIF2 on ligand-dependent transcription

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