Recognition of H3K9 methylation by GLP is required for efficient establishment of H3K9 methylation, rapid target gene repression, and mouse viability.
about
Epigenetic modulators, modifiers and mediators in cancer aetiology and progressionHistone methyltransferases: novel targets for tumor and developmental defectsChromatin states and nuclear organization in development--a view from the nuclear laminaNucleation and spreading of a heterochromatic domain in fission yeast.EHMT2 directs DNA methylation for efficient gene silencing in mouse embryos.Tet3 Reads 5-Carboxylcytosine through Its CXXC Domain and Is a Potential Guardian against NeurodegenerationA two-state activation mechanism controls the histone methyltransferase Suv39h1.Jmjd2c facilitates the assembly of essential enhancer-protein complexes at the onset of embryonic stem cell differentiation.Regulation of cell differentiation and function by the euchromatin histone methyltranserfases G9a and GLP.Sound of silence: the properties and functions of repressive Lys methyltransferases.Functional coupling between writers, erasers and readers of histone and DNA methylation.H3K9me3-Dependent Heterochromatin: Barrier to Cell Fate Changes.The expanding role of the Ehmt2/G9a complex in neurodevelopment.Nonhistone Lysine Methylation in the Regulation of Cancer Pathways.Are there specific readers of oxidized 5-methylcytosine bases?Writing, erasing and reading histone lysine methylations.JMJD1C Ensures Mouse Embryonic Stem Cell Self-Renewal and Somatic Cell Reprogramming through Controlling MicroRNA Expression.The lysine methyltransferase Ehmt2/G9a is dispensable for skeletal muscle development and regeneration.Pharmacologic control of chromatin looping.Striking a balance: regulation of transposable elements by Zfp281 and Mll2 in mouse embryonic stem cells.H3K14ac is linked to methylation of H3K9 by the triple Tudor domain of SETDB1.GLP inhibits heterochromatin clustering and myogenic differentiation by repressing MeCP2.Discovery of Potent and Selective Inhibitors for G9a-Like Protein (GLP) Lysine Methyltransferase.The level and distribution pattern of HP1β in the embryonic brain correspond to those of H3K9me1/me2 but not of H3K9me3.Structure-activity relationship studies of G9a-like protein (GLP) inhibitors.Allosteric Activation Dictates PRC2 Activity Independent of Its Recruitment to Chromatin.Increased H3K9 methylation and impaired expression of Protocadherins are associated with the cognitive dysfunctions of the Kleefstra syndrome.
P2860
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P2860
Recognition of H3K9 methylation by GLP is required for efficient establishment of H3K9 methylation, rapid target gene repression, and mouse viability.
description
2015 nî lūn-bûn
@nan
2015 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2015 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
name
Recognition of H3K9 methylatio ...... pression, and mouse viability.
@ast
Recognition of H3K9 methylatio ...... pression, and mouse viability.
@en
type
label
Recognition of H3K9 methylatio ...... pression, and mouse viability.
@ast
Recognition of H3K9 methylatio ...... pression, and mouse viability.
@en
prefLabel
Recognition of H3K9 methylatio ...... pression, and mouse viability.
@ast
Recognition of H3K9 methylatio ...... pression, and mouse viability.
@en
P2093
P2860
P356
P1433
P1476
Recognition of H3K9 methylatio ...... pression, and mouse viability.
@en
P2093
Lingjun Meng
Shaorong Gao
P2860
P304
P356
10.1101/GAD.254425.114
P577
2015-01-30T00:00:00Z