Resolution of acute malarial infections by T cell-dependent non-antibody-mediated mechanisms of immunity.
about
CD4+ and CD8+ T lymphocytes both contribute to acquired immunity to blood-stage Plasmodium chabaudi AST-cell-dependent immunity and thrombocytopenia in rats infected with Plasmodium chabaudi.Heterogeneity in cytokine profiles of Babesia bovis-specific bovine CD4+ T cells clones activated in vitro.Roles of CD4(+) T cells and gamma interferon in protective immunity against Babesia microti infection in mice.Cellular immunity, but not gamma interferon, is essential for resolution of Babesia microti infection in BALB/c mice.Gammadelta T cells but not NK cells are essential for cell-mediated immunity against Plasmodium chabaudi malaria.Identification of two Th1 cell epitopes on the Babesia bovis-encoded 77-kilodalton merozoite protein (Bb-1) by use of truncated recombinant fusion proteins.Human babesiosis.Splenic gammadelta T cells regulated by CD4+ T cells are required to control chronic Plasmodium chabaudi malaria in the B-cell-deficient mouseSignalling through the IL-2 receptor γ(c) peptide (CD132) is essential for the expression of immunity to Plasmodium chabaudi adami blood-stage malaria.Cellular and humoral immune responses to well-defined blood stage antigens (major merozoite surface antigen) of Plasmodium falciparum in adults from an Indian zone where malaria is endemic.Specific immune responses are required to control parasitemia in Babesia equi infectionA Novel Model of Asymptomatic Plasmodium Parasitemia That Recapitulates Elements of the Human Immune Response to Chronic Infection.Macrophage-mediated but gamma interferon-independent innate immune responses control the primary wave of Plasmodium yoelii parasitemiaBovine helper T cell clones recognize five distinct epitopes on Babesia bovis merozoite antigens.Macrophage activation during Plasmodium chabaudi AS infection in resistant C57BL/6 and susceptible A/J miceHuman T cell recognition of the blood stage antigen Plasmodium hypoxanthine guanine xanthine phosphoribosyl transferase (HGXPRT) in acute malaria.CD28 costimulation is required for the expression of T-cell-dependent cell-mediated immunity against blood-stage Plasmodium chabaudi malaria parasites.Blood transfusion alters the course and outcome of Plasmodium chabaudi AS infection in mice.
P2860
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P2860
Resolution of acute malarial infections by T cell-dependent non-antibody-mediated mechanisms of immunity.
description
1990 nî lūn-bûn
@nan
1990 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
1990 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
1990年の論文
@ja
1990年論文
@yue
1990年論文
@zh-hant
1990年論文
@zh-hk
1990年論文
@zh-mo
1990年論文
@zh-tw
1990年论文
@wuu
name
Resolution of acute malarial i ...... diated mechanisms of immunity.
@ast
Resolution of acute malarial i ...... diated mechanisms of immunity.
@en
type
label
Resolution of acute malarial i ...... diated mechanisms of immunity.
@ast
Resolution of acute malarial i ...... diated mechanisms of immunity.
@en
prefLabel
Resolution of acute malarial i ...... diated mechanisms of immunity.
@ast
Resolution of acute malarial i ...... diated mechanisms of immunity.
@en
P2093
P2860
P1476
Resolution of acute malarial i ...... diated mechanisms of immunity.
@en
P2093
L A Cavacini
W P Weidanz
P2860
P304
P407
P577
1990-09-01T00:00:00Z