Release of neuronal HMGB1 by ethanol through decreased HDAC activity activates brain neuroimmune signaling. - Wikidata - awgldk - TriplyDB

Release of neuronal HMGB1 by ethanol through decreased HDAC activity activates brain neuroimmune signaling.

Release of neuronal HMGB1 by ethanol through decreased HDAC activity activates brain neuroimmune signaling.

http://www.wikidata.org/entity/Q35097291

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Ethanol-Induced Neurodegeneration and Glial Activation in the Developing Brain Neuroimmune Function and the Consequences of Alcohol Exposure Stress sounds the alarmin: The role of the danger-associated molecular pattern HMGB1 in stress-induced neuroinflammatory priming Adolescent Alcohol Exposure Persistently Impacts Adult Neurobiology and Behavior Mechanisms of neuroimmune gene induction in alcoholism Exposure of neonatal rats to alcohol has differential effects on neuroinflammation and neuronal survival in the cerebellum and hippocampus.Genome-wide association data suggest ABCB1 and immune-related gene sets may be involved in adult antisocial behavior Ethanol, TLR3, and TLR4 Agonists Have Unique Innate Immune Responses in Neuron-Like SH-SY5Y and Microglia-Like BV2 Toll-like receptor signaling and stages of addiction.P2X4 receptor regulates alcohol-induced responses in microglia The epigenetic landscape of alcoholism.Fetal alcohol spectrum disorders and neuroimmune changes Chronic ethanol exposure produces time- and brain region-dependent changes in gene coexpression networks Brain pathways to recovery from alcohol dependence Mathematical model on Alzheimer's disease Inhibition of HMGB1 release via salvianolic acid B-mediated SIRT1 up-regulation protects rats against non-alcoholic fatty liver disease.The redox state of the alarmin HMGB1 is a pivotal factor in neuroinflammatory and microglial priming: A role for the NLRP3 inflammasome.Characterization of the Inflammatory Properties of Actively Released HMGB1 in Juvenile Idiopathic Arthritis.Alcohol resistance in Drosophila is modulated by the Toll innate immune pathway Immune-system-dependent anti-tumor activity of a plant-derived polyphenol rich fraction in a melanoma mouse model.Microglial-derived miRNA let-7 and HMGB1 contribute to ethanol-induced neurotoxicity via TLR7.Neuroimmune basis of alcoholic brain damage.Histone-mediated epigenetics in addiction.Alterations in Histone Acetylases and Acetylated Histone Expression in Aging, Ethanol-Fed Rats.Histone deacetylase-high mobility group box-1 pathway targeted by hypaconitine suppresses the apoptosis of endothelial cells.Danger Signals and Inflammasomes: Stress-Evoked Sterile Inflammation in Mood Disorders.Role of epigenetics in modulation of immune response at the junction of host-pathogen interaction and danger molecule signaling.Impact of the Innate Immune Response in the Actions of Ethanol on the Central Nervous System.Sterile Neuroinflammation and Strategies for Therapeutic Intervention The role of neuroimmune signaling in alcoholism.Histone Deacetylase Gene Expression Following Binge Alcohol Consumption in Rats and Humans.Omega-3 polyunsaturated fatty acid supplementation attenuates microglial-induced inflammation by inhibiting the HMGB1/TLR4/NF-κB pathway following experimental traumatic brain injury.Role of interleukin-10 (IL-10) in regulation of GABAergic transmission and acute response to ethanol Organ-Organ Crosstalk and Alcoholic Liver Disease.Genetic and Pharmacologic Manipulation of TLR4 Has Minimal Impact on Ethanol Consumption in Rodents.The danger-associated molecular pattern HMGB1 mediates the neuroinflammatory effects of methamphetamine.Gut region-dependent alterations of nitrergic myenteric neurons after chronic alcohol consumption.Glial and neuroinflammatory targets for treating substance use disorders.Innately activated TLR4 signal in the nucleus accumbens is sustained by CRF amplification loop and regulates impulsivity.HMGB1/IL-1β complexes regulate neuroimmune responses in alcoholism.

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P2860

Release of neuronal HMGB1 by ethanol through decreased HDAC activity activates brain neuroimmune signaling.

http://www.wikidata.org/entity/Q35097291

description

2014 nî lūn-bûn

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2014 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի փետրվարին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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Release of neuronal HMGB1 by e ...... s brain neuroimmune signaling.

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Release of neuronal HMGB1 by e ...... s brain neuroimmune signaling.

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Fulton T Crews

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Jian Y Zou

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P2860

Binge alcohol drinking is associated with GABAA alpha2-regulated Toll-like receptor 4 (TLR4) expression in the central amygdala

The nuclear protein HMGB1 is secreted by monocytes via a non-classical, vesicle-mediated secretory pathway

Systemic LPS causes chronic neuroinflammation and progressive neurodegeneration

Expression of ethanol-induced behavioral sensitization is associated with alteration of chromatin remodeling in mice

Release of chromatin protein HMGB1 by necrotic cells triggers inflammation

TNF alpha potentiates glutamate neurotoxicity by inhibiting glutamate uptake in organotypic brain slice cultures: neuroprotection by NF kappa B inhibition

High-mobility group box 1 protein (HMGB1): nuclear weapon in the immune arsenal

Involvement of TLR4/type I IL-1 receptor signaling in the induction of inflammatory mediators and cell death induced by ethanol in cultured astrocytes

Non-stereoselective reversal of neuropathic pain by naloxone and naltrexone: involvement of toll-like receptor 4 (TLR4)

Alcoholism and inflammation: neuroimmunology of behavioral and mood disorders

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