Crimpy inhibits the BMP homolog Gbb in motoneurons to enable proper growth control at the Drosophila neuromuscular junction
about
The Crossroads of Synaptic Growth Signaling, Membrane Traffic and Neurological Disease: Insights from DrosophilaThe Wnt and BMP families of signaling morphogens at the vertebrate neuromuscular junctionGrowth differentiation factor 6 as a putative risk factor in neuromuscular degenerationAnterograde Activin signaling regulates postsynaptic membrane potential and GluRIIA/B abundance at the Drosophila neuromuscular junctionBAR-SH3 sorting nexins are conserved interacting proteins of Nervous wreck that organize synapses and promote neurotransmissionGrowth factors in synaptic function.Epsin 1 Promotes Synaptic Growth by Enhancing BMP Signal Levels in Motoneuron Nuclei.FoxO limits microtubule stability and is itself negatively regulated by microtubule disruptionTransmission, Development, and Plasticity of Synapses.Production of a mouse line with a conditional Crim1 mutant allele.Crim1 has cell-autonomous and paracrine roles during embryonic heart development.Postsynaptic glutamate receptors regulate local BMP signaling at the Drosophila neuromuscular junction.Crim1 maintains retinal vascular stability during development by regulating endothelial cell Vegfa autocrine signaling.The multiple activities of BMPs during spinal cord development.Bone morphogenetic protein signaling in vertebrate motor neurons and neuromuscular communication.Matters of context guide future research in TGFβ superfamily signaling.Agonists and Antagonists of TGF-β Family Ligands.TGF-β Family Signaling in Drosophila.A large-scale gene discovery for the red palm weevil Rhynchophorus ferrugineus (Coleoptera: Curculionidae).Context-dependent proangiogenic function of bone morphogenetic protein signaling is mediated by disabled homolog 2.The equilibrium between antagonistic signaling pathways determines the number of synapses in Drosophila.Neuroligin 4 regulates synaptic growth via the bone morphogenetic protein (BMP) signaling pathway at the Drosophila neuromuscular junction.Drosophila Nociceptive Sensitization Requires BMP Signaling via the Canonical SMAD Pathway.Secreted tissue inhibitor of matrix metalloproteinase restricts trans-synaptic signaling to coordinate synaptogenesis.Bone Morphogenetic Protein Glass Bottom Boat (BMP5/6/7/8) and its receptor Wishful Thinking (BMPRII) are required for injury-induced allodynia in Drosophila
P2860
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P2860
Crimpy inhibits the BMP homolog Gbb in motoneurons to enable proper growth control at the Drosophila neuromuscular junction
description
2011 nî lūn-bûn
@nan
2011 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Crimpy inhibits the BMP homolo ...... sophila neuromuscular junction
@ast
Crimpy inhibits the BMP homolo ...... sophila neuromuscular junction
@en
type
label
Crimpy inhibits the BMP homolo ...... sophila neuromuscular junction
@ast
Crimpy inhibits the BMP homolo ...... sophila neuromuscular junction
@en
prefLabel
Crimpy inhibits the BMP homolo ...... sophila neuromuscular junction
@ast
Crimpy inhibits the BMP homolo ...... sophila neuromuscular junction
@en
P2860
P356
P1433
P1476
Crimpy inhibits the BMP homolo ...... sophila neuromuscular junction
@en
P2093
Heather T Broihier
Rebecca E James
P2860
P304
P356
10.1242/DEV.066142
P407
P577
2011-08-01T00:00:00Z