S100A9 induced inflammatory responses are mediated by distinct damage associated molecular patterns (DAMP) receptors in vitro and in vivo. - Wikidata - awgldk - TriplyDB

S100A9 induced inflammatory responses are mediated by distinct damage associated molecular patterns (DAMP) receptors in vitro and in vivo.

S100A9 induced inflammatory responses are mediated by distinct damage associated molecular patterns (DAMP) receptors in vitro and in vivo.

http://www.wikidata.org/entity/Q35112587

about

Combined toll-like receptor 3/7/9 deficiency on host cells results in T-cell-dependent control of tumour growth S100A8/A9 and S100A9 reduce acute lung injury.Cecal Ligation and Puncture Results in Long-Term Central Nervous System Myeloid Inflammation.MCAM, as a novel receptor for S100A8/A9, mediates progression of malignant melanoma through prominent activation of NF-κB and ROS formation upon ligand binding.Topical application of the anti-microbial chemical triclosan induces immunomodulatory responses through the S100A8/A9-TLR4 pathway Biomarkers of inflammation and innate immunity in atrophic nonunion fracture.S100P is associated with proliferation and migration in nasopharyngeal carcinoma.Tumor-derived factors modulating dendritic cell function.Impaired response of the bronchial epithelium to inflammation characterizes severe equine asthma.Expression, Purification, and Antimicrobial Activity of S100A12.CD8+ T cells produce a dialyzable antigen-specific activator of dendritic cells.Alarmin S100A8 Activates Alveolar Epithelial Cells in the Context of Acute Lung Injury in a TLR4-Dependent Manner.S100A9 gene silencing inhibits the release of pro-inflammatory cytokines by blocking the IL-17 signalling pathway in mice with acute pancreatitis.Immunothrombotic Activity of Damage-Associated Molecular Patterns and Extracellular Vesicles in Secondary Organ Failure Induced by Trauma and Sterile Insults.Coevolution of the Toll-Like Receptor 4 Complex with Calgranulins and Lipopolysaccharide.Association between serum S100A9 levels and liver necroinflammation in chronic hepatitis B.HBx-induced S100A9 in NF-κB dependent manner promotes growth and metastasis of hepatocellular carcinoma cells.S100A8/A9 in Inflammation.Exacerbation of hepatic injury during rodent malaria by myeloid-related protein 14.S100A9-Driven Amyloid-Neuroinflammatory Cascade in Traumatic Brain Injury as a Precursor State for Alzheimer's Disease Genetic screen in myeloid cells identifies TNF-α autocrine secretion as a factor increasing MDSC suppressive activity via Nos2 up-regulation Pattern Recognition Receptors and the Host Cell Death Molecular Machinery

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S100A9 induced inflammatory responses are mediated by distinct damage associated molecular patterns (DAMP) receptors in vitro and in vivo.

http://www.wikidata.org/entity/Q35112587

description

2015 nî lūn-bûn

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2015 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2015 թվականի փետրվարին հրատարակված գիտական հոդված

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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name

S100A9 induced inflammatory re ...... eceptors in vitro and in vivo.

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S100A9 induced inflammatory re ...... eceptors in vitro and in vivo.

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JOURNAL.PONE.0115828

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S100A9 induced inflammatory re ...... eceptors in vitro and in vivo.

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Alison A Humbles

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Allison L Miller

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Bo Chen

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Brian Naiman

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Chew-Shun Chang

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Daniel C Rowe

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De Yang

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Gary P Sims

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Kim Rosenthal

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Lori Clarke

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P2860

Identification of human S100A9 as a novel target for treatment of autoimmune disease via binding to quinoline-3-carboxamides

RAGE mediates a novel proinflammatory axis: a central cell surface receptor for S100/calgranulin polypeptides

Defective LPS Signaling in C3H/HeJ and C57BL/10ScCr Mice: Mutations in Tlr4 Gene

Proinflammatory activities of S100: proteins S100A8, S100A9, and S100A8/A9 induce neutrophil chemotaxis and adhesion

Chemotactic activity of S100A7 (Psoriasin) is mediated by the receptor for advanced glycation end products and potentiates inflammation with highly homologous but functionally distinct S100A15

Induction of immunological tolerance by apoptotic cells requires caspase-dependent oxidation of high-mobility group box-1 protein

S100B and S100A6 differentially modulate cell survival by interacting with distinct RAGE (receptor for advanced glycation end products) immunoglobulin domains

Inhibition of dendritic cell differentiation and accumulation of myeloid-derived suppressor cells in cancer is regulated by S100A9 protein

RAGE is a nucleic acid receptor that promotes inflammatory responses to DNA

High-mobility group box 1 protein (HMGB1): nuclear weapon in the immune arsenal

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e0115828

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scholarly article

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10.1371/JOURNAL.PONE.0115828

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