Inhibition of filamentation can be used to treat disseminated candidiasis - Wikidata - awgldk - TriplyDB

Inhibition of filamentation can be used to treat disseminated candidiasis

Inhibition of filamentation can be used to treat disseminated candidiasis

http://www.wikidata.org/entity/Q35091490

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Evolution and Application of Inteins in Candida species: A Review Harnessing single cell sorting to identify cell division genes and regulators in bacteria An automated high-throughput cell-based multiplexed flow cytometry assay to identify novel compounds to target Candida albicans virulence-related proteins Morphological plasticity promotes resistance to phagocyte killing of uropathogenic Escherichia coli Systematic screens of a Candida albicans homozygous deletion library decouple morphogenetic switching and pathogenicity.Candida albicans virulence and drug-resistance requires the O-acyltransferase Gup1p.Using C. elegans for antimicrobial drug discovery.Hyphal development in Candida albicans requires two temporally linked changes in promoter chromatin for initiation and maintenance.Morphogenesis is not required for Candida albicans-Staphylococcus aureus intra-abdominal infection-mediated dissemination and lethal sepsis.The transcriptional regulator Nrg1p controls Candida albicans biofilm formation and dispersion Power of yeast for analysis of eukaryotic translation initiation Modulation of morphogenesis in Candida albicans by various small molecules.eIF4E is an important determinant of adhesion and pseudohyphal growth of the yeast S. cerevisiae.A histone deacetylase adjusts transcription kinetics at coding sequences during Candida albicans morphogenesis.Candida albicans hyphal initiation and elongation G1/S transcription factor orthologues Swi4p and Swi6p are important but not essential for cell proliferation and influence hyphal development in the fungal pathogen Candida albicans.Antifungal activity of phlorotannins against dermatophytes and yeasts: approaches to the mechanism of action and influence on Candida albicans virulence factor.Conjugated linoleic acid inhibits hyphal growth in Candida albicans by modulating Ras1p cellular levels and downregulating TEC1 expression.A Novel Small Molecule Inhibitor of Candida albicans Biofilm Formation, Filamentation and Virulence with Low Potential for the Development of Resistance.Distinct stages during colonization of the mouse gastrointestinal tract by Candida albicans Use of a genetically engineered strain to evaluate the pathogenic potential of yeast cell and filamentous forms during Candida albicans systemic infection in immunodeficient mice.Physiologic expression of the Candida albicans pescadillo homolog is required for virulence in a murine model of hematogenously disseminated candidiasis The cell wall of the human pathogen Candida glabrata: differential incorporation of novel adhesin-like wall proteins Efficacy of a genetically engineered Candida albicans tet-NRG1 strain as an experimental live attenuated vaccine against hematogenously disseminated candidiasis.Examination of the pathogenic potential of Candida albicans filamentous cells in an animal model of haematogenously disseminated candidiasis.Antifungal activity of tamoxifen: in vitro and in vivo activities and mechanistic characterization Effective concentration-based serum pharmacodynamics for antifungal azoles in a murine model of disseminated Candida albicans infection.Human fungal pathogen Candida albicans in the postgenomic era: an overview.Candidiasis drug discovery and development: new approaches targeting virulence for discovering and identifying new drugs.Targeting Candida albicans filamentation for antifungal drug development.Potential of anti-Candida antibodies in immunoprophylaxis.Attenuation of Candida albicans virulence with focus on disruption of its vacuole functions Antifungal activity of phenolic-rich Lavandula multifida L. essential oil.New Claims for Wild Carrot (Daucus carota subsp. carota) Essential Oil.Therapeutic activity of a Saccharomyces cerevisiae-based probiotic and inactivated whole yeast on vaginal candidiasis.Dectin-1 is required for miR155 upregulation in murine macrophages in response to Candida albicans.Chemical inhibitors of Candida albicans hyphal morphogenesis target endocytosis.The Hog1 MAP Kinase Promotes the Recovery from Cell Cycle Arrest Induced by Hydrogen Peroxide in Candida albicans.Antifungal activity of Ferulago capillaris essential oil against Candida, Cryptococcus, Aspergillus and dermatophyte species.Antifungal Activity of Thapsia villosa Essential Oil against Candida, Cryptococcus, Malassezia, Aspergillus and Dermatophyte Species.

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P2860

Inhibition of filamentation can be used to treat disseminated candidiasis

http://www.wikidata.org/entity/Q35091490

description

2006 nî lūn-bûn

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2006 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

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2006 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2006年の論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年论文

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Inhibition of filamentation can be used to treat disseminated candidiasis

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Inhibition of filamentation can be used to treat disseminated candidiasis

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Inhibition of filamentation can be used to treat disseminated candidiasis

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Inhibition of filamentation can be used to treat disseminated candidiasis

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Inhibition of filamentation can be used to treat disseminated candidiasis

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Inhibition of filamentation can be used to treat disseminated candidiasis

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Antimicrobial Agents and Chemotherapy

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Inhibition of filamentation can be used to treat disseminated candidiasis

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P2093

Alex Monreal

http://www.w3.org/2001/XMLSchema#string

Alexander P Bryant

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Angelika Fretzen

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Anna L Lazzell

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Carlos Monteagudo

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Erik O Solberg

http://www.w3.org/2001/XMLSchema#string

G Todd Milne

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Jose L Lopez-Ribot

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Stephen P Saville

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P2860

Delaying the empiric treatment of candida bloodstream infection until positive blood culture results are obtained: a potential risk factor for hospital mortality

Nonfilamentous C. albicans mutants are avirulent

NRG1, a repressor of filamentous growth in C.albicans, is down-regulated during filament induction

NRG1 represses yeast-hypha morphogenesis and hypha-specific gene expression in Candida albicans.

The direct cost and incidence of systemic fungal infections.

Resistance of Candida species to antifungal agents: molecular mechanisms and clinical consequences.

TUP1, CPH1 and EFG1 make independent contributions to filamentation in candida albicans.

Engineered control of cell morphology in vivo reveals distinct roles for yeast and filamentous forms of Candida albicans during infection.

Need for early antifungal treatment confirmed in experimental disseminated Candida albicans infection.

Small-molecule inhibitors of the budded-to-hyphal-form transition in the pathogenic yeast Candida albicans.

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3312-3316

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10.1128/AAC.00628-06

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10

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50

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2006-10-01T00:00:00Z

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17005810

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1610055

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