Kinetics of disease progression and host response in a rat model of bubonic plague. - Wikidata - awgldk - TriplyDB

Kinetics of disease progression and host response in a rat model of bubonic plague.

Kinetics of disease progression and host response in a rat model of bubonic plague.

http://www.wikidata.org/entity/Q35087782

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Role of the Yersinia pestis yersiniabactin iron acquisition system in the incidence of flea-borne plague The RACK1 signaling scaffold protein selectively interacts with Yersinia pseudotuberculosis virulence function Polyphosphate and omptins: novel bacterial procoagulant agents Proteolytic inactivation of tissue factor pathway inhibitor by bacterial omptins Yersinia pestis and Yersinia pseudotuberculosis infection: a regulatory RNA perspective The Role of Early-Phase Transmission in the Spread of Yersinia pestis Temporal Progression of Pneumonic Plague in Blood of Nonhuman Primate: A Transcriptomic Analysis Dermal neutrophil, macrophage and dendritic cell responses to Yersinia pestis transmitted by fleas Dissemination of a highly virulent pathogen: tracking the early events that define infection New insights into how Yersinia pestis adapts to its mammalian host during bubonic plague Omics strategies for revealing Yersinia pestis virulence The single substitution I259T, conserved in the plasminogen activator Pla of pandemic Yersinia pestis branches, enhances fibrinolytic activity.Invited review: Breaking barriers--attack on innate immune defences by omptin surface proteases of enterobacterial pathogens.Early-phase transmission of Yersinia pestis by unblocked fleas as a mechanism explaining rapidly spreading plague epizootics.Characterization of systemic and pneumonic murine models of plague infection using a conditionally virulent strain.Defective innate cell response and lymph node infiltration specify Yersinia pestis infection.CD8(+) T cells restrict Yersinia pseudotuberculosis infection: bypass of anti-phagocytosis by targeting antigen-presenting cells Absence of inflammation and pneumonia during infection with nonpigmented Yersinia pestis reveals a new role for the pgm locus in pathogenesis.Immune defense against pneumonic plague YopH inhibits early pro-inflammatory cytokine responses during plague pneumonia.Comparative Analyses of Transcriptional Profiles in Mouse Organs Using a Pneumonic Plague Model after Infection with Wild-Type Yersinia pestis CO92 and Its Braun Lipoprotein Mutant.Glutathionylation of Yersinia pestis LcrV and Its Effects on Plague Pathogenesis Histopathological observation of immunized rhesus macaques with plague vaccines after subcutaneous infection of Yersinia pestis.Remote monitoring of the progression of primary pneumonic plague in Brown Norway rats in high-capacity, high-containment housing.Imaging of bubonic plague dynamics by in vivo tracking of bioluminescent Yersinia pestis.Transcriptomic and innate immune responses to Yersinia pestis in the lymph node during bubonic plague Outer membrane protein X (Ail) contributes to Yersinia pestis virulence in pneumonic plague and its activity is dependent on the lipopolysaccharide core length.Primary pneumonic plague in the African Green monkey as a model for treatment efficacy evaluation.Bioluminescence imaging to track bacterial dissemination of Yersinia pestis using different routes of infection in mice Yersinia pestis intracellular parasitism of macrophages from hosts exhibiting high and low severity of plague Role of the Yersinia pestis plasminogen activator in the incidence of distinct septicemic and bubonic forms of flea-borne plague.Distinct CCR2(+) Gr1(+) cells control growth of the Yersinia pestis ΔyopM mutant in liver and spleen during systemic plague.Efficacy of ciprofloxacin-gentamicin combination therapy in murine bubonic plague.Mathematical relationship between cytokine concentrations and pathogen levels during infection.How the structural gene products of Yersinia pestis relate to virulence.Interaction of Yersinia pestis with macrophages: limitations in YopJ-dependent apoptosis.Histamine signaling through the H(2) receptor in the Peyer's patch is important for controlling Yersinia enterocolitica infection.Plague in Guinea pigs and its prevention by subunit vaccines Adaptive response of Yersinia pestis to extracellular effectors of innate immunity during bubonic plague.Model systems to study plague pathogenesis and develop new therapeutics

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P2860

Kinetics of disease progression and host response in a rat model of bubonic plague.

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2005年の論文

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Kinetics of disease progression and host response in a rat model of bubonic plague.

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The American Journal of Pathology

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Kinetics of disease progression and host response in a rat model of bubonic plague.

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Brian B Gowen

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Daniel Long

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Donald Gardner

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Florent Sebbane

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P2860

Genome sequence of the Brown Norway rat yields insights into mammalian evolution

Dendritic cells and the control of immunity

Inhibition of the mitogen-activated protein kinase kinase superfamily by a Yersinia effector

Yersinia enterocolitica promotes deactivation of macrophage mitogen-activated protein kinases extracellular signal-regulated kinase-1/2, p38, and c-Jun NH2-terminal kinase. Correlation with its inhibitory effect on tumor necrosis factor-alpha produc

Role of YopP in suppression of tumor necrosis factor alpha release by macrophages during Yersinia infection

The virulence plasmid of Yersinia, an antihost genome

Yersinia pestis--etiologic agent of plague

Clinical gram-positive sepsis: does it fundamentally differ from gram-negative bacterial sepsis?

Association between virulence of Yersinia pestis and suppression of gamma interferon and tumor necrosis factor alpha.

Yersinia pestis and plague.

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