The complement inhibitors Crry and factor H are critical for preventing autologous complement activation on renal tubular epithelial cells.
about
Structural basis for sialic acid-mediated self-recognition by complement factor HTissue-specific deletion of Crry from mouse proximal tubular epithelial cells increases susceptibility to renal ischemia-reperfusion injury.Cellular pathophysiology of ischemic acute kidney injuryComplement activation and toll-like receptor-2 signaling contribute to cytokine production after renal ischemia/reperfusion.Annexin A2 Enhances Complement Activation by Inhibiting Factor HBlocking properdin, the alternative pathway, and anaphylatoxin receptors ameliorates renal ischemia-reperfusion injury in decay-accelerating factor and CD59 double-knockout miceComplement Factor B Production in Renal Tubular Cells and Its Role in Sodium Transporter Expression During Polymicrobial Sepsis.Distinct roles for the complement regulators factor H and Crry in protection of the kidney from injury.Cyclosporine induces endothelial cell release of complement-activating microparticles.The role of the complement system in acute kidney injury.Acute kidney injury: a conspiracy of Toll-like receptor 4 on endothelia, leukocytes, and tubules.Complement as a multifaceted modulator of kidney transplant injury.Mechanisms of maladaptive repair after AKI leading to accelerated kidney ageing and CKD.Factor H C-Terminal Domains Are Critical for Regulation of Platelet/Granulocyte Aggregate Formation.Modulation of the Alternative Pathway of Complement by Murine Factor H-Related Proteins.Complement factor H protects mice from ischemic acute kidney injury but is not critical for controlling complement activation by glomerular IgM.
P2860
Q27696183-C9304E78-19EE-46E5-A8C7-E29F32ACD4D3Q34276119-13D08658-E54D-43E5-8645-EE67420A50D0Q35484725-CBAACD02-5512-482C-A704-36CC29377BF1Q36119405-18B1212E-B0B3-4C90-9083-F7058C89C215Q36495141-6985333D-F3B9-4AE9-AEA6-0A70EB852980Q36718295-8B31D316-0CBA-401A-A710-C54C7857CC92Q36803645-1AC2A011-A9C0-4D34-ADCF-03EC2BDFCBBDQ37017177-5E7DFDB4-39B4-4A32-B830-3F30F9C8665CQ37260192-1D77FC70-96A7-45DC-8BB6-1148A0A0FD65Q37275159-C760D7B1-CF3F-4BD9-819F-87A0EEE5425DQ37950251-8325FB24-CEB7-49A3-8DE7-802C9D97F4F2Q38217273-19AB2192-F7FD-466A-BEAA-56C7040D89B6Q38341214-CFAAACF7-72C0-4441-9191-591B629D9041Q47100595-5DA1A964-C075-4DF8-959C-64EACCCC8E1EQ47349991-8519D71E-63D2-47C0-B1C1-8A4C3FA7FCB5Q49497379-B8F287EE-91BC-4DD1-914F-CB67B0A8B928
P2860
The complement inhibitors Crry and factor H are critical for preventing autologous complement activation on renal tubular epithelial cells.
description
2010 nî lūn-bûn
@nan
2010 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
The complement inhibitors Crry ...... enal tubular epithelial cells.
@ast
The complement inhibitors Crry ...... enal tubular epithelial cells.
@en
type
label
The complement inhibitors Crry ...... enal tubular epithelial cells.
@ast
The complement inhibitors Crry ...... enal tubular epithelial cells.
@en
prefLabel
The complement inhibitors Crry ...... enal tubular epithelial cells.
@ast
The complement inhibitors Crry ...... enal tubular epithelial cells.
@en
P2093
P2860
P356
P1476
The complement inhibitors Crry ...... renal tubular epithelial cells
@en
P2093
Amanda Holland-Neidermyer
Brandon Renner
Claudia Amura
Heather N Orth
Hector Molina
Joshua M Thurman
Kathrin Coleman
Kathryn Pierce
Michael K Pangburn
Ryan Goldberg
P2860
P304
P356
10.4049/JIMMUNOL.1000111
P407
P577
2010-07-30T00:00:00Z