Hyper-Acetylation of Histone H3K56 Limits Break-Induced Replication by Inhibiting Extensive Repair Synthesis.
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Pathways and Mechanisms that Prevent Genome Instability in Saccharomyces cerevisiaeChromosome-wide histone deacetylation by sirtuins prevents hyperactivation of DNA damage-induced signaling upon replicative stress.Multi-BRCT scaffolds use distinct strategies to support genome maintenance.Nicotinamide Suppresses the DNA Damage Sensitivity of Saccharomyces cerevisiae Independently of Sirtuin Deacetylases.Environmental change drives accelerated adaptation through stimulated copy number variation.HST1 increases replicative lifespan of a sir2Δ mutant in the absence of PDE2 in Saccharomyces cerevisiae.
P2860
Hyper-Acetylation of Histone H3K56 Limits Break-Induced Replication by Inhibiting Extensive Repair Synthesis.
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2015 nî lūn-bûn
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2015 թուականի Փետրուարին հրատարակուած գիտական յօդուած
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2015 թվականի փետրվարին հրատարակված գիտական հոդված
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2015年の論文
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2015年論文
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2015年論文
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2015年論文
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2015年論文
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2015年論文
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2015年论文
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name
Hyper-Acetylation of Histone H ...... ng Extensive Repair Synthesis.
@ast
Hyper-Acetylation of Histone H ...... ng Extensive Repair Synthesis.
@en
type
label
Hyper-Acetylation of Histone H ...... ng Extensive Repair Synthesis.
@ast
Hyper-Acetylation of Histone H ...... ng Extensive Repair Synthesis.
@en
prefLabel
Hyper-Acetylation of Histone H ...... ng Extensive Repair Synthesis.
@ast
Hyper-Acetylation of Histone H ...... ng Extensive Repair Synthesis.
@en
P2093
P2860
P1433
P1476
Hyper-Acetylation of Histone H ...... ng Extensive Repair Synthesis.
@en
P2093
Eun Yong Shim
Kyungjae Myung
Sang Eun Lee
Stephanie Smith
Yoo Jung Kim
P2860
P304
P356
10.1371/JOURNAL.PGEN.1004990
P577
2015-02-23T00:00:00Z