Preliminary evidence that Clostridium perfringens type A enterotoxin is present in a 160,000-Mr complex in mammalian membranes.
about
Cloning, nucleotide sequencing, and expression of the Clostridium perfringens enterotoxin gene in Escherichia coliIdentification of a prepore large-complex stage in the mechanism of action of Clostridium perfringens enterotoxinEnteric bacterial toxins: mechanisms of action and linkage to intestinal secretion.Characterization of membrane-associated Clostridium perfringens enterotoxin following pronase treatment.Identification of a Clostridium perfringens enterotoxin region required for large complex formation and cytotoxicity by random mutagenesis.Expression from the Clostridium perfringens cpe promoter in C. perfringens and Bacillus subtilis.Studies of Clostridium perfringens enterotoxin action at different temperatures demonstrate a correlation between complex formation and cytotoxicityEvidence that a region(s) of the Clostridium perfringens enterotoxin molecule remains exposed on the external surface of the mammalian plasma membrane when the toxin is sequestered in small or large complexes.Molecular cloning of the 3' half of the Clostridium perfringens enterotoxin gene and demonstration that this region encodes receptor-binding activityDeletion analysis of the Clostridium perfringens enterotoxin.Correlation between plasmid content and infectivity in Borrelia burgdorferi.Noncytotoxic Clostridium perfringens enterotoxin (CPE) variants localize CPE intestinal binding and demonstrate a relationship between CPE-induced cytotoxicity and enterotoxicityMolecular genetics and pathogenesis of Clostridium perfringens.Mapping of functional regions of Clostridium perfringens type A enterotoxin.A conjugated synthetic peptide corresponding to the C-terminal region of Clostridium perfringens type A enterotoxin elicits an enterotoxin-neutralizing antibody response in miceFine mapping of the N-terminal cytotoxicity region of Clostridium perfringens enterotoxin by site-directed mutagenesisClostridium perfringens epsilon-toxin acts on MDCK cells by forming a large membrane complex.
P2860
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P2860
Preliminary evidence that Clostridium perfringens type A enterotoxin is present in a 160,000-Mr complex in mammalian membranes.
description
1989 nî lūn-bûn
@nan
1989 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
1989 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
1989年の論文
@ja
1989年論文
@yue
1989年論文
@zh-hant
1989年論文
@zh-hk
1989年論文
@zh-mo
1989年論文
@zh-tw
1989年论文
@wuu
name
Preliminary evidence that Clos ...... omplex in mammalian membranes.
@ast
Preliminary evidence that Clos ...... omplex in mammalian membranes.
@en
type
label
Preliminary evidence that Clos ...... omplex in mammalian membranes.
@ast
Preliminary evidence that Clos ...... omplex in mammalian membranes.
@en
prefLabel
Preliminary evidence that Clos ...... omplex in mammalian membranes.
@ast
Preliminary evidence that Clos ...... omplex in mammalian membranes.
@en
P2860
P1476
Preliminary evidence that Clos ...... complex in mammalian membranes
@en
P2093
P2860
P304
P407
P577
1989-02-01T00:00:00Z