The transcription factor PU.1 is required for the development of IL-9-producing T cells and allergic inflammation. - Wikidata - awgldk - TriplyDB

The transcription factor PU.1 is required for the development of IL-9-producing T cells and allergic inflammation.

The transcription factor PU.1 is required for the development of IL-9-producing T cells and allergic inflammation.

http://www.wikidata.org/entity/Q35105032

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Evolutionary divergence and functions of the human interleukin (IL) gene family Recent advances in understanding the molecular mechanisms of the development and function of Th17 cells The TNF-family cytokine TL1A: from lymphocyte costimulator to disease co-conspirator Change of paradigm: CD8+ T cells as important helper for CD4+ T cells during asthma and autoimmune encephalomyelitis T Helper Subsets, Peripheral Plasticity, and the Acute Phase Protein, α1-Antitrypsin The mucosal immune system in the oral cavity-an orchestra of T cell diversity STAT6-dependent and -independent mechanisms in Th2 polarization Mechanisms underlying helper T-cell plasticity: implications for immune-mediated disease.Antigen Presentation, Autoantigens, and Immune Regulation in Multiple Sclerosis and Other Autoimmune Diseases The development and in vivo function of T helper 9 cells BATF-JUN is critical for IRF4-mediated transcription in T cells High-dimensional genomic data bias correction and data integration using MANCIE An increased expression profile of Th9/IL-9 correlated with Th17/IL-17 in patients with immune thrombocytopenia.TGF-β activation and function in immunity.The ST2/IL-33 Axis in Immune Cells during Inflammatory Diseases.Cytokines (interleukin-9, IL-17, IL-22, IL-25 and IL-33) and asthma.Dynamics of Th9 cells and their potential role in immunopathogenesis of murine schistosomiasis.A highly selective, label-free, homogenous luminescent switch-on probe for the detection of nanomolar transcription factor NF-kappaB.The signaling suppressor CIS controls proallergic T cell development and allergic airway inflammation.Human TH9 cells are skin-tropic and have autocrine and paracrine proinflammatory capacity.IL-33 induces IL-9 production in human CD4+ T cells and basophils.The transcription factor PU.1 regulates γδ T cell homeostasis Role of Tyk-2 in Th9 and Th17 cells in allergic asthma.BCL6 controls Th9 cell development by repressing Il9 transcription.Nitric oxide enhances Th9 cell differentiation and airway inflammation.Th17 cells demonstrate stable cytokine production in a proallergic environment A brief history of IL-9 Differentiation and recruitment of Th9 cells stimulated by pleural mesothelial cells in human Mycobacterium tuberculosis infection.Complex interactions of transcription factors in mediating cytokine biology in T cells.Transcriptional and epigenetic control of T helper cell specification: molecular mechanisms underlying commitment and plasticity.DNA-binding small molecules as inhibitors of transcription factors.The role of interleukin-9 in lymphoma.Antigen-specific Th9 cells exhibit uniqueness in their kinetics of cytokine production and short retention at the inflammatory site.In-depth profiling and analysis of host and viral microRNAs in Japanese flounder (Paralichthys olivaceus) infected with megalocytivirus reveal involvement of microRNAs in host-virus interaction in teleost fish.Murine Th9 cells promote the survival of myeloid dendritic cells in cancer immunotherapy.Lineage-specific effects of 1,25-dihydroxyvitamin D(3) on the development of effector CD4 T cells.Involvement of IL-9 in Th17-associated inflammation and angiogenesis of psoriasis Transcriptional and epigenetic regulation of T-helper lineage specification.Immunomodulatory effects of transforming growth factor-β in the liver.IL-9 regulates allergen-specific Th1 responses in allergic contact dermatitis

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The transcription factor PU.1 is required for the development of IL-9-producing T cells and allergic inflammation.

http://www.wikidata.org/entity/Q35105032

description

2010 nî lūn-bûn

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2010 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2010 թվականի մայիսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

The transcription factor PU.1 ...... lls and allergic inflammation.

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The transcription factor PU.1 ...... lls and allergic inflammation.

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The transcription factor PU.1 ...... lls and allergic inflammation.

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The transcription factor PU.1 ...... lls and allergic inflammation.

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The transcription factor PU.1 ...... lls and allergic inflammation.

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The transcription factor PU.1 ...... lls and allergic inflammation.

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Nature Immunology

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The transcription factor PU.1 ...... ells and allergic inflammation

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Ayele-Nati Ahyi

http://www.w3.org/2001/XMLSchema#string

Carl McKinley

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Evelyn T Nguyen

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Gretta L Stritesky

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Hua-Chen Chang

http://www.w3.org/2001/XMLSchema#string

Ling Han

http://www.w3.org/2001/XMLSchema#string

Michael J Robertson

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Narayanan B Perumal

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Qing Yu

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Robert S Tepper

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P2860

Global mapping of H3K4me3 and H3K27me3 reveals specificity and plasticity in lineage fate determination of differentiating CD4+ T cells

IL-17 and Th17 Cells

TH17 cells in development: an updated view of their molecular identity and genetic programming

IL-4 inhibits TGF-beta-induced Foxp3+ T cells and, together with TGF-beta, generates IL-9+ IL-10+ Foxp3(-) effector T cells

The lineage decisions of helper T cells

Interleukin-9 enhances resistance to the intestinal nematode Trichuris muris.

IL-9-deficient mice establish fundamental roles for IL-9 in pulmonary mastocytosis and goblet cell hyperplasia but not T cell development.

Mast cells are essential intermediaries in regulatory T-cell tolerance.

IL-9- and mast cell-mediated intestinal permeability predisposes to oral antigen hypersensitivity.

Plasticity of CD4+ T cell lineage differentiation.

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527-534

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10.1038/NI.1867

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6

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11

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Stephen L. Nutt

Ritobrata Goswami

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2010-05-02T00:00:00Z

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43355533

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20431622

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3136246

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