Enhancers compete with a long non-coding RNA for regulation of the Kcnq1 domain - Wikidata - awgldk - TriplyDB

Enhancers compete with a long non-coding RNA for regulation of the Kcnq1 domain

Enhancers compete with a long non-coding RNA for regulation of the Kcnq1 domain

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A HuGE Review and Meta-Analyses of Genetic Associations in New Onset Diabetes after Kidney Transplantation Characteristics of Antisense Transcript Promoters and the Regulation of Their Activity Conservation of Repeats at the Mammalian KCNQ1OT1-CDKN1C Region Suggests a Role in Genomic Imprinting Transancestral fine-mapping of four type 2 diabetes susceptibility loci highlights potential causal regulatory mechanisms.LncRNAs in vertebrates: advances and challenges.Knockdown of Long Non-Coding RNA KCNQ1OT1 Restrained Glioma Cells' Malignancy by Activating miR-370/CCNE2 Axis.Non-coding RNAs in muscle differentiation and musculoskeletal disease.Sex chromosomes drive gene expression and regulatory dimorphisms in mouse embryonic stem cells.Blocked transcription through KvDMR1 results in absence of methylation and gene silencing resembling Beckwith-Wiedemann syndrome.On the Origin of lncRNAs: Missing Link Found.Reciprocal F1 Hybrids of Two Inbred Mouse Strains Reveal Parent-of-Origin and Perinatal Diet Effects on Behavior and Expression A novel cancer syndrome caused by -deficiency in the golden Syrian hamster

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P2860

Enhancers compete with a long non-coding RNA for regulation of the Kcnq1 domain

http://www.wikidata.org/entity/Q35097959

description

2014 nî lūn-bûn

@nan

2014 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի դեկտեմբերին հրատարակված գիտական հոդված

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2014年の論文

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2014年学术文章

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2014年学术文章

@zh-cn

2014年学术文章

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2014年学术文章

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2014年学术文章

@zh-sg

2014年學術文章

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name

Enhancers compete with a long non-coding RNA for regulation of the Kcnq1 domain

@ast

Enhancers compete with a long non-coding RNA for regulation of the Kcnq1 domain

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type

label

Enhancers compete with a long non-coding RNA for regulation of the Kcnq1 domain

@ast

Enhancers compete with a long non-coding RNA for regulation of the Kcnq1 domain

@en

prefLabel

Enhancers compete with a long non-coding RNA for regulation of the Kcnq1 domain

@ast

Enhancers compete with a long non-coding RNA for regulation of the Kcnq1 domain

@en

P1433

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Nucleic Acids Research

P1476

Enhancers compete with a long non-coding RNA for regulation of the Kcnq1 domain

@en

P2093

Bryant M Schultz

http://www.w3.org/2001/XMLSchema#string

Gwendolyn A Gallicio

http://www.w3.org/2001/XMLSchema#string

Lena N Lupey

http://www.w3.org/2001/XMLSchema#string

Matteo Cesaroni

http://www.w3.org/2001/XMLSchema#string

Nora Engel

http://www.w3.org/2001/XMLSchema#string

P2860

A user's guide to the encyclopedia of DNA elements (ENCODE)

The Kcnq1ot1 long non-coding RNA affects chromatin conformation and expression of Kcnq1, but does not regulate its imprinting in the developing heart

Holt-Oram syndrome is caused by mutations in TBX5, a member of the Brachyury (T) gene family

Elongation of the Kcnq1ot1 transcript is required for genomic imprinting of neighboring genes

An antisense RNA regulates the bidirectional silencing property of the Kcnq1 imprinting control region

Mediator and cohesin connect gene expression and chromatin architecture

A maternally methylated CpG island in KvLQT1 is associated with an antisense paternal transcript and loss of imprinting in Beckwith-Wiedemann syndrome

Different TBX5 interactions in heart and limb defined by Holt-Oram syndrome mutations

VISTA Enhancer Browser--a database of tissue-specific human enhancers

Loss of imprinting of a paternally expressed transcript, with antisense orientation to KVLQT1, occurs frequently in Beckwith-Wiedemann syndrome and is independent of insulin-like growth factor II imprinting

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745-759

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scholarly article

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10.1093/NAR/GKU1324

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2

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43

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