Tau reduction prevents disease in a mouse model of Dravet syndrome - Wikidata - awgldk - TriplyDB

Tau reduction prevents disease in a mouse model of Dravet syndrome

Tau reduction prevents disease in a mouse model of Dravet syndrome

http://www.wikidata.org/entity/Q35115219

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Pathogenesis and new candidate treatments for infantile spasms and early life epileptic encephalopathies: A view from preclinical studies Spreading depolarization in the brainstem mediates sudden cardiorespiratory arrest in mouse SUDEP models Mechanisms of sudden unexplained death in epilepsy Tau protein is essential for stress-induced brain pathology Phosphorylation of tau at Y18, but not tau-fyn binding, is required for tau to modulate NMDA receptor-dependent excitotoxicity in primary neuronal culture.Tau reduction diminishes spatial learning and memory deficits after mild repetitive traumatic brain injury in mice.Tau reduction prevents Aβ-induced axonal transport deficits by blocking activation of GSK3β.Network dysfunction in α-synuclein transgenic mice and human Lewy body dementia.Early rescue of interneuron disease trajectory in developmental epilepsies.Expression of A152T human tau causes age-dependent neuronal dysfunction and loss in transgenic mice.Pathway-driven discovery of epilepsy genes.Increased 4R-Tau Induces Pathological Changes in a Human-Tau Mouse Model.Single-Gene Determinants of Epilepsy Comorbidity.One Mutation Deserves Another in the Quest for Antiepileptogenesis.Tau post-translational modifications in wild-type and human amyloid precursor protein transgenic mice.Atypical, non-standard functions of the microtubule associated Tau protein.Accelerated kindling epileptogenesis in Tg4510 tau transgenic mice, but not in tau knockout mice.Tau-based therapies in neurodegeneration: opportunities and challenges.Maintenance of the Innate Seizure Threshold by Cyclooxygenase-2 is Not Influenced by the Translational Silencer, T-cell Intracellular Antigen-1.Cognition and dementia in older patients with epilepsy.Tau-Induced Pathology in Epilepsy and Dementia: Notions from Patients and Animal Models.Tau in neurodegenerative disease.

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Tau reduction prevents disease in a mouse model of Dravet syndrome

http://www.wikidata.org/entity/Q35115219

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2014 nî lūn-bûn

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2014 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի օգոստոսին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

Tau reduction prevents disease in a mouse model of Dravet syndrome

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Tau reduction prevents disease in a mouse model of Dravet syndrome

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Tau reduction prevents disease in a mouse model of Dravet syndrome

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Tau reduction prevents disease in a mouse model of Dravet syndrome

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Tau reduction prevents disease in a mouse model of Dravet syndrome

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Tau reduction prevents disease in a mouse model of Dravet syndrome

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Annals of Neurology

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Tau reduction prevents disease in a mouse model of Dravet syndrome

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P2093

Ania L Gheyara

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Biljana Djukic

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Kaitlyn Ho

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Lennart Mucke

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Mariel M Finucane

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Pascal E Sanchez

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Ravikumar Ponnusamy

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Ryan J Craft

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Weikun Guo

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P2860

Alzheimer mechanisms and therapeutic strategies

Selective inhibition of Alzheimer disease-like tau aggregation by phenothiazines

Inhibitory interneuron deficit links altered network activity and cognitive dysfunction in Alzheimer model.

Levetiracetam suppresses neuronal network dysfunction and reverses synaptic and cognitive deficits in an Alzheimer's disease model.

Tau-targeted treatment strategies in Alzheimer's disease

Dendritic function of tau mediates amyloid-beta toxicity in Alzheimer's disease mouse models

A proposed diagnostic scheme for people with epileptic seizures and with epilepsy: report of the ILAE Task Force on Classification and Terminology

Modification of seizure activity by electrical stimulation. II. Motor seizure

Sudden unexpected death in epilepsy: mechanisms, prevalence, and prevention

Revised terminology and concepts for organization of seizures and epilepsies: report of the ILAE Commission on Classification and Terminology, 2005-2009

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10.1002/ANA.24230

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3

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76

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