Shigella type III secretion protein MxiI is recognized by Naip2 to induce Nlrc4 inflammasome activation independently of Pkcδ.
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Cellular Aspects of Shigella Pathogenesis: Focus on the Manipulation of Host Cell ProcessesShigella IpaH Family Effectors as a Versatile Model for Studying Pathogenic BacteriaThe NAIP/NLRC4 inflammasomesPyroptotic cell death defends against intracellular pathogensInternational Union of Basic and Clinical Pharmacology. XCVI. Pattern recognition receptors in health and diseaseA second wave of Salmonella T3SS1 activity prolongs the lifespan of infected epithelial cells.Shigella IpaH7.8 E3 ubiquitin ligase targets glomulin and activates inflammasomes to demolish macrophages.Regulation of inflammasome activation.NAIP proteins are required for cytosolic detection of specific bacterial ligands in vivo.Genetic functions of the NAIP family of inflammasome receptors for bacterial ligands in micePost-translational regulation of inflammasomes.T cell-intrinsic ASC critically promotes T(H)17-mediated experimental autoimmune encephalomyelitisManipulation of the host cell death pathway by Shigella.Role of inflammasomes in intestinal inflammation and Crohn's disease.The NAIP-NLRC4 inflammasome in innate immune detection of bacterial flagellin and type III secretion apparatus.Shigella manipulates host immune responses by delivering effector proteins with specific roles.Role of virulence factors on host inflammatory response induced by diarrheagenic Escherichia coli pathotypes.Characterization of a multicomponent live, attenuated Shigella flexneri vaccine.Macrophage Apoptosis Triggered by IpaD from Shigella flexneri.General Strategies in Inflammasome Biology.The Orchestra and Its Maestro: Shigella's Fine-Tuning of the Inflammasome Platforms.How Do the Virulence Factors of Shigella Work Together to Cause Disease?Multi-receptor detection of individual bacterial products by the innate immune system.Mechanisms governing inflammasome activation, assembly and pyroptosis induction.Posttranslational Modification as a Critical Determinant of Cytoplasmic Innate Immune Recognition.NAIP inflammasomes give the NOD to bacterial ligands.Deviant Behavior: Tick-Borne Pathogens and Inflammasome Signaling.Shigella hijacks the glomulin-cIAPs-inflammasome axis to promote inflammation.
P2860
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P2860
Shigella type III secretion protein MxiI is recognized by Naip2 to induce Nlrc4 inflammasome activation independently of Pkcδ.
description
2014 nî lūn-bûn
@nan
2014 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
Shigella type III secretion pr ...... ivation independently of Pkcδ.
@ast
Shigella type III secretion pr ...... ivation independently of Pkcδ.
@en
type
label
Shigella type III secretion pr ...... ivation independently of Pkcδ.
@ast
Shigella type III secretion pr ...... ivation independently of Pkcδ.
@en
prefLabel
Shigella type III secretion pr ...... ivation independently of Pkcδ.
@ast
Shigella type III secretion pr ...... ivation independently of Pkcδ.
@en
P2093
P2860
P1433
P1476
Shigella type III secretion pr ...... ivation independently of Pkcδ.
@en
P2093
Chihiro Sasakawa
Gabriel Núñez
Hitomi Mimuro
Luigi Franchi
Raul Muñoz-Planillo
Shiho Suzuki
Toshihiko Suzuki
P2860
P304
P356
10.1371/JOURNAL.PPAT.1003926
P577
2014-02-06T00:00:00Z